GLUT1 deficiency in cardiomyocytes does not accelerate the transition from compensated hypertrophy to heart failure

Journal of Molecular and Cellular Cardiology

Volumn 72, Issue , 2014, Pages 95-103

  Pereira, Renata O ^a,b   Wende, Adam R ^a,c   Olsen, Curtis ^a   Soto, Jamie ^a,b   Rawlings, Tenley ^a   Zhu, Yi ^a   Riehle, Christian ^a,b   Abel, E Dale ^a,b  

^a UNIVERSITY OF UTAH SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF IOWA   (United States)

^c University of Alabama at Birmingham   (United States)

Author keywords

Cardiac hypertrophy; Cardiac metabolism; Glucose transport

Indexed keywords

GLUCOSE TRANSPORTER 1; GLUCOSE; SLC2A1 PROTEIN, MOUSE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTA CONSTRICTION; ARTICLE; CAPILLARY DENSITY; CONTROLLED STUDY; FATTY ACID OXIDATION; GLUCOSE OXIDATION; GLUCOSE TRANSPORT; GLUCOSE UTILIZATION; GLYCOLYSIS; HEART FAILURE; HEART HYPERTROPHY; HEART MITOCHONDRION; HEART MUSCLE CELL; HEART MUSCLE FIBROSIS; HEART VENTRICLE OVERLOAD; HEMODYNAMIC STRESS; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; ANIMAL; C57BL MOUSE; DEFICIENCY; GENE EXPRESSION; GENETICS; HEART CONTRACTION; HEART LEFT VENTRICLE HYPERTROPHY; HEART MUSCLE; HEART OUTPUT; KNOCKOUT MOUSE; METABOLISM; ORGAN CULTURE TECHNIQUE; OXYGEN CONSUMPTION; PATHOLOGY; PATHOPHYSIOLOGY; PRIMARY CELL CULTURE; TRANSPORT AT THE CELLULAR LEVEL;

ANIMALS; BIOLOGICAL TRANSPORT; CARDIAC OUTPUT; GENE EXPRESSION; GLUCOSE; GLUCOSE TRANSPORTER TYPE 1; HEART FAILURE; HYPERTROPHY, LEFT VENTRICULAR; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MITOCHONDRIA, HEART; MYOCARDIAL CONTRACTION; MYOCARDIUM; MYOCYTES, CARDIAC; ORGAN CULTURE TECHNIQUES; OXYGEN CONSUMPTION; PRIMARY CELL CULTURE;

EID: 84901232068     PISSN: 00222828     EISSN: 10958584     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2014.02.011     Document Type: Article

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