DLK initiates a transcriptional program that couples apoptotic and regenerative responses to axonal injury

Proceedings of the National Academy of Sciences of the United States of America

Volumn 110, Issue 10, 2013, Pages 4039-4044

  Watkins, Trent A ^a   Wang, Bei ^a   Huntwork Rodriguez, Sarah ^a   Yang, Jing ^a   Jiang, Zhiyu ^a   Eastham Anderson, Jeffrey ^a   Modrusan, Zora ^a   Kaminker, Joshua S ^a   Tessier Lavigne, Marc ^a   Lewcock, Joseph W ^a  

^a GENENTECH INC   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DUAL LEUCINE ZIPPER KINASE; ENZYME; PHOSPHATIDYLINOSITOL 3,4,5 TRISPHOSPHATE 3 PHOSPHATASE; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; AXONAL INJURY; CELL DEGENERATION; CELL PROTECTION; CELL REGENERATION; CELL SURVIVAL; CONTROLLED STUDY; GENE EXPRESSION; MOUSE; NONHUMAN; OPTIC NERVE INJURY; PRIORITY JOURNAL; RETINA GANGLION CELL;

ANIMALS; APOPTOSIS; AXONS; MAP KINASE KINASE KINASES; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; NERVE DEGENERATION; NERVE REGENERATION; OPTIC NERVE INJURIES; PTEN PHOSPHOHYDROLASE; RETINAL GANGLION CELLS; TRANSCRIPTION, GENETIC;

EID: 84874601803     PISSN: 00278424     EISSN: 10916490     Source Type: Journal    
DOI: 10.1073/pnas.1211074110     Document Type: Article

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