JunD attenuates phenylephrine-mediated cardiomyocyte hypertrophy by negatively regulating AP-1 transcriptional activity

Cardiovascular Research

Volumn 71, Issue 1, 2006, Pages 108-117

  Hilfiker Kleiner, Denise ^a   Hilfiker, Andres ^a   Castellazzi, Marc ^b   Wollert, Kai C ^a   Trautwein, Christian ^c   Schunkert, Heribert ^d   Drexler, Helmut ^a  

^a HANNOVER MEDICAL SCHOOL   (Germany)

^b INSERM   (France)

^c UNIVERSITY HOSPITAL RWTH AACHEN   (Germany)

^d UNIVERSITY OF LÜBECK   (Germany)

Author keywords

Adrenergic agonists; Cardiomyocyte; Hypertrophy; Signal transduction

Indexed keywords

ALPHA ADRENERGIC RECEPTOR STIMULATING AGENT; ATRIAL NATRIURETIC FACTOR; BETA GALACTOSIDASE; DNA; HOMODIMER; LEUCINE; LUCIFERASE; MESSENGER RNA; OLIGONUCLEOTIDE; PHENYLEPHRINE; PROTEIN C FOS; STRESS ACTIVATED PROTEIN KINASE; TRANSCRIPTION FACTOR AP 1;

ADENOVIRUS; ANIMAL CELL; ARTICLE; BINDING AFFINITY; CELL GROWTH; CELL HYPERPLASIA; CELL ISOLATION; CONTROLLED STUDY; DNA BINDING; ENZYME ACTIVITY; GENE EXPRESSION; HEART MUSCLE CELL; MUTATION; NONHUMAN; NUCLEOTIDE SEQUENCE; ONCOGENE C FOS; PLASMID; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; RAT; TRANSCRIPTION INITIATION; WILD TYPE;

ADRENERGIC ALPHA-AGONISTS; ANIMALS; ATRIAL NATRIURETIC FACTOR; CARDIOMEGALY; DNA; ELECTROPHORETIC MOBILITY SHIFT ASSAY; MYOCYTES, CARDIAC; PHENYLEPHRINE; PROTO-ONCOGENE PROTEINS C-FOS; PROTO-ONCOGENE PROTEINS C-JUN; RATS; RATS, SPRAGUE-DAWLEY; RECEPTORS, ADRENERGIC, ALPHA; RNA, MESSENGER; STIMULATION, CHEMICAL; TRANSCRIPTION FACTOR AP-1; TRANSCRIPTION, GENETIC; TRANSFECTION;

EID: 33745011604     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.cardiores.2006.02.032     Document Type: Article

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