-
1
-
-
0033920968
-
A neuropathological analysis of experimental autoimmune encephalomyelitis with predominant brain stem and cerebellar involvement and differences between active and passive induction
-
Muller, D. M., M. P. Pender, and J. M. Greer. 2000. A neuropathological analysis of experimental autoimmune encephalomyelitis with predominant brain stem and cerebellar involvement and differences between active and passive induction. Acta Neuropathol. 100: 174-182. (Pubitemid 30456140)
-
(2000)
Acta Neuropathologica
, vol.100
, Issue.2
, pp. 174-182
-
-
Muller, D.M.1
Pender, M.P.2
Greer, J.M.3
-
2
-
-
12444329705
-
IFN-g determines distinct clinical outcomes in autoimmune encephalomyelitis
-
Wensky, A. K., G. C. Furtado, M. C. Marcondes, S. Chen, D. Manfra, S. A. Lira, D. Zagzag, and J. J. Lafaille. 2005. IFN-g determines distinct clinical outcomes in autoimmune encephalomyelitis. J. Immunol. 174: 1416-1423.
-
(2005)
J. Immunol.
, vol.174
, pp. 1416-1423
-
-
Wensky, A.K.1
Furtado, G.C.2
Marcondes, M.C.3
Chen, S.4
Manfra, D.5
Lira, S.A.6
Zagzag, D.7
Lafaille, J.J.8
-
3
-
-
0033920319
-
Antibody association with a novel model for primary progressive multiple sclerosis: Induction of relapsing-remitting and progressive forms of EAE in H2(s) mouse strains
-
Tsunoda, I., L. Q. Kuang, D. J. Theil, and R. S. Fujinami. 2000. Antibody association with a novel model for primary progressive multiple sclerosis: induction of relapsing-remitting and progressive forms of EAE in H2s mouse strains. Brain Pathol. 10: 402-418. (Pubitemid 30431293)
-
(2000)
Brain Pathology
, vol.10
, Issue.3
, pp. 402-418
-
-
Tsunoda, I.1
Kuang, L.-Q.2
Theil, D.J.3
Fujinami, R.S.4
-
4
-
-
58149159798
-
Regional CNS responses to IFN-g determine lesion localization patterns during EAE pathogenesis
-
Lees, J. R., P. T. Golumbek, J. Sim, D. Dorsey, and J. H. Russell. 2008. Regional CNS responses to IFN-g determine lesion localization patterns during EAE pathogenesis. J. Exp. Med. 205: 2633-2642.
-
(2008)
J. Exp. Med.
, vol.205
, pp. 2633-2642
-
-
Lees, J.R.1
Golumbek, P.T.2
Sim, J.3
Dorsey, D.4
Russell, J.H.5
-
5
-
-
0030029655
-
Immunogenic and encephalitogenic epitope clusters of myelin proteolipid protein
-
Greer, J. M., R. A. Sobel, A. Sette, S. Southwood, M. B. Lees, and V. K. Kuchroo. 1996. Immunogenic and encephalitogenic epitope clusters of myelin proteolipid protein. J. Immunol. 156: 371-379. (Pubitemid 26006985)
-
(1996)
Journal of Immunology
, vol.156
, Issue.1
, pp. 371-379
-
-
Greer, J.M.1
Sobel, R.A.2
Sette, A.3
Southwood, S.4
Lees, M.B.5
Kuchroo, V.K.6
-
6
-
-
0031688565
-
Autoimmunity to myelin oligodendrocyte glycoprotein in rats mimics the spectrum of multiple sclerosis pathology
-
Storch, M. K., A. Stefferl, U. Brehm, R. Weissert, E. Wallström, M. Kerschensteiner, T. Olsson, C. Linington, and H. Lassmann. 1998. Autoimmunity to myelin oligodendrocyte glycoprotein in rats mimics the spectrum of multiple sclerosis pathology. Brain Pathol. 8: 681-694. (Pubitemid 28460927)
-
(1998)
Brain Pathology
, vol.8
, Issue.4
, pp. 681-694
-
-
Storch, M.K.1
Stefferl, A.2
Brehm, U.3
Weissert, R.4
Wallstrom, E.5
Kerschensteiner, M.6
Olsson, T.7
Linington, C.8
Lassmann, H.9
-
7
-
-
67449116625
-
Spontaneous relapsing-remitting EAE in the SJL/J mouse: MOG-reactive transgenic T cells recruit endogenous MOG-specific B cells
-
Pöllinger, B., G. Krishnamoorthy, K. Berer, H. Lassmann, M. R. Bösl, R. Dunn, H. S. Domingues, A. Holz, F. C. Kurschus, and H. Wekerle. 2009. Spontaneous relapsing-remitting EAE in the SJL/J mouse: MOG-reactive transgenic T cells recruit endogenous MOG-specific B cells. J. Exp. Med. 206: 1303-1316.
