-
1
-
-
0037453125
-
A New Paradigm for the Treatment of Sepsis: Is It Time to Consider Combination Therapy?
-
Cross, A.S., and S.M. Opal. 2003. A new paradigm for the treatment of sepsis: is it time to consider combination therapy? Annals of Internal Medicine 138(6): 502-505. (Pubitemid 38129029)
-
(2003)
Annals of Internal Medicine
, vol.138
, Issue.6
, pp. 502-505
-
-
Cross, A.S.1
Opal, S.M.2
-
2
-
-
34547515353
-
The host response to endotoxin, antilipopolysaccharide strategies, and the management of severe sepsis
-
DOI 10.1016/j.ijmm.2007.03.006, PII S1438422107000434
-
Opal, S.M. 2007. The host response to endotoxin, antilipopolysaccharide strategies, and the management of severe sepsis. International Journal of Medical Microbiology 297(5): 365-377. (Pubitemid 47188050)
-
(2007)
International Journal of Medical Microbiology
, vol.297
, Issue.5
, pp. 365-377
-
-
Opal, S.M.1
-
3
-
-
0037451929
-
The epidemiology of sepsis in the United States from 1979 through 2000
-
DOI 10.1056/NEJMoa022139
-
Martin, G.S., D.M. Mannino, S. Eaton, and M. Moss. 2003. The epidemiology of sepsis in the United States from 1979 through 2000. The New England Journal of Medicine 348(16): 1546-1554. (Pubitemid 36437931)
-
(2003)
New England Journal of Medicine
, vol.348
, Issue.16
, pp. 1546-1554
-
-
Martin, G.S.1
Mannino, D.M.2
Eaton, S.3
Moss, M.4
-
5
-
-
0029147346
-
Incidence, risk factors, and outcome of severe sepsis and septic shock in adults. A multicenter prospective study in intensive care units
-
French ICU Group for Severe Sepsis
-
Brun-Buisson, C., F. Doyon, J. Carlet, P. Dellamonica, F. Gouin, A. Lepoutre, J.C. Mercier, G. Offenstadt, and B. Régnier. 1995. Incidence, risk factors, and outcome of severe sepsis and septic shock in adults. A multicenter prospective study in intensive care units. French ICU Group for Severe Sepsis. JAMA 274(12): 968-974.
-
(1995)
JAMA
, vol.274
, Issue.12
, pp. 968-974
-
-
Brun-Buisson, C.1
Doyon, F.2
Carlet, J.3
Dellamonica, P.4
Gouin, F.5
Lepoutre, A.6
Mercier, J.C.7
Offenstadt, G.8
Régnier, B.9
-
6
-
-
77951204279
-
The response of the host microcirculation to bacterial sepsis: Does the pathogen matter?
-
Legrand, M., E. Klijn, D. Payen, and C. Ince. 2010. The response of the host microcirculation to bacterial sepsis: Does the pathogen matter? Journal of Molecular Medicine 88(2): 127-133.
-
(2010)
Journal of Molecular Medicine
, vol.88
, Issue.2
, pp. 127-133
-
-
Legrand, M.1
Klijn, E.2
Payen, D.3
Ince, C.4
-
7
-
-
0026708196
-
Gram-negative sepsis and the adult respiratory distress syndrome
-
Martin, M.A., and H.J. Silverman. 1992. Gram-negative sepsis and the adult respiratory distress syndrome. Clinical Infectious Diseases 14: 1213-1228.
-
(1992)
Clinical Infectious Diseases
, vol.14
, pp. 1213-1228
-
-
Martin, M.A.1
Silverman, H.J.2
-
8
-
-
0024419385
-
The association of circulating endotoxin with the development of the adult respiratory distress syndrome
-
Parsons, P.E., G.S. Worthen, E.E. Moore, R.M. Tate, and P.M. Henson. 1989. The association of circulating endotoxin with the development of the adult respiratory distress syndrome. The American Review of Respiratory Disease 140: 294-301. (Pubitemid 19218338)
-
(1989)
American Review of Respiratory Disease
, vol.140
, Issue.2 I
, pp. 294-301
-
-
Parsons, P.E.1
Worthen, G.S.2
Moore, E.E.3
Tate, R.M.4
Henson, P.M.5
-
9
-
-
0028888644
-
Clinical risks for development of the acute respiratory distress syndrome
-
Hudson, L.D., J.A. Milberg, D. Anardi, and R.J. Maunder. 1995. Clinical risks for development of the acute respiratory distress syndrome. American Journal of Respiratory and Critical Care Medicine 151(2 Pt 1): 293-301.
