Glucose depletion activates mmu-miR-466h-5p expression through oxidative stress and inhibition of histone deacetylation

Nucleic Acids Research

Volumn 40, Issue 15, 2012, Pages 7291-7302

  Druz, Aliaksandr ^a,b   Betenbaugh, Michael ^b   Shiloach, Joseph ^a  

^a NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES   (United States)

^b Johns Hopkins University   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GLUCOSE; GLUTATHIONE; HISTONE; HISTONE DEACETYLASE; HISTONE DEACETYLASE 2; HYDROGEN PEROXIDE; MICRORNA; MICRORNA 466H 5P; MICRORNA 669C; REACTIVE OXYGEN METABOLITE; UNCLASSIFIED DRUG;

ACETYLATION; ANIMAL CELL; ARTICLE; CELL LINE; CHROMOSOME 2; CONTROLLED STUDY; DEACETYLATION; DEPLETION; ENZYME ACTIVITY; ENZYME INHIBITION; GENE; GENE ACTIVATION; GENE EXPRESSION; GLUCOSE DEPLETION; GLUCOSE DEPRIVATION; INHIBITION KINETICS; INTRON; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROMOTER REGION; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; RNA ISOLATION; SFMBT2 GENE; WESTERN BLOTTING;

ACETYLATION; ANIMALS; CELL LINE; GENE EXPRESSION REGULATION; GLUCOSE; HISTONE DEACETYLASE INHIBITORS; HISTONE DEACETYLASES; HISTONES; HYDROGEN PEROXIDE; MICE; MICRORNAS; OXIDATIVE STRESS; PROMOTER REGIONS, GENETIC; REACTIVE OXYGEN SPECIES; TRANSCRIPTION FACTORS;

EID: 84867300941     PISSN: 03051048     EISSN: 13624962     Source Type: Journal    
DOI: 10.1093/nar/gks452     Document Type: Article

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