Nuclear PIM1 confers resistance to rapamycin-impaired endothelial proliferation

Biochemical and Biophysical Research Communications

Volumn 429, Issue 1-2, 2012, Pages 24-30

  Walpen, Thomas ^a   Kalus, Ina ^a   Schwaller, Jürg ^b   Peier, Martin A ^a   Battegay, Edouard J ^a,c   Humar, Rok ^a,c  

^a UNIVERSITY HOSPITAL ZURICH   (Switzerland)

^b UNIVERSITY OF BASEL   (Switzerland)

^c UNIVERSITY OF ZURICH   (Switzerland)

Author keywords

Endothelial cells; MTOR; Nucleus; PIM1; Proliferation; Rapamycin

Indexed keywords

MAMMALIAN TARGET OF RAPAMYCIN; PROTEIN KINASE B; PROTEIN KINASE PIM 1; RAPAMYCIN;

ANIMAL CELL; ARTICLE; CARBOXY TERMINAL SEQUENCE; CELL PROLIFERATION; DELETION MUTANT; ENDOTHELIUM CELL; ENZYME ACTIVITY; ENZYME INHIBITION; GROWTH INHIBITION; MOUSE; NONHUMAN; NUCLEAR LOCALIZATION SIGNAL; PRIORITY JOURNAL; PROTEIN EXPRESSION;

ANIMALS; ANTIBIOTICS, ANTINEOPLASTIC; CELL NUCLEUS; CELL PROLIFERATION; CELLS, CULTURED; DRUG RESISTANCE, NEOPLASM; ENDOTHELIAL CELLS; MICE; PROTO-ONCOGENE PROTEINS C-PIM-1; SEQUENCE DELETION; SIROLIMUS;

EID: 84870391145     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2012.10.106     Document Type: Article

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