Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 306, Issue 8, 2014, Pages

  Collins, Helen E ^a   He, Lan ^b   Zou, Luyun ^a   Qu, Jing ^c   Zhou, Lufang ^c   Litovsky, Silvio H ^a   Yang, Qinglin ^b   Young, Martin E ^c   Marchase, Richard B ^d   Chatham, John C ^a  

^a University of Alabama at Birmingham   (United States)

^b UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

^c University of Alabama at Birmingham   (United States)

^d UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

Author keywords

Cardiomyocytes; ER stress; Mitochondria; STIM1

Indexed keywords

CALCIUM; CCAAT ENHANCER BINDING PROTEIN; STROMAL INTERACTION MOLECULE 1;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM TRANSPORT; ECHOCARDIOGRAPHY; ENDOPLASMIC RETICULUM; ENDOPLASMIC RETICULUM STRESS; HEART; HEART FUNCTION; HEART MUSCLE CELL; HEART MUSCLE FIBROSIS; HOMEOSTASIS; IMMUNOBLOTTING; INFLAMMATORY INFILTRATE; MALE; MITOCHONDRION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; TRANSMISSION ELECTRON MICROSCOPY;

CARDIOMYOCYTES; ER STRESS; MITOCHONDRIA; STIM1; STORE-OPERATED CA2+ ENTRY;

ANIMALS; CARDIOMYOPATHY, DILATED; ENDOPLASMIC RETICULUM; ENDOPLASMIC RETICULUM STRESS; HEART; HOMEOSTASIS; MALE; MEMBRANE GLYCOPROTEINS; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MICE, TRANSGENIC; MITOCHONDRIA, HEART; MYOCYTES, CARDIAC; VENTRICULAR FUNCTION, LEFT;

EID: 84900824214     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00075.2014     Document Type: Article

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