-
(1995)
American Journal of Respiratory and Critical Care Medicine
, vol.151
, Issue.2 PART 1
, pp. 293-301
-
-
Hudson, L.D.1
Milberg, J.A.2
Anardi, D.3
Maunder, R.J.4
-
10
-
-
0023028369
-
Shock and tissue injury induced by recombinant human cachectin
-
Tracey, K.J., B. Beutler, S.F. Lowry, J. Merryweather, S. Wolpe, I. W. Milsark, R.J. Hariri, T.J. Fahey 3rd, A. Zentella, J.D. Albert, et al. 1986. Shock and tissue injury induced by recombinant human cachectin. Science 234(4775): 470-474. (Pubitemid 17181429)
-
(1986)
Science
, vol.234
, Issue.4775
, pp. 470-474
-
-
Tracey, K.J.1
Beutler, B.2
Lowry, S.F.3
-
11
-
-
0033538467
-
HMG-1 as a late mediator of endotoxin lethality in mice
-
DOI 10.1126/science.285.5425.248
-
Wang, H., O. Bloom, M. Zhang, J.M. Vishnubhakat, M. Ombrellino, J. Che, A. Frazier, H. Yang, S. Ivanova, L. Borovikova, K.R. Manogue, E. Faist, E. Abraham, J. Andersson, U. Andersson, P.E. Molina, N.N. Abumrad, A. Sama, and K.J. Tracey. 1999. HMG-1 as a late mediator of endotoxin lethality in mice. Science 285(5425): 248-251. (Pubitemid 29329996)
-
(1999)
Science
, vol.285
, Issue.5425
, pp. 248-251
-
-
Wang, H.1
Bloom, O.2
Zhang, M.3
Vishnubhakat, J.M.4
Ombrellino, M.5
Che, J.6
Frazier, A.7
Yang, H.8
Ivanova, S.9
Borovikova, L.10
Manogue, K.R.11
Faist, E.12
Abraham, E.13
Andersson, J.14
Andersson, U.15
Molina, P.E.16
Abumrad, N.N.17
Sama, A.18
Tracey, K.J.19
-
12
-
-
0033574558
-
Increased permeability of primary cultured brain microvessel endothelial cell monolayers following TNF-alpha exposure
-
DOI 10.1016/S0024-3205(99)00139-3, PII S0024320599001393
-
Mark, K.S., and D.W. Miller. 1999. Increased permeability of primary cultured brain microvessel endothelial cell monolayers following TNF-alpha exposure. Life Sciences 64(21): 1941-1953. (Pubitemid 29215195)
-
(1999)
Life Sciences
, vol.64
, Issue.21
, pp. 1941-1953
-
-
Mark, K.S.1
Miller, D.W.2
-
13
-
-
0032005048
-
Tumor necrosis factor-alpha increases sodium and chloride conductance across the tight junction of CACO-2 BBE, a human intestinal epithelial cell line
-
DOI 10.1007/s002329900333
-
Marano, C.W., S.A. Lewis, L.A. Garulacan, A.P. Soler, and J.M. Mullin. 1998. Tumor necrosis factor-alpha increases sodium and chloride conductance across the tight junction of CACO-2 BBE, a human intestinal epithelial cell line. The Journal of Membrane Biology 161(3): 263-274. (Pubitemid 28085051)
-
(1998)
Journal of Membrane Biology
, vol.161
, Issue.3
, pp. 263-274
-
-
Marano, C.W.1
Lewis, S.A.2
Garulacan, L.A.3
Peralta, S.A.4
Mullin, J.M.5
-
14
-
-
0031009086
-
TNF mediates lung leak, but not neutrophil accumulation, in lungs of rats given IL-1 intratracheally
-
Hybertson, B.M., E.K. Jepson, O.J. Cho, J.H. Clarke, Y.M. Lee, and J.E. Repine. 1997. TNF mediates lung leak, but not neutrophil accumulation, in lungs of rats given IL-1 intratracheally. American Journal of Respiratory and Critical Care Medicine 155(6): 1972-1976. (Pubitemid 27256127)
-
(1997)
American Journal of Respiratory and Critical Care Medicine
, vol.155
, Issue.6
, pp. 1972-1976
-
-
Hybertson, B.M.1
Jepson, E.K.2
Cho, O.J.3
Clarke, J.H.4
Lee, Y.M.5
Repine, J.E.6
-
15
-
-
0031912020
-
Bradykinin and tumor necrosis factor-alpha alter albumin transport in vivo: A comparative study
-
DOI 10.1006/mvre.1997.2041
-
Saulpaw, C.E., and W.L. Joyner. 1997. Bradykinin and tumor necrosis factor-alpha alter albumin transport in vivo: a comparative study. Microvascular Research 54(3): 221-232. (Pubitemid 28066514)
