Is BID required for NOD signalling?

Nature

Volumn 488, Issue 7412, 2012, Pages

  Nachbur, Ueli ^a,b   Vince, James E ^a,b   O Reilly, Lorraine A ^a,b   Strasser, Andreas ^a,b   Silke, John ^a,b  

^a WALTER AND ELIZA HALL INSTITUTE OF MEDICAL RESEARCH   (Australia)

^b UNIVERSITY OF MELBOURNE   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

BIM PROTEIN; GRANULOCYTE COLONY STIMULATING FACTOR; INTERLEUKIN 6; KERATINOCYTE DERIVED CHEMOKINE; MITOGEN ACTIVATED PROTEIN KINASE; MONOCYTE CHEMOTACTIC PROTEIN 1; NUCLEOTIDE BINDING OLIGOMERIZATION DOMAIN PROTEIN;

ANIMAL CELL; ANIMAL TISSUE; CONTROLLED STUDY; CYTOKINE RELEASE; INNATE IMMUNITY; MOUSE; NONHUMAN; NOTE; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ANIMALS; BH3 INTERACTING DOMAIN DEATH AGONIST PROTEIN; EPITHELIAL CELLS; HUMANS; IMMUNITY, INNATE; INFLAMMATION; INTESTINAL MUCOSA;

EID: 84865251377     PISSN: 00280836     EISSN: 14764687     Source Type: Journal    
DOI: 10.1038/nature11366     Document Type: Note

References (7)

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7. Kaufmann, T. et al. The BH3-only protein bid is dispensable for DNA damage-and replicative stress-induced apoptosis or cell-cycle arrest. Cell 129,423-433 (2007).