Essential role of smooth muscle STIM1 in hypertension and cardiovascular dysfunction

Arteriosclerosis, Thrombosis, and Vascular Biology

Volumn 36, Issue 9, 2016, Pages 1900-1909

  Kassan, Modar ^a,b,d   Ait Aissa, Karima ^b   Radwan, Eman ^b   Mali, Vishal ^b   Haddox, Samuel ^b   Gabani, Mohanad ^d   Zhang, Wei ^c   Belmadani, Souad ^b   Irani, Kaikobad ^d   Trebak, Mohamed ^c   Matrougui, Khalid ^a,b  

^a TULANE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b EASTERN VIRGINIA MEDICAL SCHOOL   (United States)

^c PENN STATE COLLEGE OF MEDICINE   (United States)

^d UNIVERSITY OF IOWA   (United States)

Author keywords

Cardiac hypertrophy; Endothelial nitric oxide synthase; ER stress; Hypertension; Nicotinamide adenine dinucleotide phosphate; Stromal interaction molecule 1; Vascular reactivity

Indexed keywords

ACTIVATING TRANSCRIPTION FACTOR 6; ANGIOTENSIN II; CYCLIC GMP; ENDOTHELIAL NITRIC OXIDE SYNTHASE; GLUCOSE REGULATED PROTEIN 78; GROWTH ARREST AND DNA DAMAGE INDUCIBLE PROTEIN 153; NITRITE; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE; STROMAL INTERACTION MOLECULE 1; TRANSFORMING GROWTH FACTOR BETA; DDIT3 PROTEIN, MOUSE; NOS3 PROTEIN, MOUSE; STIM1 PROTEIN, MOUSE; VASODILATOR AGENT;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLOOD VESSEL INJURY; BLOOD VESSEL REACTIVITY; CARDIOVASCULAR DISEASE; CONTROLLED STUDY; ENDOPLASMIC RETICULUM STRESS; ENDOTHELIAL DYSFUNCTION; ENZYME ACTIVITY; ENZYME PHOSPHORYLATION; GENE DELETION; HEART INJURY; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; HYPERTENSION; IN VIVO STUDY; KNOCKOUT MOUSE; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; SYSTOLIC BLOOD PRESSURE; UPREGULATION; VASCULAR SMOOTH MUSCLE; WILD TYPE; WILD TYPE MOUSE; ANIMAL; BLOOD PRESSURE; CARDIAC MUSCLE; CARDIOMEGALY; DEFICIENCY; DISEASE MODEL; DOSE RESPONSE; DRUG EFFECTS; FIBROSIS; GENETIC PREDISPOSITION; GENETICS; MALE; METABOLISM; PATHOLOGY; PATHOPHYSIOLOGY; PHENOTYPE; PHOSPHORYLATION; SIGNAL TRANSDUCTION; TIME FACTOR; VASODILATATION;

ANGIOTENSIN II; ANIMALS; BLOOD PRESSURE; CARDIOMEGALY; CYCLIC GMP; DISEASE MODELS, ANIMAL; DOSE-RESPONSE RELATIONSHIP, DRUG; ENDOPLASMIC RETICULUM STRESS; FIBROSIS; GENETIC PREDISPOSITION TO DISEASE; HYPERTENSION; MALE; MICE, KNOCKOUT; MUSCLE, SMOOTH, VASCULAR; MYOCARDIUM; NADPH OXIDASE; NITRIC OXIDE SYNTHASE TYPE III; NITRITES; PHENOTYPE; PHOSPHORYLATION; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION; STROMAL INTERACTION MOLECULE 1; TIME FACTORS; TRANSCRIPTION FACTOR CHOP; TRANSFORMING GROWTH FACTOR BETA; VASODILATION; VASODILATOR AGENTS;

EID: 84983782668     PISSN: 10795642     EISSN: 15244636     Source Type: Journal    
DOI: 10.1161/ATVBAHA.116.307869     Document Type: Article

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