-
(1998)
Microvascular Research
, vol.54
, Issue.3
, pp. 221-232
-
-
Saulpaw, C.E.1
Joyner, W.L.2
-
16
-
-
20144380566
-
Persistent elevation of high mobility group box-1 protein (HMGB1) in patients with severe sepsis and septic shock
-
DOI 10.1097/01.CCM.0000155991.88802.4D
-
Sundén-Cullberg, J., A. Norrby-Teglund, A. Rouhiainen, H. Rauvala, G. Herman, K.J. Tracey, M.L. Lee, J. Andersson, L. Tokics, and C.J. Treutiger. 2005. Persistent elevation of high mobility group box-1 protein (HMGB1) in patients with severe sepsis and septic shock. Critical Care Medicine 33(3): 564-573. (Pubitemid 40365558)
-
(2005)
Critical Care Medicine
, vol.33
, Issue.3
, pp. 564-573
-
-
Sunden-Cullberg, J.1
Norrby-Teglund, A.2
Rouhiainen, A.3
Rauvala, H.4
Herman, G.5
Tracey, K.J.6
Lee, M.L.7
Andersson, J.8
Tokics, L.9
Treutiger, C.J.10
-
17
-
-
0034664791
-
HMG-1 as a mediator of acute lung inflammation
-
Abraham, E., J. Arcaroli, A. Carmody, H. Wang, and K.J. Tracey. 2000. HMG-1 as a mediator of acute lung inflammation. Journal of Immunology 165(6): 2950-2954.
-
(2000)
Journal of Immunology
, vol.165
, Issue.6
, pp. 2950-2954
-
-
Abraham, E.1
Arcaroli, J.2
Carmody, A.3
Wang, H.4
Tracey, K.J.5
-
18
-
-
0037769813
-
Inflammation-promoting activity of HMGB1 on human microvascular endothelial cells
-
DOI 10.1182/blood-2002-05-1300
-
Fiuza, C., M. Bustin, S. Talwar, M. Tropea, E. Gerstenberger, J.H. Shelhamer, and A.F. Suffredini. 2003. Inflammation-promoting activity of HMGB1 on human microvascular endothelial cells. Blood 101(7): 2652-2660. (Pubitemid 36857626)
-
(2003)
Blood
, vol.101
, Issue.7
, pp. 2652-2660
-
-
Fiuza, C.1
Bustin, M.2
Talwar, S.3
Tropea, M.4
Gerstenberger, E.5
Shelhamer, J.H.6
Suffredini, A.F.7
-
19
-
-
0034754014
-
VEGF increases permeability of the endothelial cell monolayer by activation of PKB/akt, endothelial nitric-oxide synthase, and MAP kinase pathways
-
DOI 10.1006/mvre.2001.2338
-
Lal, B.K., S. Varma, P.J. Pappas, R.W. Hobson 2nd, and W.N. Durán. 2001. VEGF increases permeability of the endothelial cell monolayer by activation of PKB/akt, endothelial nitric-oxide synthase, and MAP kinase pathways. Microvascular Research 62 (3): 252-262. (Pubitemid 33027911)
-
(2001)
Microvascular Research
, vol.62
, Issue.3
, pp. 252-262
-
-
Lal, B.K.1
Varma, S.2
Pappas, P.J.3
Hobson II, R.W.4
Duran, W.N.5
-
20
-
-
0037235049
-
VEGF increases endothelial permeability by separate signaling pathways involving ERK-1/2 and nitric oxide
-
Breslin, J.W., P.J. Pappas, J.J. Cerveira, R.W. Hobson 2nd, and W. N. Durán. 2003. VEGF increases endothelial permeability by separate signaling pathways involving ERK-1/2 and nitric oxide. American Journal of Physiology. Heart and Circulatory Physiology 284(1): H92-H100. (Pubitemid 36076274)
-
(2003)
American Journal of Physiology - Heart and Circulatory Physiology
, vol.284
, Issue.1
-
-
Breslin, J.W.1
Pappas, P.J.2
Cerveira, J.J.3
Hobson II, R.W.4
Duran, W.N.5
-
21
-
-
58949103283
-
Pro-osteogenic phenotype of human aortic valve interstitial cells is associated with higher levels of Toll-like receptors 2 and 4 and enhanced expression of bone morphogenetic protein 2
-
Yang, X., D.A. Fullerton, X. Su, L. Ao, J.C. Cleveland Jr., and X. Meng. 2009. Pro-osteogenic phenotype of human aortic valve interstitial cells is associated with higher levels of Toll-like receptors 2 and 4 and enhanced expression of bone morphogenetic protein 2. Journal of the American College of Cardiology 53(6): 491-500.