-
(2009)
J. Exp. Med.
, vol.206
, pp. 1303-1316
-
-
Pöllinger, B.1
Krishnamoorthy, G.2
Berer, K.3
Lassmann, H.4
Bösl, M.R.5
Dunn, R.6
Domingues, H.S.7
Holz, A.8
Kurschus, F.C.9
Wekerle, H.10
-
8
-
-
7244227768
-
T-cell properties determine disease site, clinical presentation, and cellular pathology of experimental autoimmune encephalomyelitis
-
Abromson-Leeman, S., R. Bronson, Y. Luo, M. Berman, R. Leeman, J. Leeman, and M. Dorf. 2004. T-cell properties determine disease site, clinical presentation, and cellular pathology of experimental autoimmune encephalomyelitis. Am. J. Pathol. 165: 1519-1533. (Pubitemid 39435152)
-
(2004)
American Journal of Pathology
, vol.165
, Issue.5
, pp. 1519-1533
-
-
Abromson-Leeman, S.1
Bronson, R.2
Luo, Y.3
Berman, M.4
Leeman, R.5
Leeman, J.6
Dorf, M.7
-
9
-
-
77956103058
-
EAE mediated by a non-IFN-γ/non-IL-17 pathway
-
Kroenke, M. A., S. W. Chensue, and B. M. Segal. 2010. EAE mediated by a non-IFN-γ/non-IL-17 pathway. Eur. J. Immunol. 40: 2340-2348.
-
(2010)
Eur. J. Immunol.
, vol.40
, pp. 2340-2348
-
-
Kroenke, M.A.1
Chensue, S.W.2
Segal, B.M.3
-
12
-
-
0034333272
-
q mice as highly susceptible to MOG-induced EAE with minimal adjuvant requirement
-
q mice as highly susceptible to MOG-induced EAE with minimal adjuvant requirement. J. Neuroimmunol. 111: 23-33.
-
(2000)
J. Neuroimmunol.
, vol.111
, pp. 23-33
-
-
Abdul-Majid, K.B.1
Jirholt, J.2
Stadelmann, C.3
Stefferl, A.4
Kjellén, P.5
Wallström, E.6
Holmdahl, R.7
Lassmann, H.8
Olsson, T.9
Harris, R.A.10
-
13
-
-
0033802454
-
In situ tolerance within the central nervous system as a mechanism for preventing autoimmunity
-
Brabb, T., P. von Dassow, N. Ordonez, B. Schnabel, B. Duke, and J. Goverman. 2000. In situ tolerance within the central nervous system as a mechanism for preventing autoimmunity. J. Exp. Med. 192: 871-880.
-
(2000)
J. Exp. Med.
, vol.192
, pp. 871-880
-
-
Brabb, T.1
Von Dassow, P.2
Ordonez, N.3
Schnabel, B.4
Duke, B.5
Goverman, J.6
-
14
-
-
84877800541
-
The influence of T cell Ig mucin-3 signaling on central nervous system autoimmune disease is determined by the effector function of the pathogenic T cells
-
Lee, S. Y., and J. M. Goverman. 2013. The influence of T cell Ig mucin-3 signaling on central nervous system autoimmune disease is determined by the effector function of the pathogenic T cells. J. Immunol. 190: 4991-4999.
-
(2013)
J. Immunol.
, vol.190
, pp. 4991-4999
-
-
Lee, S.Y.1
Goverman, J.M.2
-
15
-
-
80053321927
-
Neutrophils cascading their way to inflammation
-
Sadik, C. D., N. D. Kim, and A. D. Luster. 2011. Neutrophils cascading their way to inflammation. Trends Immunol. 32: 452-460.
