Lung microenvironment contributes to the resistance of alveolar macrophages to develop tolerance to endotoxin

Critical Care Medicine

Volumn 40, Issue 11, 2012, Pages 2987-2996

  Philippart, François ^a,b   Fitting, Catherine ^a   Cavaillon, Jean Marc ^a  

^a INSTITUT PASTEUR   (France)

^b SAINT JOSEPH HOSPITAL   (France)

Author keywords

B lymphocytes; endotoxin; inflammation; interferon ; tumor necrosis factor

Indexed keywords

ENDOTOXIN; GAMMA INTERFERON; GAMMA INTERFERON RECEPTOR; GRANULOCYTE MACROPHAGE COLONY STIMULATING FACTOR; INTERLEUKIN 15; INTERLEUKIN 18; LIPOPOLYSACCHARIDE; MESSENGER RNA; TUMOR NECROSIS FACTOR;

ADOPTIVE TRANSFER; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; B LYMPHOCYTE; CONTROLLED STUDY; CYTOKINE PRODUCTION; EX VIVO STUDY; EXPERIMENTAL MODEL; GENE EXPRESSION; HOMEOSTASIS; IMMUNOLOGICAL TOLERANCE; IMMUNOMODULATION; IN VITRO STUDY; IN VIVO STUDY; LEUKOCYTE; LUNG; LUNG ALVEOLUS MACROPHAGE; LYMPHOCYTE FUNCTION; MACROPHAGE ACTIVATION; MONOCYTE; MOUSE; NONHUMAN; PERITONEUM MACROPHAGE; PRIORITY JOURNAL; WILD TYPE;

ANIMALS; B-LYMPHOCYTES; BASE SEQUENCE; CELLULAR MICROENVIRONMENT; CYTOKINES; DNA PRIMERS; DRUG TOLERANCE; ENDOTOXINS; INTERFERON-GAMMA; INTERFERONS; LUNG; MACROPHAGES, ALVEOLAR; MICE; MICE, INBRED BALB C; MICE, INBRED C57BL; MICE, KNOCKOUT; POLYMERASE CHAIN REACTION;

EID: 84868199745     PISSN: 00903493     EISSN: 15300293     Source Type: Journal    
DOI: 10.1097/CCM.0b013e31825b8d57     Document Type: Article

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