Preconditioning protects by inhibiting the mitochondrial permeability transition

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 287, Issue 2 56-2, 2004, Pages

  Hausenloy, Derek J ^a   Yellon, Derek M ^a   Mani Babu, Siva ^a   Duchen, Michael R ^b  

^a UNIVERSITY COLLEGE HOSPITAL   (United Kingdom)

^b UNIVERSITY COLLEGE LONDON   (United Kingdom)

Author keywords

Ischemia reperfusion; Myocardial preconditioning; Oxidative stress

Indexed keywords

5 HYDROXYDECANOIC ACID; CLAMIKALANT; CYCLOSPORIN A; DIAZOXIDE; GLIBENCLAMIDE; NICORANDIL; POTASSIUM CHANNEL BLOCKING AGENT; RHODAMINE; TETRAMETHYLRHODAMINE METHYL ESTER; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CELL DEATH; CONTROLLED STUDY; HEART INFARCTION PREVENTION; HEART MUSCLE CELL; ILLUMINATION; ISCHEMIA; MALE; MITOCHONDRION; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; RELIABILITY; REPERFUSION; REPRODUCIBILITY;

ANIMALS; CELL HYPOXIA; DIAZOXIDE; FLUORESCENCE; INTRACELLULAR MEMBRANES; ISCHEMIC PRECONDITIONING, MYOCARDIAL; LASERS; MALE; MEMBRANE POTENTIALS; MICROSCOPY, CONFOCAL; MITOCHONDRIA, HEART; MYOCYTES, CARDIAC; NICORANDIL; OXIDATIVE STRESS; PERMEABILITY; RATS; RATS, SPRAGUE-DAWLEY; RHODAMINES;

EID: 3242733040     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00678.2003     Document Type: Article

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