-
1
-
-
33947239659
-
The failing heart-an engine out of fuel
-
Neubauer S. The failing heart-an engine out of fuel. NEngl J Med 2007, 356:1140-1151.
-
(2007)
NEngl J Med
, vol.356
, pp. 1140-1151
-
-
Neubauer, S.1
-
2
-
-
33746033433
-
Outcome of heart failure with preserved ejection fraction in a population-based study
-
Bhatia R.S., Tu J.V., Lee D.S., et al. Outcome of heart failure with preserved ejection fraction in a population-based study. NEngl J Med 2006, 355:260-269.
-
(2006)
NEngl J Med
, vol.355
, pp. 260-269
-
-
Bhatia, R.S.1
Tu, J.V.2
Lee, D.S.3
-
4
-
-
33746088001
-
Trends in prevalence and outcome of heart failure with preserved ejection fraction
-
Owan T.E., Hodge D.O., Herges R.M., Jacobsen S.J., Roger V.L., Redfield M.M. Trends in prevalence and outcome of heart failure with preserved ejection fraction. NEngl J Med 2006, 355:251-259.
-
(2006)
NEngl J Med
, vol.355
, pp. 251-259
-
-
Owan, T.E.1
Hodge, D.O.2
Herges, R.M.3
Jacobsen, S.J.4
Roger, V.L.5
Redfield, M.M.6
-
5
-
-
73249124082
-
Left ventricular hypertrophy increases cardiovascular risk independently of in-office and out-of-office blood pressure values
-
Bombelli M., Facchetti R., Carugo S., et al. Left ventricular hypertrophy increases cardiovascular risk independently of in-office and out-of-office blood pressure values. JHypertens 2009, 27:2458-2464.
-
(2009)
JHypertens
, vol.27
, pp. 2458-2464
-
-
Bombelli, M.1
Facchetti, R.2
Carugo, S.3
-
6
-
-
0034713820
-
Left ventricular hypertrophy
-
Lorell B.H., Carabello B.A. Left ventricular hypertrophy. Circulation 2000, 102:470-479.
-
(2000)
Circulation
, vol.102
, pp. 470-479
-
-
Lorell, B.H.1
Carabello, B.A.2
-
8
-
-
0034137229
-
Genetic contributions to left ventricular hypertrophy
-
Arnett D. Genetic contributions to left ventricular hypertrophy. Curr Hypertens Rep 2000, 2:50-55.
-
(2000)
Curr Hypertens Rep
, vol.2
, pp. 50-55
-
-
Arnett, D.1
-
9
-
-
67649694817
-
Novel genetic variants contributing to left ventricular hypertrophy: the HyperGEN study
-
Arnett D.K., Devereux R.B., Rao D.C., et al. Novel genetic variants contributing to left ventricular hypertrophy: the HyperGEN study. JHypertens 2009, 27:1585-1593.
-
(2009)
JHypertens
, vol.27
, pp. 1585-1593
-
-
Arnett, D.K.1
Devereux, R.B.2
Rao, D.C.3
-
10
-
-
26944484376
-
Distinct QTLs are linked to cardiac left ventricular mass in a sex-specific manner in a normotensive inbred rat intercross
-
Llamas B., Jiang Z., Rainville M.L., Picard S., Deschepper C. Distinct QTLs are linked to cardiac left ventricular mass in a sex-specific manner in a normotensive inbred rat intercross. Mammalian Genome 2005, 16:700-711.
-
(2005)
Mammalian Genome
, vol.16
, pp. 700-711
-
-
Llamas, B.1
Jiang, Z.2
Rainville, M.L.3
Picard, S.4
Deschepper, C.5
-
11
-
-
67650532164
-
Cardiac pathways distinguish two epistatic modules enacting BP quantitative trait loci and candidate gene analysis
-
Chauvet C., Menard A., Tremblay J., et al. Cardiac pathways distinguish two epistatic modules enacting BP quantitative trait loci and candidate gene analysis. Hypertens Res 2009, 32:631-637.
