-
1
-
-
0036911129
-
Molecular mechanisms of myocardial remodeling. The role of aldosterone
-
Delcayre C and Swynghedauw B: Molecular mechanisms of myocardial remodeling. The role of aldosterone. J Mol Cell Cardiol 34: 1577-1584, 2002.
-
(2002)
J Mol Cell Cardiol
, vol.34
, pp. 1577-1584
-
-
Delcayre, C.1
Swynghedauw, B.2
-
2
-
-
0030657676
-
Extracellular matrix remodeling in heart failure: A role for de novo angiotensin II generation
-
Weber KT: Extracellular matrix remodeling in heart failure: a role for de novo angiotensin II generation. Circulation 96: 4065-4082, 1997.
-
(1997)
Circulation
, vol.96
, pp. 4065-4082
-
-
Weber, K.T.1
-
3
-
-
0030347198
-
Angiotensin-converting enzyme-independent pathways of angiotensin II formation in human tissues and cardiovascular diseases
-
Urata H, Nishimura H, Ganten D and Arakawa K: Angiotensin-converting enzyme-independent pathways of angiotensin II formation in human tissues and cardiovascular diseases. Blood Press Suppl 2: 22-28, 1996.
-
(1996)
Blood Press Suppl
, vol.2
, pp. 22-28
-
-
Urata, H.1
Nishimura, H.2
Ganten, D.3
Arakawa, K.4
-
4
-
-
0025341362
-
Side-to-side slippage of myocytes participates in ventricular wall remodeling acutely after myocardial infarction in rats
-
Olivetti G, Capasso JM, Sonnenblick EH and Anversa P: Side-to-side slippage of myocytes participates in ventricular wall remodeling acutely after myocardial infarction in rats. Circ Res 67: 23-34, 1990.
-
(1990)
Circ Res
, vol.67
, pp. 23-34
-
-
Olivetti, G.1
Capasso, J.M.2
Sonnenblick, E.H.3
Anversa, P.4
-
5
-
-
0034727695
-
Limitation of excessive extracellular matrix turnover may contribute to survival benefit of spironolactone therapy in patients with congestive heart failure: Insights from the randomized aldactone evaluation study (RALES). Rales Investigators
-
Zannad F, Alla F, Dousset B, et al: Limitation of excessive extracellular matrix turnover may contribute to survival benefit of spironolactone therapy in patients with congestive heart failure: insights from the randomized aldactone evaluation study (RALES). Rales Investigators. Circulation 102: 2700-2706, 2000.
-
(2000)
Circulation
, vol.102
, pp. 2700-2706
-
-
Zannad, F.1
Alla, F.2
Dousset, B.3
-
6
-
-
0038351745
-
Aldosterone antagonism in addition to angiotensin-converting enzyme inhibitors in heart failure
-
Bauersachs J and Fraccarollo D: Aldosterone antagonism in addition to angiotensin-converting enzyme inhibitors in heart failure. Minerva Cardioangiol 51: 155-164, 2003.
-
(2003)
Minerva Cardioangiol
, vol.51
, pp. 155-164
-
-
Bauersachs, J.1
Fraccarollo, D.2
-
7
-
-
0037183651
-
Elevated plasma aldosterone levels despite complete inhibition of the vascular angiotensin-converting enzyme in chronic heart failure
-
Jorde UP, Vittorio T, Katz SD, et al: Elevated plasma aldosterone levels despite complete inhibition of the vascular angiotensin-converting enzyme in chronic heart failure. Circulation 106: 1055-1057, 2002.
-
(2002)
Circulation
, vol.106
, pp. 1055-1057
-
-
Jorde, U.P.1
Vittorio, T.2
Katz, S.D.3
-
8
-
-
0033533456
-
Comparison of candesartan, enalapril, and their combination in congestive heart failure: Randomized evaluation of strategies for left ventricular dysfunction (RESOLVD) pilot study. The RESOLVD Pilot Study Investigators
-
McKelvie RS, Yusuf S, Pericak D, et al: Comparison of candesartan, enalapril, and their combination in congestive heart failure: randomized evaluation of strategies for left ventricular dysfunction (RESOLVD) pilot study. The RESOLVD Pilot Study Investigators. Circulation 100: 1056-1064, 1999.
-
(1999)
Circulation
, vol.100
, pp. 1056-1064
-
-
McKelvie, R.S.1
Yusuf, S.2
Pericak, D.3
-
9
-
-
0034192641
-
The spontaneously hypertensive rat as a model of human cardiovascular disease: Evidence of exacerbated cardiopulmonary injury and oxidative stress from inhaled emission particulate matter
-
Kodavanti UP, Schladweiler MC, Ledbetter AD, et al: The spontaneously hypertensive rat as a model of human cardiovascular disease: evidence of exacerbated cardiopulmonary injury and oxidative stress from inhaled emission particulate matter. Toxicol Appl Pharmacol 164: 250-263, 2000.