-
(2011)
Trends Immunol.
, vol.32
, pp. 452-460
-
-
Sadik, C.D.1
Kim, N.D.2
Luster, A.D.3
-
16
-
-
35349024347
-
Induction of the genes for Cxcl9 and Cxcl10 is dependent on IFN-γ but shows differential cellular expression in experimental autoimmune encephalomyelitis and by astrocytes and microglia in vitro
-
DOI 10.1002/glia.20587
-
Carter, S. L., M. Müller, P. M. Manders, and I. L. Campbell. 2007. Induction of the genes for Cxcl9 and Cxcl10 is dependent on IFN-γ but shows differential cellular expression in experimental autoimmune encephalomyelitis and by astrocytes and microglia in vitro. Glia 55: 1728-1739. (Pubitemid 47612859)
-
(2007)
GLIA
, vol.55
, Issue.16
, pp. 1728-1739
-
-
Carter, S.L.1
Muller, M.2
Manders, P.M.3
Campbell, I.L.4
-
17
-
-
0033646462
-
Chemokines and chemokine receptors in inflammation of the nervous system: Manifold roles and exquisite regulation
-
Huang, D., Y. Han, M. R. Rani, A. Glabinski, C. Trebst, T. Sørensen, M. Tani, J.Wang, P. Chien, S. O'Bryan, et al. 2000. Chemokines and chemokine receptors in inflammation of the nervous system: manifold roles and exquisite regulation. Immunol. Rev. 177: 52-67.
-
(2000)
Immunol. Rev.
, vol.177
, pp. 52-67
-
-
Huang, D.1
Han, Y.2
Rani, M.R.3
Glabinski, A.4
Trebst, C.5
Sørensen, T.6
Tani, M.7
Wang, J.8
Chien, P.9
O'Bryan, S.10
-
18
-
-
0035811006
-
Biologic consequences of Stat1-independent IFN signaling
-
DOI 10.1073/pnas.111163898
-
Gil, M. P., E. Bohn, A. K. O'Guin, C. V. Ramana, B. Levine, G. R. Stark, H. W. Virgin, and R. D. Schreiber. 2001. Biologic consequences of Stat1-independent IFN signaling. Proc. Natl. Acad. Sci. USA 98: 6680-6685. (Pubitemid 32538276)
-
(2001)
Proceedings of the National Academy of Sciences of the United States of America
, vol.98
, Issue.12
, pp. 6680-6685
-
-
Gil, M.P.1
Bohn, E.2
O'Guin, A.K.3
Ramana, C.V.4
Levine, B.5
Stark, G.R.6
Virgin, H.W.7
Schreiber, R.D.8
-
19
-
-
42249114634
-
+CXC chemokine pathway is essential for the development of central nervous system autoimmune disease
-
+CXC chemokine pathway is essential for the development of central nervous system autoimmune disease. J. Exp. Med. 205: 811-823.
-
(2008)
J. Exp. Med.
, vol.205
, pp. 811-823
-
-
Carlson, T.1
Kroenke, M.2
Rao, P.3
Lane, T.E.4
Segal, B.5
-
20
-
-
0034162486
-
IFN-γ shapes immune invasion of the central nervous system via regulation of chemokines
-
Tran, E. H., E. N. Prince, and T. Owens. 2000. IFN-γ shapes immune invasion of the central nervous system via regulation of chemokines. J. Immunol. 164: 2759-2768. (Pubitemid 30115510)
-
(2000)
Journal of Immunology
, vol.164
, Issue.5
, pp. 2759-2768
-
-
Tran, E.H.1
Prince, E.N.2
Owens, T.3
-
21
-
-
84904268878
-
Site-specific chemokine expression regulates central nervous system inflammation and determines clinical phenotype in autoimmune encephalomyelitis
-
Stoolman, J. S., P. C. Duncker, A. K. Huber, and B. M. Segal. 2014. Site-specific chemokine expression regulates central nervous system inflammation and determines clinical phenotype in autoimmune encephalomyelitis. J. Immunol. 193: 564-570.
