Disruption of the complement anaphylatoxin receptor C5L2 exacerbates inflammation in allergic contact dermatitis

Journal of Immunology

Volumn 191, Issue 8, 2013, Pages 4001-4009

  Wang, Ruobing ^a,b   Lu, Bao ^a   Gerard, Craig ^a,b   Gerard, Norma P ^a,b,c  

^a BOSTON CHILDREN S HOSPITAL   (United States)

^b HARVARD MEDICAL SCHOOL   (United States)

^c BETH ISRAEL DEACONESS MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

COMPLEMENT COMPONENT C5A RECEPTOR; COMPLEMENT RECEPTOR; CXCL1 CHEMOKINE; CXCL2 CHEMOKINE; GAMMA INTERFERON; INTERLEUKIN 10; INTERLEUKIN 12P40; INTERLEUKIN 17; INTERLEUKIN 6; MESSENGER RNA; MONOCYTE CHEMOTACTIC PROTEIN 1; OXAZOLONE; PROTEIN C5L2; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CCL2 GENE; CELL TRANSPORT; CONTACT SENSITIVITY; CONTROLLED STUDY; CXCL1 GENE; CXCL2 GENE; DISEASE EXACERBATION; FEMALE; GENE EXPRESSION; IL6 GENE; INFLAMMATORY CELL; MOUSE; NONHUMAN; PHARMACOLOGICAL BLOCKING; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; QUANTITATIVE ANALYSIS; SKIN ALLERGY;

ANIMALS; ANTIBODIES, MONOCLONAL; ARRESTINS; COMPLEMENT C5A; DERMATITIS, ALLERGIC CONTACT; FEMALE; INFLAMMATION; LYMPH NODES; MICE; MICE, INBRED BALB C; MICE, KNOCKOUT; RECEPTOR, ANAPHYLATOXIN C5A; RECEPTORS, CHEMOKINE; SIGNAL TRANSDUCTION;

EID: 84885457010     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1301626     Document Type: Article

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