Evidence that the lung adenocarcinoma EML4-ALK fusion gene is not caused by exposure to secondhand tobacco smoke during childhood

Cancer Epidemiology Biomarkers and Prevention

Volumn 23, Issue 7, 2014, Pages 1432-1434

  Ryan, Bríd M ^a   Wang, Yi ^b,e   Jen, Jin ^b,c   Yi, Eunhee S ^b   Olivo Marston, Susan ^d   Yang, Ping ^b   Harris, Curtis C ^a  

^a NATIONAL CANCER INSTITUTE   (United States)

^b Division of Epidemiology Department of Health Sciences Research ^*  (United States)

^c MAYO CLINIC   (United States)

^d OHIO STATE UNIVERSITY   (United States)

^e WENZHOU MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

ARTICLE; DNA DAMAGE; EML4 ALK FUSION GENE; ENVIRONMENTAL EXPOSURE; FEMALE; FUSION GENE; GENE FREQUENCY; GENE LOCUS; HUMAN; LUNG ADENOCARCINOMA; MAJOR CLINICAL STUDY; MALE; PASSIVE SMOKING; PRIORITY JOURNAL; ADENOCARCINOMA; ADULT; ADVERSE EFFECTS; CHILD; FLUORESCENCE IN SITU HYBRIDIZATION; GENETICS; LUNG NEOPLASMS;

EML4-ALK FUSION PROTEIN, HUMAN; ONCOPROTEIN; PASSIVE SMOKING;

ADENOCARCINOMA; ADULT; CHILD; FEMALE; HUMANS; IN SITU HYBRIDIZATION, FLUORESCENCE; LUNG NEOPLASMS; MALE; ONCOGENE PROTEINS, FUSION; TOBACCO SMOKE POLLUTION;

EID: 84903975969     PISSN: 10559965     EISSN: None     Source Type: Journal    
DOI: 10.1158/1055-9965.EPI-14-0224     Document Type: Article

References (8)

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