Pannexin 1 deficiency can induce hearing loss

Biochemical and Biophysical Research Communications

Volumn 463, Issue 1-2, 2015, Pages 143-147

  Zhao, Hong Bo ^a   Zhu, Yan ^a   Liang, Chun ^a   Chen, Jin ^a  

^a UNIVERSITY OF KENTUCKY MEDICAL CENTER   (United States)

Author keywords

Caspasae 3; Cell degeneration; Deafness; Gap junction; Inner ear; Pannexin 1

Indexed keywords

CASPASE 3; GAP JUNCTION PROTEIN; GLYCOPROTEIN; PANNEXIN 1; PROTEIN; UNCLASSIFIED DRUG; CASP3 PROTEIN, MOUSE; NERVE PROTEIN; PANX1 PROTEIN, MOUSE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; AUDITORY STIMULATION; BRAIN STEM RESPONSE; CELL DEGENERATION; CELL NUCLEUS; COCHLEA; CONTROLLED STUDY; CORTI ORGAN; DISEASE SEVERITY; DISTORTION PRODUCT OTOACOUSTIC EMISSION; ENZYME ACTIVITY; GAP JUNCTION; GENE DELETION; HAIR CELL; HEARING; HEARING ACUITY; HEARING IMPAIRMENT; IN VIVO STUDY; INNER EAR; MOUSE; NEUROEPITHELIUM; NONHUMAN; OUTER HAIR CELL; PANNEXIN 1 DEFICIENCY; PRIORITY JOURNAL; PROTEIN DEFICIENCY; ANIMAL; DEFICIENCY; DISEASE MODEL; EVOKED BRAIN STEM AUDITORY RESPONSE; GENETICS; HEARING LOSS; KNOCKOUT MOUSE; METABOLISM; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; SPONTANEOUS OTOACOUSTIC EMISSION;

VERTEBRATA;

ANIMALS; APOPTOSIS; CASPASE 3; COCHLEA; CONNEXINS; DISEASE MODELS, ANIMAL; EVOKED POTENTIALS, AUDITORY, BRAIN STEM; HEARING LOSS; MICE; MICE, KNOCKOUT; NERVE TISSUE PROTEINS; OTOACOUSTIC EMISSIONS, SPONTANEOUS;

EID: 84934942164     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2015.05.049     Document Type: Article

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