-
(2009)
Journal of the American College of Cardiology
, vol.53
, Issue.6
, pp. 491-500
-
-
Yang, X.1
Fullerton, D.A.2
Su, X.3
Ao, L.4
Cleveland Jr., J.C.5
Meng, X.6
-
22
-
-
79953295363
-
Oxidized low-density lipoprotein induces bone morphogenetic protein-2 in coronary artery endothelial cells via Toll-like receptors 2 and 4
-
Su X, Ao L, Shi Y, Johnson TR, Fullerton DA, Meng X. 2011. Oxidized low-density lipoprotein induces bone morphogenetic protein-2 in coronary artery endothelial cells via Toll-like receptors 2 and 4. Journal of Biological Chemistry 286(14): 12213-12220.
-
(2011)
Journal of Biological Chemistry
, vol.286
, Issue.14
, pp. 12213-12220
-
-
Su, X.1
Ao, L.2
Shi, Y.3
Johnson, T.R.4
Fullerton, D.A.5
Meng, X.6
-
23
-
-
1542285224
-
Amphoterin as an extracellular regulator of cell motility: From discovery to disease
-
DOI 10.1111/j.1365-2796.2003.01301.x
-
Huttunen, H.J., and H. Rauvala. 2004. Amphoterin as an extracellular regulator of cell motility: From discovery to disease. Journal of Internal Medicine 255(3): 351-366. (Pubitemid 38315276)
-
(2004)
Journal of Internal Medicine
, vol.255
, Issue.3
, pp. 351-366
-
-
Huttunen, H.J.1
Rauvala, H.2
-
24
-
-
0034753236
-
Dual roles for HMGB1: DNA binding and cytokine
-
DOI 10.1179/096805101101532792
-
Czura, C.J., H. Wang, and K.J. Tracey. 2001. Dual roles for HMGB1: DNA binding and cytokine. Journal of Endotoxin Research 7(4): 315-321. (Pubitemid 33041072)
-
(2001)
Journal of Endotoxin Research
, vol.7
, Issue.4
, pp. 315-321
-
-
Czura, C.J.1
Wang, H.2
Tracey, K.J.3
-
25
-
-
0034698830
-
High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes
-
DOI 10.1084/jem.192.4.565
-
Andersson, U., H. Wang, K. Palmblad, A.C. Aveberger, O. Bloom, H. Erlandsson-Harris, A. Janson, R. Kokkola, M. Zhang, and H. Yang. 2000. High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes. The Journal of Experimental Medicine 192(4): 565-570. (Pubitemid 30666257)
-
(2000)
Journal of Experimental Medicine
, vol.192
, Issue.4
, pp. 565-570
-
-
Andersson, U.1
Wang, H.2
Palmblad, K.3
Aveberger, A.-C.4
Bloom, O.5
Erlandsson-Harris, H.6
Janson, A.7
Kokkola, R.8
Zhang, M.9
Yang, H.10
Tracey, K.J.11
-
26
-
-
56049128160
-
Therapeutic potential of HMGB1-targeting agents in sepsis
-
Wang, H., S. Zhu, R. Zhou, W. Li, and A.E. Sama. 2008. Therapeutic potential of HMGB1-targeting agents in sepsis. Expert Reviews in Molecular Medicine 10: e32.
