A disintegrin and metalloproteinase 15 contributes to atherosclerosis by mediating endothelial barrier dysfunction via src family kinase activity

Arteriosclerosis, Thrombosis, and Vascular Biology

Volumn 32, Issue 10, 2012, Pages 2444-2451

  Sun, Chongxiu ^b   Wu, MacK H ^a   Lee, Eugene S ^b   Yuan, Sarah Y ^a  

^a UNIVERSITY OF SOUTH FLORIDA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

endothelial dysfunction; inflammation; intercellular junctions; metalloproteinase; vascular permeability

Indexed keywords

APOLIPOPROTEIN E; DISINTEGRIN; HIGH DENSITY LIPOPROTEIN CHOLESTEROL; MATRIX METALLOPROTEINASE 15; PLAKOGLOBIN; PROTEIN KINASE YES; PROTEIN TYROSINE KINASE; SMALL INTERFERING RNA; VASCULAR ENDOTHELIAL CADHERIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; AORTA ATHEROSCLEROSIS; ARTICLE; ATHEROSCLEROTIC PLAQUE; CELL INFILTRATION; CELL JUNCTION; CELL MIGRATION; CONTROLLED STUDY; CYTOPLASM; ENDOTHELIAL DYSFUNCTION; ENZYME ACTIVITY; HUMAN; HUMAN CELL; IN VITRO STUDY; INHIBITION KINETICS; MACROPHAGE; MONOCYTE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN TARGETING; SIGNAL TRANSDUCTION; SITE DIRECTED MUTAGENESIS; SMOOTH MUSCLE FIBER; STRUCTURE ACTIVITY RELATION; UMBILICAL VEIN ENDOTHELIAL CELL;

ADAM PROTEINS; ANIMALS; APOLIPOPROTEINS E; ATHEROSCLEROSIS; CELL MOVEMENT; CELLS, CULTURED; DISEASE MODELS, ANIMAL; ENDOTHELIUM, VASCULAR; HUMANS; MEMBRANE PROTEINS; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MONOCYTES; PROTO-ONCOGENE PROTEINS C-YES; RNA, SMALL INTERFERING; SIGNAL TRANSDUCTION; SRC-FAMILY KINASES;

EID: 84866625277     PISSN: 10795642     EISSN: 15244636     Source Type: Journal    
DOI: 10.1161/ATVBAHA.112.252205     Document Type: Article

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