Kir6.2 limits Ca2+ overload and mitochondrial oscillations of ventricular myocytes in response to metabolic stress

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 305, Issue 10, 2013, Pages

  Storey, Nina M ^a   Stratton, Rebecca C ^a   Rainbow, Richard D ^a   Standen, Nicholas B ^a   Lodwick, David ^a  

^a UNIVERSITY OF LEICESTER   (United Kingdom)

Author keywords

Calcium; Cardioprotection; Ischemiareperfusion injury; Mitochondrial permeability transition

Indexed keywords

ADENOSINE TRIPHOSPHATE; ADENOVIRUS VECTOR; CALCIUM ION; GLIBENCLAMIDE; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR6.2; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; SHORT HAIRPIN RNA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CALCIUM OVERLOAD; CELL CULTURE; CELL ISOLATION; CELL SURVIVAL; CONTROLLED STUDY; GENE SILENCING; HEART MITOCHONDRION; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART MUSCLE ISCHEMIA; HEART PROTECTION; INCUBATION TIME; ISCHEMIC PRECONDITIONING; MALE; METABOLIC INHIBITION; METABOLIC STRESS; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRIAL PERMEABILITY; NONHUMAN; OSCILLATION; OXIDATIVE STRESS; POTASSIUM CONDUCTANCE; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; REPERFUSION INJURY; RNA INTERFERENCE; SIGNAL TRANSDUCTION; WESTERN BLOTTING;

EID: 84887604980     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00540.2013     Document Type: Article

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