ATP sensitive K+ channels are critical for maintaining myocardial perfusion and high energy phosphates in the failing heart

Journal of Molecular and Cellular Cardiology

Volumn 92, Issue , 2016, Pages 116-121

  Jameel, Mohammad N ^a   Xiong, Qiang ^a   Mansoor, Abdul ^a   Bache, Robert J ^a   Zhang, Jianyi ^a,b  

^a UNIVERSITY OF MINNESOTA MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

Author keywords

Heart failure; KATP channels; Myocardial blood flow

Indexed keywords

ADENOSINE RECEPTOR; ADENOSINE TRIPHOSPHATE; GLIBENCLAMIDE; MITOCHONDRIAL K(ATP) CHANNEL; POTASSIUM CHANNEL;

ANIMAL; CARDIAC MUSCLE; CORONARY ARTERY BLOOD FLOW; DISEASE MODEL; DOG; DRUG EFFECTS; HEART FAILURE; HUMAN; METABOLISM; MITOCHONDRION; MYOCARDIAL ISCHEMIA; OXIDATIVE PHOSPHORYLATION; OXYGEN CONSUMPTION; PATHOLOGY; VASODILATATION;

ADENOSINE TRIPHOSPHATE; ANIMALS; CORONARY CIRCULATION; DISEASE MODELS, ANIMAL; DOGS; GLYBURIDE; HEART FAILURE; HUMANS; MITOCHONDRIA; MYOCARDIAL ISCHEMIA; MYOCARDIUM; OXIDATIVE PHOSPHORYLATION; OXYGEN CONSUMPTION; POTASSIUM CHANNELS; RECEPTORS, PURINERGIC P1; VASODILATION;

EID: 84958546481     PISSN: 00222828     EISSN: 10958584     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2016.02.005     Document Type: Article

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