The cuprizone model: Regional heterogeneity of pathology

APMIS

Volumn 120, Issue 8, 2012, Pages 648-657

  Wergeland, Stig ^a,b   Torkildsen, Øivind ^a,b   Myhr, Kjell Morten ^a,b   Mørk, Sverre Jarl ^a,b   Bø, Lars ^a,b  

^a HAUKELAND UNIVERSITY HOSPITAL   (Norway)

^b UNIVERSITY OF BERGEN   (Norway)

Author keywords

Complement; Cuprizone; Multiple sclerosis

Indexed keywords

CUPRIZONE; PROTEIN NOGO A;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ASTROCYTOSIS; B LYMPHOCYTE; BLOOD BRAIN BARRIER; BRAIN CORTEX; BRAIN TISSUE; CELL LOSS; CELL STRUCTURE; COMPLEMENT ACTIVATION; CONTROLLED STUDY; CORPUS CALLOSUM; DEMYELINATION; DRUG EXPOSURE; FEMALE; GRAY MATTER; HISTOPATHOLOGY; LYMPHOCYTIC INFILTRATION; MACROPHAGE ACTIVATION; MICROGLIA; MOUSE; NONHUMAN; OLIGODENDROGLIA; PATHOPHYSIOLOGY; PRIORITY JOURNAL; REMYELINIZATION; T LYMPHOCYTE; WHITE MATTER;

ANIMALS; CEREBRAL CORTEX; COMPLEMENT SYSTEM PROTEINS; CORPUS CALLOSUM; CUPRIZONE; DEMYELINATING DISEASES; FEMALE; GLIOSIS; HYPOXIA-INDUCIBLE FACTOR 1, ALPHA SUBUNIT; LYMPHOCYTES; MACROPHAGES; MICE; MICE, INBRED C57BL; MICROGLIA; MITOCHONDRIA; MULTIPLE SCLEROSIS; MYELIN SHEATH; OLIGODENDROGLIA;

EID: 84863845255     PISSN: 09034641     EISSN: 16000463     Source Type: Journal    
DOI: 10.1111/j.1600-0463.2012.02882.x     Document Type: Article

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