-
(2009)
Hypertens Res
, vol.32
, pp. 631-637
-
-
Chauvet, C.1
Menard, A.2
Tremblay, J.3
-
12
-
-
58149136321
-
Distinct genomic replacements from Lewis correct diastolic dysfunction, attenuate hypertension, and reduce left ventricular hypertrophy in Dahl salt-sensitive rats
-
Deng A.Y., Nattel S., Shi Y., et al. Distinct genomic replacements from Lewis correct diastolic dysfunction, attenuate hypertension, and reduce left ventricular hypertrophy in Dahl salt-sensitive rats. JHypertens 2008, 26:1935-1943.
-
(2008)
JHypertens
, vol.26
, pp. 1935-1943
-
-
Deng, A.Y.1
Nattel, S.2
Shi, Y.3
-
13
-
-
0033974788
-
Development of different phenotypes of hypertensive heart failure: systolic versus diastolic failure in Dahl salt-sensitive rats
-
Doi R., Masuyama T., Yamamoto K., et al. Development of different phenotypes of hypertensive heart failure: systolic versus diastolic failure in Dahl salt-sensitive rats. JHypertens 2000, 18:111-120.
-
(2000)
JHypertens
, vol.18
, pp. 111-120
-
-
Doi, R.1
Masuyama, T.2
Yamamoto, K.3
-
14
-
-
33646132038
-
Development of heart failure in chronic hypertensive Dahl rats: focus on heart failure with preserved ejection fraction
-
Klotz S., Hay I., Zhang G., Maurer M., Wang J., Burkhoff D. Development of heart failure in chronic hypertensive Dahl rats: focus on heart failure with preserved ejection fraction. Hypertension 2006, 47:901-911.
-
(2006)
Hypertension
, vol.47
, pp. 901-911
-
-
Klotz, S.1
Hay, I.2
Zhang, G.3
Maurer, M.4
Wang, J.5
Burkhoff, D.6
-
15
-
-
0037108639
-
Outcome of congestive heart failure in elderly persons: influence of left ventricular systolic function: the Cardiovascular Health Study
-
Gottdiener J.S., McClelland R.L., Marshall R., et al. Outcome of congestive heart failure in elderly persons: influence of left ventricular systolic function: the Cardiovascular Health Study. Ann Intern Med 2002, 137:631-639.
-
(2002)
Ann Intern Med
, vol.137
, pp. 631-639
-
-
Gottdiener, J.S.1
McClelland, R.L.2
Marshall, R.3
-
16
-
-
31944435692
-
Epistasis, not numbers, regulates functions of clustered Dahl rat quantitative trait loci applicable to human hypertension
-
Charron S., Duong C., Menard A., et al. Epistasis, not numbers, regulates functions of clustered Dahl rat quantitative trait loci applicable to human hypertension. Hypertension 2005, 46:1300-1308.
-
(2005)
Hypertension
, vol.46
, pp. 1300-1308
-
-
Charron, S.1
Duong, C.2
Menard, A.3
-
17
-
-
42049114663
-
Submegabase resolution of epistatically interacting quantitative trait loci for blood pressure applicable for essential hypertension
-
Chauvet C., Charron S., Menard A., Xiao C., Roy J., Deng A.Y. Submegabase resolution of epistatically interacting quantitative trait loci for blood pressure applicable for essential hypertension. JHypertens 2008, 26:893-901.
-
(2008)
JHypertens
, vol.26
, pp. 893-901
-
-
Chauvet, C.1
Charron, S.2
Menard, A.3
Xiao, C.4
Roy, J.5
Deng, A.Y.6
-
18
-
-
79958767623
-
Alpha-Kinase 2 is a novel candidate gene for inherited hypertension in Dahl rats
-
Chauvet C., Crespo K., Menard A., et al. alpha-Kinase 2 is a novel candidate gene for inherited hypertension in Dahl rats. JHypertens 2011, 29:1320-1326.
-
(2011)
JHypertens
, vol.29
, pp. 1320-1326
-
-
Chauvet, C.1
Crespo, K.2
Menard, A.3
-
19
-
-
7244242357
-
Chemokines in the pathogenesis of vascular disease
-
Charo I.F., Taubman M.B. Chemokines in the pathogenesis of vascular disease. Circ Res 2004, 95:858-866.
