Lack of caspase-3 attenuates immobilization-induced muscle atrophy and loss of tension generation along with mitigation of apoptosis and inflammation

Muscle and Nerve

Volumn 47, Issue 5, 2013, Pages 711-721

  Zhu, Shimei ^a,b   Nagashima, Michio ^a,b   Khan, Mohammed A S ^a,b   Yasuhara, Shingo ^a,b   Kaneki, Masao ^a,b   Martyn, J A Jeevendra ^a,b  

^a MASSACHUSETTS GENERAL HOSPITAL   (United States)

^b HARVARD MEDICAL SCHOOL   (United States)

Author keywords

Apoptosis; Caspase 3; Immobilization; Inflammation; Skeletal muscle

Indexed keywords

CASPASE 3; MESSENGER RNA; MONOCYTE CHEMOTACTIC PROTEIN 1;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL INFILTRATION; CONTROLLED STUDY; GASTROCNEMIUS MUSCLE; HINDLIMB; IMMOBILIZATION; INFLAMMATION; MACROPHAGE; MALE; MOUSE; MUSCLE ATROPHY; MUSCLE MASS; MUSCLE TWITCH; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SOLEUS MUSCLE; WILD TYPE;

ANIMALS; APOPTOSIS; CASPASE 3; CHEMOKINE CCL2; IMMOBILIZATION; INFLAMMATION; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MUSCLE, SKELETAL; MUSCULAR ATROPHY;

EID: 84876811477     PISSN: 0148639X     EISSN: 10974598     Source Type: Journal    
DOI: 10.1002/mus.23642     Document Type: Article

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