-
(2008)
Expert Reviews in Molecular Medicine
, vol.10
-
-
Wang, H.1
Zhu, S.2
Zhou, R.3
Li, W.4
Sama, A.E.5
-
27
-
-
0032834765
-
Histamine induces tyrosine phosphorylation of endothelial cell-to-cell adherens junctions
-
Andriopoulou, P., P. Navarro, A. Zanetti, M.G. Lampugnani, and E. Dejana. 1999. Histamine induces tyrosine phosphorylation of endothelial cell-to-cell adherens junctions. Arteriosclerosis, Thrombosis, and Vascular Biology 19(10): 2286-2297. (Pubitemid 29477828)
-
(1999)
Arteriosclerosis, Thrombosis, and Vascular Biology
, vol.19
, Issue.10
, pp. 2286-2297
-
-
Andriopoulou, P.1
Navarro, P.2
Zanetti, A.3
Lampugnani, M.G.4
Dejana, E.5
-
28
-
-
0031809132
-
Increased tyrosine phosphorylation causes redistribution of adherens junction and tight junction proteins and perturbs paracellular barrier function in MDCK epithelia
-
Collares-Buzato, C.B., M.A. Jepson, N.L. Simmons, and B.H. Hirst. 1998. Increased tyrosine phosphorylation causes redistribution of adherens junction and tight junction proteins and perturbs paracellular barrier function in MDCK epithelia. European Journal of Cell Biology 76(2): 85-92. (Pubitemid 28312630)
-
(1998)
European Journal of Cell Biology
, vol.76
, Issue.2
, pp. 85-92
-
-
Collares-Buzato, C.B.1
Jepson, M.A.2
Simmons, N.L.3
Hirst, B.H.4
-
29
-
-
0042845966
-
Protein kinase C modifications of VE-cadherin, p120, and beta-catenin contribute to endothelial barrier dysregulation induced by thrombin
-
Konstantoulaki, M., P. Kouklis, and A.B. Malik. 2003. Protein kinase C modifications of VE-cadherin, p120, and beta-catenin contribute to endothelial barrier dysregulation induced by thrombin. American Journal of Physiology. Lung Cellular and Molecular Physiology 285(2): L434-L442. (Pubitemid 36909460)
-
(2003)
American Journal of Physiology - Lung Cellular and Molecular Physiology
, vol.285
, Issue.2
-
-
Konstantoulaki, M.1
Kouklis, P.2
Malik, A.B.3
-
30
-
-
77957233298
-
VE-cadherin: At the front, center, and sides of endothelial cell organization and function
-
Harris, E.S., and W.J. Nelson. 2010. VE-cadherin: At the front, center, and sides of endothelial cell organization and function. Current Opinion in Cell Biology 22(5): 651-658.
-
(2010)
Current Opinion in Cell Biology
, vol.22
, Issue.5
, pp. 651-658
-
-
Harris, E.S.1
Nelson, W.J.2
-
31
-
-
15444348181
-
Alteration of endothelial cell monolayer integrity triggers resynthesis of vascular endothelium cadherin
-
DOI 10.1074/jbc.273.45.29786
-
Gulino, D., E. Delachanal, E. Concord, Y. Genoux, B. Morand, M. O. Valiron, E. Sulpice, R. Scaife, M. Alemany, and T. Vernet. 1998. Alteration of endothelial cell monolayer integrity triggers resynthesis of vascular endothelium cadherin. The Journal of Biological Chemistry 273(45): 29786-29793. (Pubitemid 28509991)
-
(1998)
Journal of Biological Chemistry
, vol.273
, Issue.45
, pp. 29786-29793
-
-
Gulino, D.1
Delachanal, E.2
Concord, E.3
Genoux, Y.4
Morand, B.5
Valiron, M.-O.6
Sulpice, E.7
Scaife, R.8
Alemany, M.9
Vernet, T.10
-
32
-
-
0029981294
-
Thrombin-induced increase in endothelial permeability is associated with changes in cell-to-cell junction organization
-
