Elevated activation of CaMKIIα in the CPEB3-knockout hippocampus impairs a specific form of NMDAR-dependent synaptic depotentiation

Frontiers in Cellular Neuroscience

Volumn 8, Issue Nov, 2014, Pages

  Huang, Wen Hsuan ^a   Chao, Hsu Wen ^a   Tsai, Li Yun ^a   Chung, Ming Hung ^b   Huang, Yi Shuian ^a  

^a INSTITUTE OF BIOMEDICAL SCIENCES   (Taiwan)

^b NATIONAL TSING HUA UNIVERSITY   (Taiwan)

Author keywords

CaMKII; CPEB; Depotentiation; NMDAR; Synaptic depression

Indexed keywords

ALPHA TUBULIN; BETA ACTIN; CALCINEURIN; CALCIUM CALMODULIN DEPENDENT PROTEIN KINASE II; CYTOPLASMIC POLYADENYLATION ELEMENT BINDING PROTEIN; DISKS LARGE HOMOLOG 4; DNA FRAGMENT; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; SYNAPTOPHYSIN;

ADULT; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM TRANSPORT; DNA TRANSFECTION; DOWN REGULATION; FEAR; FEMALE; GENOTYPE; HIPPOCAMPAL SLICE; HIPPOCAMPUS; IMMUNOBLOTTING; IMMUNOFLUORESCENCE MICROSCOPY; IMMUNOHISTOCHEMISTRY; LENTIVIRUS; LENTIVIRUS INFECTION; LONG TERM DEPRESSION; MALE; MEMORY; MOUSE; NERVE CELL CULTURE; NERVE CELL PLASTICITY; NONHUMAN; PLASMID; POLYMERASE CHAIN REACTION; PROTEIN EXPRESSION; SPATIAL LEARNING; SYNAPTIC POTENTIAL; SYNAPTOSOME; UPREGULATION;

EID: 84977954219     PISSN: 16625102     EISSN: None     Source Type: Journal    
DOI: 10.3389/fncel.2014.00367     Document Type: Article

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