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Volumn 194, Issue , 2011, Pages 95-111

The kreisler mutation leads to the loss of intrinsically hypoxia-activated spots in the region of the retrotrapezoid nucleus/parafacial respiratory group

Author keywords

C Fos protein; Ex vivo central nervous system preparations; Hypoxia; Mice; Parafacial respiratory group; Retrotrapezoid nucleus

Indexed keywords

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CELL; BRAIN NUCLEUS; BRAIN REGION; BRAIN STEM; BREATHING RATE; CELL HYPOXIA; CELL POPULATION; CONTROLLED STUDY; DISEASE ACTIVITY; DOWN REGULATION; EX VIVO STUDY; GENE; GENE EXPRESSION; GENE LOCATION; GENE LOCUS; GENE MUTATION; GENETIC DISORDER; HOMOZYGOSITY; LATERAL RETICULAR NUCLEUS; LUNG FUNCTION; MEDULLA OBLONGATA; MOUSE; MOUSE MUTANT; NEUROMERE; NEWBORN; NONHUMAN; ONCOGENE C FOS; PARAFACIAL RESPIRATORY CELL; PHOX2B GENE; PONS; PRIORITY JOURNAL; RESPIRATION DEPRESSION; RESPIRATORY TRACT PARAMETERS; RETROTRAPEZOID NUCLEUS; RHOMBOMERE 5; RHOMBOMERE 6; SOLITARY TRACT NUCLEUS; SPINAL CORD; SYNAPTIC TRANSMISSION; WILD TYPE;

EID: 84860424078     PISSN: 03064522     EISSN: 18737544     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2011.07.062     Document Type: Article
Times cited : (13)

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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.