ATF3-dependent cross-talk between cardiomyocytes and macrophages promotes cardiac maladaptive remodeling

International Journal of Cardiology

Volumn 198, Issue , 2015, Pages 232-240

  Koren, L ^a   Alishekevitz, D ^a   Elhanani, O ^a   Nevelsky, A ^b   Hai, T ^c   Kehat, I ^a   Shaked, Y ^a   Aronheim, A ^a  

^a TECHNION ISRAEL INSTITUTE OF TECHNOLOGY   (Israel)

^b Department of Cell Biology and Cancer Science B Rappaport Faculty of Medicine Technion Israel Institute of Technology ^*  (Israel)

^c OHIO STATE UNIVERSITY   (United States)

Author keywords

Bone marrow transplantation; Cardiac hypertrophy; Cardiac remodeling; Macrophages; Pressure overload

Indexed keywords

ACTIVATING TRANSCRIPTION FACTOR 3; PHENYLEPHRINE; ATF3 PROTEIN, MOUSE;

ABLATION THERAPY; ADOPTIVE TRANSFER; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BONE MARROW; BONE MARROW CELL; BONE MARROW TRANSPLANTATION; CELL INTERACTION; CONTROLLED STUDY; HEART MUSCLE CELL; HEART VENTRICLE HYPERTROPHY; HEART VENTRICLE OVERLOAD; HEART VENTRICLE REMODELING; IMMUNOREGULATION; KNOCKOUT MOUSE; MACROPHAGE; MACROPHAGE MIGRATION; MALE; MOUSE; MOUSE MODEL; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; WILD TYPE; ANIMAL; BIOSYNTHESIS; C57BL MOUSE; CARDIAC MUSCLE CELL; CARDIOMEGALY; DEFICIENCY; METABOLISM; PATHOLOGY; PHYSIOLOGY;

ACTIVATING TRANSCRIPTION FACTOR 3; ANIMALS; CARDIOMEGALY; MACROPHAGES; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYOCYTES, CARDIAC; VENTRICULAR REMODELING;

EID: 84940434879     PISSN: 01675273     EISSN: 18741754     Source Type: Journal    
DOI: 10.1016/j.ijcard.2015.06.099     Document Type: Article

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