The methyl-CpG-binding domain (MBD) is crucial for MeCP2’s dysfunction-induced defects in adult newborn neurons

Frontiers in Cellular Neuroscience

Volumn 9, Issue APR, 2015, Pages

  Zhao, Na ^a,b,e   Ma, Dongliang ^a   Leong, Wan Ying ^a   Han, Ju ^a   VanDongen, Antonius ^a,c   Chen, Teng ^b   Goh, Eyleen L K ^a,c,d  

^a DUKE NUS MEDICAL SCHOOL   (Singapore)

^b MEDICAL SCHOOL OF XI AN JIAOTONG UNIVERSITY   (China)

^c NATIONAL UNIVERSITY OF SINGAPORE   (Singapore)

^d KK WOMEN'S AND CHILDREN'S HOSPITAL   (Singapore)

^e NORTHWEST UNIVERSITY OF POLITICAL SCIENCE AND LAW   (China)

Author keywords

Dendrites; Methyl CpG binding domain; Newborn neurons; Rett syndrome; Spontaneous Ca^{2+< sup> oscillations}

Indexed keywords

METHYL CPG BINDING PROTEIN 2; SYNAPSIN I;

ADULT; ANIMAL CELL; ARTICLE; CALCIUM SIGNALING; DENDRITE DEVELOPMENT; DENDRITE LENGTH; DENDRITIC BRANCHING; EMBRYO; FEMALE; GENE EXPRESSION; GENE MUTATION; GENE SILENCING; HIPPOCAMPAL NEURONAL CULTURE; IMMUNOCHEMISTRY; MOUSE; NERVE CHARACTERISTICS AND FUNCTIONS; NERVOUS SYSTEM PARAMETERS; NONHUMAN; RAT; RETT SYNDROME; SYNAPSE FORMATION; SYNAPSE NUMBER; WESTERN BLOTTING;

EID: 84929591206     PISSN: 16625102     EISSN: None     Source Type: Journal    
DOI: 10.3389/fncel.2015.00158     Document Type: Article

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