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Volumn 190, Issue 12, 2014, Pages 1328-1330

Stochastic age-related epigenetic drift in the pathogenesis of idiopathic pulmonary fibrosis

Author keywords

[No Author keywords available]

Indexed keywords

DNA; HISTONE; MICRORNA; TRANSCRIPTOME;

EID: 84919402669     PISSN: 1073449X     EISSN: 15354970     Source Type: Journal    
DOI: 10.1164/rccm.201411-1953ED     Document Type: Editorial
Times cited : (13)

References (16)
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  • 2
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    • Selman M, Pardo A. Revealing the pathogenic and aging-related mechanisms of the enigmatic idiopathic pulmonary fibrosis: An integral model. Am J Respir Crit Care Med 2014;189: 1161-1172.
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  • 4
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    • (2014) Physiology (Bethesda , vol.29 , pp. 177-185
    • Hagood, J.S.1
  • 5
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    • Aging and epigenetic drift: A vicious cycle
    • Issa JP. Aging and epigenetic drift: A vicious cycle. J Clin Invest 2014;124: 24-29.
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    • Issa, J.P.1
  • 6
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    • Age-Associated epigenetic drift: Implications, and a case of epigenetic thrift?
    • Teschendorff AE, West J, Beck S. Age-Associated epigenetic drift: Implications, and a case of epigenetic thrift? Hum Mol Genet 2013; 22: R7-R15.
    • (2013) Hum Mol Genet , vol.22 , pp. R7-R15
    • Teschendorff, A.E.1    West, J.2    Beck, S.3
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    • Idiopathic pulmonary fibrosis: Aberrant recapitulation of developmental programs?
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    • (2008) PLoS Med , vol.5 , pp. e62
    • Selman, M.1    Pardo, A.2    Kaminski, N.3
  • 14
    • 84869196460 scopus 로고    scopus 로고
    • The hedgehog system machinery controls transforming growth factor-b-dependent myofibroblastic differentiation in humans: Involvement in idiopathic pulmonary fibrosis
    • Cigna N, Farrokhi Moshai E, Brayer S, Marchal-Somme J, Wémeau-Stervinou L, Fabre A, Mal H, Lese che G, Dehoux M, Soler P, et al. The hedgehog system machinery controls transforming growth factor-b-dependent myofibroblastic differentiation in humans: Involvement in idiopathic pulmonary fibrosis. Am J Pathol 2012;181: 2126-2137.
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    • Presenilin 2 deficiency causes a mild pulmonary phenotype and no changes in amyloid precursor protein processing but enhances the embryonic lethal phenotype of presenilin 1 deficiency
    • Herreman A, Hartmann D, Annaert W, Saftig P, Craessaerts K, Serneels L, Umans L, Schrijvers V, Checler F, Vanderstichele H, et al. Presenilin 2 deficiency causes a mild pulmonary phenotype and no changes in amyloid precursor protein processing but enhances the embryonic lethal phenotype of presenilin 1 deficiency. Proc Natl Acad Sci USA 1999;96: 11872-11877.
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    • Field cancerization in non-small cell lung cancer: Implications in disease pathogenesis
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    • Kadara, H.1    Wistuba, I.I.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.