-
(2014)
J. Immunol.
, vol.193
, pp. 564-570
-
-
Stoolman, J.S.1
Duncker, P.C.2
Huber, A.K.3
Segal, B.M.4
-
22
-
-
30944452780
-
Therapeutic efficacy of IL-17 neutralization in murine experimental autoimmune encephalomyelitis
-
DOI 10.1016/j.cellimm.2005.11.002, PII S000887490500242X
-
Hofstetter, H. H., S. M. Ibrahim, D. Koczan, N. Kruse, A. Weishaupt, K. V. Toyka, and R. Gold. 2005. Therapeutic efficacy of IL-17 neutralization in murine experimental autoimmune encephalomyelitis. Cell. Immunol. 237: 123-130. (Pubitemid 43117208)
-
(2005)
Cellular Immunology
, vol.237
, Issue.2
, pp. 123-130
-
-
Hofstetter, H.H.1
Ibrahim, S.M.2
Koczan, D.3
Kruse, N.4
Weishaupt, A.5
Toyka, K.V.6
Gold, R.7
-
23
-
-
33745321276
-
IL-17 plays an important role in the development of experimental autoimmune encephalomyelitis
-
Komiyama, Y., S. Nakae, T. Matsuki, A. Nambu, H. Ishigame, S. Kakuta, K. Sudo, and Y. Iwakura. 2006. IL-17 plays an important role in the development of experimental autoimmune encephalomyelitis. J. Immunol. 177: 566-573. (Pubitemid 43939170)
-
(2006)
Journal of Immunology
, vol.177
, Issue.1
, pp. 566-573
-
-
Komiyama, Y.1
Nakae, S.2
Matsuki, T.3
Nambu, A.4
Ishigame, H.5
Kakuta, S.6
Sudo, K.7
Iwakura, Y.8
-
24
-
-
64849085127
-
IL-17 signaling-independent central nervous system autoimmunity is negatively regulated by TGF-β
-
Gonzalez-García, I., Y. Zhao, S. Ju, Q. Gu, L. Liu, J. K. Kolls, and B. Lu. 2009. IL-17 signaling-independent central nervous system autoimmunity is negatively regulated by TGF-β. J. Immunol. 182: 2665-2671.
-
(2009)
J. Immunol.
, vol.182
, pp. 2665-2671
-
-
Gonzalez-García, I.1
Zhao, Y.2
Ju, S.3
Gu, Q.4
Liu, L.5
Kolls, J.K.6
Lu, B.7
-
25
-
-
77952763410
-
IL-17RC is required for IL-17A- and IL-17F-dependent signaling and the pathogenesis of experimental autoimmune encephalomyelitis
-
Hu, Y., N. Ota, I. Peng, C. J. Refino, D. M. Danilenko, P. Caplazi, and W. Ouyang. 2010. IL-17RC is required for IL-17A- and IL-17F-dependent signaling and the pathogenesis of experimental autoimmune encephalomyelitis. J. Immunol. 184: 4307-4316.
-
(2010)
J. Immunol.
, vol.184
, pp. 4307-4316
-
-
Hu, Y.1
Ota, N.2
Peng, I.3
Refino, C.J.4
Danilenko, D.M.5
Caplazi, P.6
Ouyang, W.7
-
26
-
-
61749093508
-
IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice
-
Haak, S., A. L. Croxford, K. Kreymborg, F. L. Heppner, S. Pouly, B. Becher, and A. Waisman. 2009. IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice. J. Clin. Invest. 119: 61-69.
-
(2009)
J. Clin. Invest.
, vol.119
, pp. 61-69
-
-
Haak, S.1
Croxford, A.L.2
Kreymborg, K.3
Heppner, F.L.4
Pouly, S.5
Becher, B.6
Waisman, A.7
-
27
-
-
79957915063
-
IL-23 modulated myelin-specific T cells induce EAE via an IFNγ driven, IL-17 independent pathway
-
Kroenke, M. A., and B. M. Segal. 2011. IL-23 modulated myelin-specific T cells induce EAE via an IFNγ driven, IL-17 independent pathway. Brain Behav. Immun. 25: 932-937.
-
(2011)
Brain Behav. Immun.
, vol.25
, pp. 932-937
-
-
Kroenke, M.A.1
Segal, B.M.2
-
28
-
-
0344389016
-
Interleukin-17 mRNA expression in blood and CSF mononuclear cells is augmented in multiple sclerosis
-
Matusevicius, D., P. Kivisäkk, B. He, N. Kostulas, V. Ozenci, S. Fredrikson, and H. Link. 1999. Interleukin-17 mRNA expression in blood and CSF mononuclear cells is augmented in multiple sclerosis. Mult. Scler. 5: 101-104.