Rabiet, M.J., J.L. Plantier, Y. Rival, Y. Genoux, M.G. Lampugnani, and E. Dejana. 1996. Thrombin-induced increase in endothelial permeability is associated with changes in cell-to-cell junction organization. Arteriosclerosis, Thrombosis, and Vascular Biology 16(3): 488-496. (Pubitemid 26084325)
-
(1996)
Arteriosclerosis, Thrombosis, and Vascular Biology
, vol.16
, Issue.3
, pp. 488-496
-
-
Rabiet, M.-J.1
Plantier, J.-L.2
Rival, Y.3
Genoux, Y.4
Lampugnani, M.-G.5
Dejana, E.6
-
33
-
-
0034102390
-
Inflammatory mediators induce sequestration of VE-cadherin in cultured human endothelial cells
-
DOI 10.1023/A:1007025325451
-
Alexander, J.S., B.C. Alexander, L.A. Eppihimer, N. Goodyear, R. Haque, C.P. Davis, T.J. Kalogeris, D.L. Carden, Y.N. Zhu, and C. G. Kevil. 2000. Inflammatory mediators induce sequestration of VE-cadherin in cultured human endothelial cells. Inflammation 24 (2): 99-113. (Pubitemid 30127816)
-
(2000)
Inflammation
, vol.24
, Issue.2
, pp. 99-113
-
-
Alexander, J.S.1
Alexander, B.C.2
Eppihimer, L.A.3
Goodyear, N.4
Haque, R.5
Davis, C.P.6
Kalogeris, T.J.7
Carden, D.L.8
Zhu, Y.-N.9
Kevil, C.G.10
-
34
-
-
0029067198
-
Tumour necrosis factor-alpha-mediated dyscohesion of epithelial cells is associated with disordered expression of cadherin/beta-catenin and disassembly of actin filaments
-
Tabibzadeh, S., Q.F. Kong, S. Kapur, P.G. Satyaswaroop, and K. Aktories. 1995. Tumour necrosis factor-alpha-mediated dyscohesion of epithelial cells is associated with disordered expression of cadherin/beta-catenin and disassembly of actin filaments. Human Reproduction 10(4): 994-1004.
-
(1995)
Human Reproduction
, vol.10
, Issue.4
, pp. 994-1004
-
-
Tabibzadeh, S.1
Kong, Q.F.2
Kapur, S.3
Satyaswaroop, P.G.4
Aktories, K.5
-
35
-
-
0031819246
-
The role of cadherin endocytosis in endothelial barrier regulation: Involvement of protein kinase C and actin-cadherin interactions
-
DOI 10.1023/A:1022325017013
-
Alexander, J.S., S.A. Jackson, E. Chaney, C.G. Kevil, and F.R. Haselton. 1998. The role of cadherin endocytosis in endothelial barrier regulation: Involvement of protein kinase C and actin-cadherin interactions. Inflammation 22(4): 419-433. (Pubitemid 28376857)
-
(1998)
Inflammation
, vol.22
, Issue.4
, pp. 419-433
-
-
Alexander, J.S.1
Jackson, S.A.2
Chaney, E.3
Kevil, C.G.4
Haselton, F.R.5
-
36
-
-
0032053801
-
Role of cadherin internalization in hydrogen peroxide-mediated endothelial permeability
-
DOI 10.1016/S0891-5849(97)00433-4, PII S0891584997004334
-
Kevil, C.G., N. Ohno, D.C. Gute, N. Okayama, S.A. Robinson, E. Chaney, and J.S. Alexander. 1998. Role of cadherin internalization in hydrogen peroxide-mediated endothelial permeability. Free Radical Biology & Medicine 24(6): 1015-1022. (Pubitemid 28186243)
-
(1998)
Free Radical Biology and Medicine
, vol.24
, Issue.6
, pp. 1015-1022
-
-
Kevil, C.G.1
Ohno, N.2
Gute, D.C.3
Okayama, N.4
Robinson, S.A.5
Chaney, E.6
Alexander, J.S.7
-
37
-
-
0036635932
-
The role of p38 map kinase in tumor necrosis factor-induced redistribution of vascular endothelial cadherin and increased endothelial permeability
-
Nwariaku, F.E., J. Chang, X. Zhu, Z. Liu, S.L. Duffy, N.H. Halaihel, L. Terada, and R.H. Turnage. 2002. The role of p38 map kinase in tumor necrosis factor-induced redistribution of vascular endothelial cadherin and increased endothelial permeability. Shock 18(1): 82-85.