-
(2000)
Toxicol Appl Pharmacol
, vol.164
, pp. 250-263
-
-
Kodavanti, U.P.1
Schladweiler, M.C.2
Ledbetter, A.D.3
-
10
-
-
0033962321
-
Behavioral validation of the spontaneously hypertensive rat (SHR) as an animal model of attention-deficit/hyperactivity disorder (AD/HD)
-
Sagvolden T: Behavioral validation of the spontaneously hypertensive rat (SHR) as an animal model of attention-deficit/hyperactivity disorder (AD/HD). Neurosci Biobehav Rev 24: 31-39, 2000.
-
(2000)
Neurosci Biobehav Rev
, vol.24
, pp. 31-39
-
-
Sagvolden, T.1
-
11
-
-
34547735173
-
High-glucose-induced regulation of intracellular ANG II synthesis and nuclear redistribution in cardiac myocytes
-
Singh VP, Le B, Bhat VB, et al: High-glucose-induced regulation of intracellular ANG II synthesis and nuclear redistribution in cardiac myocytes. Am J Physiol Heart Circ Physiol 293: H939-H948, 2007.
-
(2007)
Am J Physiol Heart Circ Physiol
, vol.293
-
-
Singh, V.P.1
Le, B.2
Bhat, V.B.3
-
12
-
-
2442680120
-
Proteinuria, a target for renoprotection in patients with type 2 diabetic nephropathy: Lessons from RENAAL
-
de Zeeuw D, Remuzzi G, Parving HH, et al: Proteinuria, a target for renoprotection in patients with type 2 diabetic nephropathy: lessons from RENAAL. Kidney Int 65: 2309-2320, 2004.
-
(2004)
Kidney Int
, vol.65
, pp. 2309-2320
-
-
de Zeeuw, D.1
Remuzzi, G.2
Parving, H.H.3
-
13
-
-
0031601718
-
Interactions of transforming growth factor-beta and angiotensin II in renal fibrosis
-
Border WA and Noble NA: Interactions of transforming growth factor-beta and angiotensin II in renal fibrosis. Hypertension 31: 181-188, 1998.
-
(1998)
Hypertension
, vol.31
, pp. 181-188
-
-
Border, W.A.1
Noble, N.A.2
-
14
-
-
9644283069
-
Inhibition of diabetic nephropathy by a decoy peptide corresponding to the 'handle' region for nonproteolytic activation of prorenin
-
Ichihara A, Hayashi M, Kaneshiro Y, et al: Inhibition of diabetic nephropathy by a decoy peptide corresponding to the 'handle' region for nonproteolytic activation of prorenin. J Clin Invest 114: 1128-1135, 2004.
-
(2004)
J Clin Invest
, vol.114
, pp. 1128-1135
-
-
Ichihara, A.1
Hayashi, M.2
Kaneshiro, Y.3
-
15
-
-
33745824649
-
Prorenin receptor blockade inhibits development of glomerulosclerosis in diabetic angiotensin II type 1a receptor-deficient mice
-
Ichihara A, Suzuki F, Nakagawa T, et al: Prorenin receptor blockade inhibits development of glomerulosclerosis in diabetic angiotensin II type 1a receptor-deficient mice. J Am Soc Nephrol 17: 1950-1961, 2006.
-
(2006)
J Am Soc Nephrol
, vol.17
, pp. 1950-1961
-
-
Ichihara, A.1
Suzuki, F.2
Nakagawa, T.3
-
16
-
-
33644983896
-
Elevated blood pressure and heart rate in human renin receptor transgenic rats
-
Burcklé CA, Jan Danser AH, Müller DN, et al: Elevated blood pressure and heart rate in human renin receptor transgenic rats. Hypertension 47: 552-556, 2006.
-
(2006)
Hypertension
, vol.47
, pp. 552-556
-
-
Burcklé, C.A.1
Jan Danser, A.H.2
Müller, D.N.3
-
17
-
-
0036266596
-
Pivotal role of the renin/prorenin receptor in angiotensin II production and cellular responses to renin
-
Nguyen G, Delarue F, Burcklé C, et al: Pivotal role of the renin/prorenin receptor in angiotensin II production and cellular responses to renin. J Clin Invest 109: 1417-1427, 2002.
-
(2002)
J Clin Invest
, vol.109
, pp. 1417-1427
-
-
Nguyen, G.1
Delarue, F.2
Burcklé, C.3
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