Cardiac-specific suppression of NF-κB signaling prevents diabetic cardiomyopathy via inhibition of the renin-angiotensin system

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 307, Issue 7, 2014, Pages H1036-H1045

  Thomas, Candice M ^a,b,c   Yong, Qian Chen ^a,b,c   Rosa, Rodolfo M ^d   Seqqat, Rachid ^a,b,c   Gopal, Shanthi ^c   Casarini, Dulce E ^d   Jones, W Keith ^e   Gupta, Sudhiranjan ^a,b,c   Baker, Kenneth M ^a,b,c   Kumar, Rajesh ^a,b,c  

^a TEXAS A AND M HEALTH SCIENCE CENTER   (United States)

^b SCOTT AND WHITE MEMORIAL HOSPITAL   (United States)

^c VETERANS AFFAIRS MEDICAL CENTER   (United States)

^d FEDERAL UNIVERSITY OF SÃO PAULO   (Brazil)

^e LOYOLA UNIVERSITY CHICAGO   (United States)

Author keywords

Diabetic cardiomyopathy; I B transgenic mice; Nuclear factor B; Renin angiotensin system

Indexed keywords

ADENOSINE TRIPHOSPHATASE; CALCIUM ION; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; PHOSPHOLAMBAN; RYANODINE RECEPTOR; CALCIUM BINDING PROTEIN; SARCOPLASMIC RETICULUM CALCIUM TRANSPORTING ADENOSINE TRIPHOSPHATASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; DIABETIC CARDIOMYOPATHY; ECHOCARDIOGRAPHY; ENDOPLASMIC RETICULUM; ENZYME INHIBITION; HEART FUNCTION; MALE; MOUSE; NONHUMAN; OXIDATIVE STRESS; PROTEIN EXPRESSION; REGULATORY MECHANISM; RENIN ANGIOTENSIN ALDOSTERONE SYSTEM; SIGNAL TRANSDUCTION; STREPTOZOTOCIN-INDUCED DIABETES MELLITUS; ANIMAL; C57BL MOUSE; CALCIUM SIGNALING; DIABETIC CARDIOMYOPATHIES; EXPERIMENTAL DIABETES MELLITUS; GENETICS; HEART MUSCLE; METABOLISM; MUTATION; PATHOPHYSIOLOGY;

ANIMALS; CALCIUM SIGNALING; CALCIUM-BINDING PROTEINS; DIABETES MELLITUS, EXPERIMENTAL; DIABETIC CARDIOMYOPATHIES; MICE; MICE, INBRED C57BL; MUTATION; MYOCARDIUM; NF-KAPPA B; OXIDATIVE STRESS; RENIN-ANGIOTENSIN SYSTEM; RYANODINE RECEPTOR CALCIUM RELEASE CHANNEL; SARCOPLASMIC RETICULUM CALCIUM-TRANSPORTING ATPASES; SIGNAL TRANSDUCTION;

EID: 84907662110     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00340.2014     Document Type: Article

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