-
(2002)
Shock
, vol.18
, Issue.1
, pp. 82-85
-
-
Nwariaku, F.E.1
Chang, J.2
Zhu, X.3
Liu, Z.4
Duffy, S.L.5
Halaihel, N.H.6
Terada, L.7
Turnage, R.H.8
-
39
-
-
67650492748
-
Lipopolysaccharide-induced endothelial barrier breakdown is cyclic adenosine monophosphate dependent in vivo and in vitro
-
Schlegel, N., Y. Baumer, D. Drenckhahn, and J. Waschke. 2009. Lipopolysaccharide-induced endothelial barrier breakdown is cyclic adenosine monophosphate dependent in vivo and in vitro. Critical Care Medicine 37(5): 1735-1743.
-
(2009)
Critical Care Medicine
, vol.37
, Issue.5
, pp. 1735-1743
-
-
Schlegel, N.1
Baumer, Y.2
Drenckhahn, D.3
Waschke, J.4
-
40
-
-
33645461206
-
High mobility group box 1 protein interacts with multiple Toll-like receptors
-
Park, J.S., F. Gamboni-Robertson, Q. He, D. Svetkauskaite, J. Y. Kim, D. Strassheim, J.W. Sohn, S. Yamada, I. Maruyama, A. Banerjee, A. Ishizaka, and E. Abraham. 2006. High mobility group box 1 protein interacts with multiple Toll-like receptors. American Journal of Physiology. Cell Physiology 290(3): C917-C924.
-
(2006)
American Journal of Physiology. Cell Physiology
, vol.290
, Issue.3
-
-
Park, J.S.1
Gamboni-Robertson, F.2
He, Q.3
Svetkauskaite, D.4
Kim, J.Y.5
Strassheim, D.6
Sohn, J.W.7
Yamada, S.8
Maruyama, I.9
Banerjee, A.10
Ishizaka, A.11
Abraham, E.12
-
41
-
-
19944431800
-
RAGE is the major receptor for the proinflammatory activity of HMGB1 in rodent macrophages
-
DOI 10.1111/j.0300-9475.2005.01534.x
-
Kokkola, R., A. Andersson, G. Mullins, T. Ostberg, C.J. Treutiger, B. Arnold, P. Nawroth, U. Andersson, R.A. Harris, and H.E. Harris. 2005. RAGE is the major receptor for the proinflammatory activity of HMGB1 in rodent macrophages. Scandinavian Journal of Immunology 61(1): 1-9. (Pubitemid 40188342)
-
(2005)
Scandinavian Journal of Immunology
, vol.61
, Issue.1
, pp. 1-9
-
-
Kokkola, R.1
Andersson, A.2
Mullins, G.3
Ostberg, T.4
Treutiger, C.-J.5
Arnold, B.6
Nawroth, P.7
Andersson, U.8
Harris, R.A.9
Harris, H.E.10
-
42
-
-
3843144977
-
Regulation of monocyte migration by amphoterin (HMGB1)
-
DOI 10.1182/blood-2003-10-3536
-
Rouhiainen, A., J. Kuja-Panula, E. Wilkman, J. Pakkanen, J. Stenfors, R.K. Tuominen, M. Lepäntalo, O. Carpén, and J. Parkkinen. 2004. Regulation of monocyte migration by amphoterin (HMGB1). Blood 104(4): 1174-1182. (Pubitemid 39038040)
-
(2004)
Blood
, vol.104
, Issue.4
, pp. 1174-1182
-
-
Rouhiainen, A.1
Kuja-Panula, J.2
Wilkman, E.3
Pakkanen, J.4
Stenfors, J.5
Tuominen, R.K.6
Lepantalo, M.7
Carpen, O.8
Parkkinen, J.9
Rauvala, H.10
-
43
-
-
0141750710
-
High mobility group 1 B-box mediates activation of human endothelium
-
DOI 10.1046/j.1365-2796.2003.01204.x
-
Treutiger, C.J., G.E. Mullins, A.S. Johansson, A. Rouhiainen, H. M. Rauvala, H. Erlandsson-Harris, U. Andersson, H. Yang, K.J. Tracey, J. Andersson, and J.E. Palmblad. 2003. High mobility group 1 B-box mediates activation of human endothelium. Journal of Internal Medicine 254(4): 375-385. (Pubitemid 37211261)
-
(2003)
Journal of Internal Medicine
, vol.254
, Issue.4
, pp. 375-385
-
-
Treutiger, C.J.1
Mullins, G.E.2
Johansson, A.-S.M.3
Rouhiainen, A.4
Rauvala, H.M.E.5
Erlandsson-Harris, H.6
Andersson, U.7
Yang, H.8
Tracey, K.J.9
Andersson, J.10
Palmblad, J.E.W.11
-
44
-
-
0028851635
-
The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system
-
Hori, O., J. Brett, T. Slattery, R. Cao, J. Zhang, J.X. Chen, M. Nagashima, E.R. Lundh, S. Vijay, D. Nitecki, et al. 1995. The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system. The Journal of Biological Chemistry 270(43): 25752-25761.
