Complement C1q-induced activation of β-catenin signalling causes hypertensive arterial remodelling

Nature Communications

Volumn 6, Issue , 2015, Pages

  Sumida, Tomokazu ^a,b   Naito, Atsuhiko T ^a,b,c   Nomura, Seitaro ^a   Nakagawa, Akito ^c   Higo, Tomoaki ^c   Hashimoto, Akihito ^c   Okada, Katsuki ^c   Sakai, Taku ^c   Ito, Masamichi ^a   Yamaguchi, Toshihiro ^a   Oka, Toru ^b,c   Akazawa, Hiroshi ^a,b   Lee, Jong Kook ^b,c   Minamino, Tohru ^d   Offermanns, Stefan ^e   Noda, Tetsuo ^f   Botto, Marina ^g   Kobayashi, Yoshio ^h   Morita, Hiroyuki ^a   Manabe, Ichiro ^a   Nagai, Toshio ^h   Shiojima, Ichiro ^b,i   Komuro, Issei ^a,b,c   more..

^a UNIVERSITY OF TOKYO   (Japan)

^b JAPAN SCIENCE AND TECHNOLOGY AGENCY   (Japan)

^c OSAKA UNIVERSITY   (Japan)

^d NIIGATA UNIVERSITY GRADUATE SCHOOL OF MEDICAL AND DENTAL SCIENCES   (Japan)

^e MAX PLANCK INSTITUTE FOR HEART AND LUNG RESEARCH   (Germany)

^f CANCER INSTITUTE   (Japan)

^g IMPERIAL COLLEGE LONDON   (United Kingdom)

^h CHIBA UNIVERSITY   (Japan)

ⁱ KANSAI MEDICAL UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

BETA CATENIN; COMPLEMENT COMPONENT C1Q;

CARDIOVASCULAR DISEASE; CARDIOVASCULAR SYSTEM; DISEASE TREATMENT; HYPERTENSION; MODELING; PATHOLOGY; PROTEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; C1QA GENE; CELL PROLIFERATION; COMPLEMENT ACTIVATION; CONTROLLED STUDY; ELEVATED BLOOD PRESSURE; GENE; GENE DELETION; HUMAN; HUMAN CELL; HYPERTENSION; MACROPHAGE; MALE; MOLECULAR PATHOLOGY; MOUSE; NONHUMAN; VASCULAR ENDOTHELIUM; VASCULAR REMODELING; VASCULAR SMOOTH MUSCLE; WNT SIGNALING PATHWAY; ABDOMINAL AORTA; ANIMAL; C57BL MOUSE; CELL CULTURE; IMMUNOLOGY; KNOCKOUT MOUSE; METABOLISM; PATHOPHYSIOLOGY; PHYSIOLOGY; SMOOTH MUSCLE CELL;

ANIMALS; AORTA, ABDOMINAL; BETA CATENIN; CELL PROLIFERATION; CELLS, CULTURED; COMPLEMENT C1Q; HYPERTENSION; MACROPHAGES; MALE; MICE, INBRED C57BL; MICE, KNOCKOUT; MUSCLE, SMOOTH, VASCULAR; MYOCYTES, SMOOTH MUSCLE; VASCULAR REMODELING;

EID: 84923884270     PISSN: None     EISSN: 20411723     Source Type: Journal    
DOI: 10.1038/ncomms7241     Document Type: Article

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