-
(1999)
Mult. Scler.
, vol.5
, pp. 101-104
-
-
Matusevicius, D.1
Kivisäkk, P.2
He, B.3
Kostulas, N.4
Ozenci, V.5
Fredrikson, S.6
Link, H.7
-
29
-
-
12744274563
-
The levels of chemokines CXCL8, CCL2 and CCL5 in multiple sclerosis patients are linked to the activity of the disease
-
Bartosik-Psujek, H., and Z. Stelmasiak. 2005. The levels of chemokines CXCL8, CCL2 and CCL5 in multiple sclerosis patients are linked to the activity of the disease. Eur. J. Neurol. 12: 49-54.
-
(2005)
Eur. J. Neurol.
, vol.12
, pp. 49-54
-
-
Bartosik-Psujek, H.1
Stelmasiak, Z.2
-
30
-
-
84904250896
-
ECTRIMS abstract: Positive proof of concept of AIN457, an antibody against interleukin-17A, in relapsing-remitting multiple sclerosis
-
AIN457 for MS Study Group (Abstr.) 513
-
Havrdová, E., A. Belova, A. Goloborodko, A. Tisserant, I. Jones, H. Garren, and D. Johns; AIN457 for MS Study Group. 2012. ECTRIMS abstract: Positive proof of concept of AIN457, an antibody against interleukin-17A, in relapsing-remitting multiple sclerosis. Mult. Scler. J. 18: S4 (Abstr.) 513.
-
(2012)
Mult. Scler. J.
, vol.18
-
-
Havrdová, E.1
Belova, A.2
Goloborodko, A.3
Tisserant, A.4
Jones, I.5
Garren, H.6
Johns, D.7
-
31
-
-
76349099319
-
Younger children with MS have a distinct CSF inflammatory profile at disease onset
-
US Network of Pediatric MS Centers of Excellence
-
Chabas, D., J. Ness, A. Belman, E. A. Yeh, N. Kuntz, M. P. Gorman, J. B. Strober, I. De Kouchkovsky, C. McCulloch, T. Chitnis, et al; US Network of Pediatric MS Centers of Excellence. 2010. Younger children with MS have a distinct CSF inflammatory profile at disease onset. Neurology 74: 399-405.
-
(2010)
Neurology
, vol.74
, pp. 399-405
-
-
Chabas, D.1
Ness, J.2
Belman, A.3
Yeh, E.A.4
Kuntz, N.5
Gorman, M.P.6
Strober, J.B.7
De Kouchkovsky, I.8
McCulloch, C.9
Chitnis, T.10
-
32
-
-
84856804751
-
Neutrophils in multiple sclerosis are characterized by a primed phenotype
-
Naegele, M., K. Tillack, S. Reinhardt, S. Schippling, R. Martin, and M. Sospedra. 2012. Neutrophils in multiple sclerosis are characterized by a primed phenotype. J. Neuroimmunol. 242: 60-71.
-
(2012)
J. Neuroimmunol.
, vol.242
, pp. 60-71
-
-
Naegele, M.1
Tillack, K.2
Reinhardt, S.3
Schippling, S.4
Martin, R.5
Sospedra, M.6
-
33
-
-
0034576841
-
Peripheral blood stem cell transplantation in multiple sclerosis with busulfan and cyclophosphamide conditioning: Report of toxicity and immunological monitoring
-
Openshaw, H., B. T. Lund, A. Kashyap, R. Atkinson, I. Sniecinski, L. P. Weiner, and S. Forman. 2000. Peripheral blood stem cell transplantation in multiple sclerosis with busulfan and cyclophosphamide conditioning: report of toxicity and immunological monitoring. Biol. Blood Marrow Transplant. 6: 563-575. Diseases Stroke
-
(2000)
Biol. Blood Marrow Transplant.
, vol.6
, pp. 563-575
-
-
Openshaw, H.1
Lund, B.T.2
Kashyap, A.3
Atkinson, R.4
Sniecinski, I.5
Weiner, L.P.6
Forman, S.7
|