Inhibition of histone H3K9 acetylation by anacardic acid can correct the over-expression of Gata4 in the hearts of fetal mice exposed to alcohol during pregnancy

PLoS ONE

Volumn 9, Issue 8, 2014, Pages

  Peng, Chang ^a,c,d   Zhu, Jing ^b,d   Sun, Hui Chao ^a   Huang, Xu Pei ^e   Zhao, Wei An ^a   Zheng, Min ^a   Liu, Ling Juan ^a,b   Tian, Jie ^a  

^a CHILDREN S HOSPITAL OF CHONGQING MEDICAL UNIVERSITY   (China)

^b CHONGQING KEY LABORATORY OF PEDIATRICS   (China)

^c BEIJING UNIVERSITY OF POSTS AND TELECOMMUNICATIONS   (China)

^d Chongqing Intl Science and Technology Cooperation Center for Child Development and Disorders   (China)

^e FLORIDA ATLANTIC UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALCOHOL; ALPHA ACTIN; ANACARDIC ACID; CYCLIC AMP RESPONSIVE ELEMENT BINDING PROTEIN BINDING PROTEIN; E1A ASSOCIATED P300 PROTEIN; HISTONE ACETYLTRANSFERASE; HISTONE ACETYLTRANSFERASE GCN5; HISTONE ACETYLTRANSFERASE PCAF; HISTONE DEACETYLASE; HISTONE H3; HISTONE H3K9; MESSENGER RNA; STEROID RECEPTOR COACTIVATOR 1; TRANSCRIPTION FACTOR GATA 4; TROPONIN T; UNCLASSIFIED DRUG; GATA4 PROTEIN, MOUSE; HISTONE;

ABORTION; ALPHA ACTIN GENE; ALPHA MHC GENE; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; CTNT GENE; DOWN REGULATION; DRUG DOSE COMPARISON; FEMALE; FETUS; FETUS HEART; GENE OVEREXPRESSION; HEART DEVELOPMENT; HISTONE ACETYLATION; MOUSE; NEWBORN; NONHUMAN; OPTIMAL DRUG DOSE; PRENATAL DRUG EXPOSURE; PROMOTER REGION; STILLBIRTH; ACETYLATION; ANIMAL; CHEMISTRY; DNA SEQUENCE; DRUG EFFECTS; EMBRYOLOGY; FETUS DEVELOPMENT; GENE EXPRESSION REGULATION; HEART; INBRED MOUSE STRAIN; METABOLISM; MOLECULAR GENETICS; NUCLEOTIDE SEQUENCE; PREGNANCY; PRENATAL EXPOSURE;

ACETYLATION; ANACARDIC ACIDS; ANIMALS; BASE SEQUENCE; ETHANOL; FEMALE; FETAL DEVELOPMENT; GATA4 TRANSCRIPTION FACTOR; GENE EXPRESSION REGULATION, DEVELOPMENTAL; HEART; HISTONES; MICE, INBRED STRAINS; MOLECULAR SEQUENCE DATA; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; PROMOTER REGIONS, GENETIC; SEQUENCE ANALYSIS, DNA;

EID: 84905449708     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0104135     Document Type: Article

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