-
(2004)
Circ Res
, vol.95
, pp. 858-866
-
-
Charo, I.F.1
Taubman, M.B.2
-
20
-
-
83455202738
-
Novel genes as primary triggers for polygenic hypertension
-
Chauvet C., Menard A., Xiao C., et al. Novel genes as primary triggers for polygenic hypertension. JHypertens 2012, 30:81-86.
-
(2012)
JHypertens
, vol.30
, pp. 81-86
-
-
Chauvet, C.1
Menard, A.2
Xiao, C.3
-
21
-
-
0036178483
-
Structure, function, and inhibition of chemokines
-
Fernandez E.J., Lolis E. Structure, function, and inhibition of chemokines. Annu Rev Pharmacol Toxicol 2012, 42:469-499.
-
(2012)
Annu Rev Pharmacol Toxicol
, vol.42
, pp. 469-499
-
-
Fernandez, E.J.1
Lolis, E.2
-
23
-
-
0035076642
-
Involvement of chemokine receptor 2 and its ligand, monocyte chemoattractant protein-1, in the development of atherosclerosis: lessons from knockout mice
-
Peters W., Charo I.F. Involvement of chemokine receptor 2 and its ligand, monocyte chemoattractant protein-1, in the development of atherosclerosis: lessons from knockout mice. Curr Opin Lipidol 2001, 12:175-180.
-
(2001)
Curr Opin Lipidol
, vol.12
, pp. 175-180
-
-
Peters, W.1
Charo, I.F.2
-
24
-
-
78649489009
-
Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci
-
Franke A., McGovern D.P.B., Barrett J.C., et al. Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci. Nat Genet 2010, 42:1118-1125.
-
(2010)
Nat Genet
, vol.42
, pp. 1118-1125
-
-
Franke, A.1
McGovern, D.P.B.2
Barrett, J.C.3
-
25
-
-
0034898394
-
Involvement of polymorphisms in the chemokine system in the susceptibility for coronary artery disease (CAD): coincidence of elevated Lp(a) and MCP-1 -2518 G/G genotype in CAD patients
-
Szalai C., Duba J., Prohászka Z., et al. Involvement of polymorphisms in the chemokine system in the susceptibility for coronary artery disease (CAD): coincidence of elevated Lp(a) and MCP-1 -2518 G/G genotype in CAD patients. Atherosclerosis 2001, 158:233-239.
-
(2001)
Atherosclerosis
, vol.158
, pp. 233-239
-
-
Szalai, C.1
Duba, J.2
Prohászka, Z.3
-
26
-
-
0032572719
-
Decreased lesion formation in CCR2-/- mice reveals a role for chemokines in the initiation of atherosclerosis
-
Boring L., Gosling J., Cleary M., Charo I.F. Decreased lesion formation in CCR2-/- mice reveals a role for chemokines in the initiation of atherosclerosis. Nature 1998, 394:894-897.
-
(1998)
Nature
, vol.394
, pp. 894-897
-
-
Boring, L.1
Gosling, J.2
Cleary, M.3
Charo, I.F.4
-
27
-
-
0037674323
-
Mutation of threonine 766 in the epidermal growth factor receptor reveals a hotspot for resistance formation against selective tyrosine kinase inhibitors
-
Blencke S., Ullrich A., Daub H. Mutation of threonine 766 in the epidermal growth factor receptor reveals a hotspot for resistance formation against selective tyrosine kinase inhibitors. JBiol Chem 2003, 278:15435-15440.
-
(2003)
JBiol Chem
, vol.278
, pp. 15435-15440
-
-
Blencke, S.1
Ullrich, A.2
Daub, H.3
-
28
-
-
69349084655
-
Evolution of the Schlafen genes, a gene family associated with embryonic lethality, meiotic drive, immune processes and orthopoxvirus virulence
-
Bustos O., Naik S., Ayers G., et al. Evolution of the Schlafen genes, a gene family associated with embryonic lethality, meiotic drive, immune processes and orthopoxvirus virulence. Gene 2009, 447:1-11.
-
(2009)
Gene
, vol.447
, pp. 1-11
-
-
Bustos, O.1
Naik, S.2
Ayers, G.3
|