A defect in cell death of macrophages is a conserved feature of nonobese diabetic mouse

Biochemical and Biophysical Research Communications

Volumn 421, Issue 1, 2012, Pages 145-151

  Kim, Hun Sik ^a,b   Park, Jin Mo ^c   Lee, Myung Shik ^d  

^a University of Ulsan   (South Korea)

^b University of Ulsan College of Medicine   (South Korea)

^c San Diego   (United States)

^d Sungkyunkwan University School of Medicine   (South Korea)

Author keywords

Apoptosis; Autoimmune diabetes; Bcl X L; Cell death; Macrophage; NF B

Indexed keywords

4 [4 (4' CHLORO 2 BIPHENYLYLMETHYL) 1 PIPERAZINYL] N [4 [3 DIMETHYLAMINO 1 (PHENYLTHIOMETHYL)PROPYLAMINO] 3 NITROBENZENESULFONYL]BENZAMIDE; ACTIN; ALPHA INTERFERON; GAMMA INTERFERON; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INHIBITOR OF APOPTOSIS PROTEIN; N ACETYL S NITROSOPENICILLAMINE; PEPTIDOGLYCAN; PROTEIN BAD; PROTEIN BAX; PROTEIN BCL 2; PROTEIN BCL XL; STAT1 PROTEIN;

APOPTOSIS; ARTICLE; AUTOIMMUNE DISEASE; CELL DEATH; CYTOTOXICITY; DIABETOGENESIS; EFFECTOR CELL; IMMUNOLOGICAL TOLERANCE; INSULIN DEPENDENT DIABETES MELLITUS; MACROPHAGE; NONOBESE DIABETIC MOUSE; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; PROTEIN EXPRESSION;

ANIMALS; APOPTOSIS; BCL-X PROTEIN; BIPHENYL COMPOUNDS; DIABETES MELLITUS, TYPE 1; HUMANS; MACROPHAGES; MICE; MICE, INBRED C57BL; MICE, INBRED NOD; NF-KAPPA B; NITROPHENOLS; PIPERAZINES; SULFONAMIDES;

ANIMALIA; MUS;

EID: 84860342878     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2012.04.017     Document Type: Article

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