A pleckstrin homology-related domain in SHIP1 mediates membrane localization during Fcgamma receptor-induced phagocytosis
Ming-Lum, A. et al. 2012. A pleckstrin homology-related domain in SHIP1 mediates membrane localization during Fcgamma receptor-induced phagocytosis. FASEB J. 26: 3163-3177.
Inappropriate recruitment and activity by the Src homology region 2 domain-containing phosphatase 1 (SHP1) is responsible for receptor dominance in the SHIP-deficient NK cell
Wahle, J.A. et al. 2007. Inappropriate recruitment and activity by the Src homology region 2 domain-containing phosphatase 1 (SHP1) is responsible for receptor dominance in the SHIP-deficient NK cell. J. Immunol. 179: 8009-8015.
SHIP1 inhibition increases immunoregulatory capacity and triggers apoptosis of hematopoietic cancer cells
Brooks, R. et al. 2010. SHIP1 inhibition increases immunoregulatory capacity and triggers apoptosis of hematopoietic cancer cells. J. Immunol. 184: 3582-3589.
Evidence that inositol polyphosphate 4-phosphatase type II is a tumor suppressor that inhibits PI3K signaling
Gewinner, C. et al. 2009. Evidence that inositol polyphosphate 4-phosphatase type II is a tumor suppressor that inhibits PI3K signaling. Cancer Cell 16: 115-125.
SHIP-1 inhibits CD95/APO-1/Fas-induced apoptosis in primary T lymphocytes and T leukemic cells by promoting CD95 glycosylation independently of its phosphatase activity
Charlier, E. et al. 2010. SHIP-1 inhibits CD95/APO-1/Fas-induced apoptosis in primary T lymphocytes and T leukemic cells by promoting CD95 glycosylation independently of its phosphatase activity. Leukemia 24: 821-832.
Targeted disruption of SHIP leads to hemopoietic perturbations, lung pathology, and a shortened life span
Helgason, C.D. et al. 1998. Targeted disruption of SHIP leads to hemopoietic perturbations, lung pathology, and a shortened life span. Genes Develop. 12: 1610-1620.
Expansion of myeloid suppressor cells in SHIP-deficient mice represses allogeneic T cell responses
Ghansah, T. et al. 2004. Expansion of myeloid suppressor cells in SHIP-deficient mice represses allogeneic T cell responses. J. Immunol. 173: 7324-7330.
SHIP is a negative regulator of growth factor receptor-mediated PKB/Akt activation and myeloid cell survival
Liu, Q. et al. 1999. SHIP is a negative regulator of growth factor receptor-mediated PKB/Akt activation and myeloid cell survival. Genes Develop. 13: 786-791.
Macrophages control the retention and trafficking of B lymphocytes in the splenic marginal zone
Karlsson, M.C. et al. 2003. Macrophages control the retention and trafficking of B lymphocytes in the splenic marginal zone. J. Exp. Med. 198: 333-340.