-
(1995)
The Journal of Biological Chemistry
, vol.270
, Issue.43
, pp. 25752-25761
-
-
Hori, O.1
Brett, J.2
Slattery, T.3
Cao, R.4
Zhang, J.5
Chen, J.X.6
Nagashima, M.7
Lundh, E.R.8
Vijay, S.9
Nitecki, D.10
-
45
-
-
77954175102
-
HMGB1, a potent proinflammatory cytokine in sepsis
-
Huang, W., Y. Tang, and L. Li. 2010. HMGB1, a potent proinflammatory cytokine in sepsis. Cytokine 51: 119-126.
-
(2010)
Cytokine
, vol.51
, pp. 119-126
-
-
Huang, W.1
Tang, Y.2
Li, L.3
-
46
-
-
0037102393
-
Receptor for advanced glycation end products-binding COOH-terminal motif of amphoterin inhibits invasive migration and metastasis
-
Huttunen, H.J., C. Fages, J. Kuja-Panula, A.J. Ridley, and H. Rauvala. 2002. Receptor for advanced glycation end products-binding COOH-terminal motif of amphoterin inhibits invasive migration and metastasis. Cancer Research 62(16): 4805-4811.
-
(2002)
Cancer Research
, vol.62
, Issue.16
, pp. 4805-4811
-
-
Huttunen, H.J.1
Fages, C.2
Kuja-Panula, J.3
Ridley, A.J.4
Rauvala, H.5
-
47
-
-
0033538569
-
Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways
-
Huttunen, H.J., C. Fages, and H. Rauvala. 1999. Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways. The Journal of Biological Chemistry 274(28): 19919-19924.
-
(1999)
The Journal of Biological Chemistry
, vol.274
, Issue.28
, pp. 19919-19924
-
-
Huttunen, H.J.1
Fages, C.2
Rauvala, H.3
-
48
-
-
1542380035
-
Involvement of Toll-like Receptors 2 and 4 in Cellular Activation by High Mobility Group Box 1 Protein
-
DOI 10.1074/jbc.M306793200
-
Park, J.S., D. Svetkauskaite, Q. He, J.Y. Kim, D. Strassheim, A. Ishizaka, and E. Abraham. 2004. Involvement of toll-like receptors 2 and 4 in cellular activation by high mobility group box 1 protein. The Journal of Biological Chemistry 279(9): 7370-7377. (Pubitemid 38294612)
-
(2004)
Journal of Biological Chemistry
, vol.279
, Issue.9
, pp. 7370-7377
-
-
Park, J.S.1
Svetkauskaite, D.2
He, Q.3
Kim, J.-Y.4
Strassheim, D.5
Ishizaka, A.6
Abraham, E.7
-
49
-
-
70350448767
-
High-mobility group box 1 protein induces tissue factor expression in vascular endothelial cells via activation of NF-kappaB and Egr-1
-
Lv, B., H. Wang, Y. Tang, Z. Fan, X. Xiao, and F. Chen. 2009. High-mobility group box 1 protein induces tissue factor expression in vascular endothelial cells via activation of NF-kappaB and Egr-1. Thrombosis and Haemostasis 102(2): 352-359.
-
(2009)
Thrombosis and Haemostasis
, vol.102
, Issue.2
, pp. 352-359
-
-
Lv, B.1
Wang, H.2
Tang, Y.3
Fan, Z.4
Xiao, X.5
Chen, F.6
-
50
-
-
78049291345
-
HMGB1 activates nuclear factor-κB signaling by RAGE and increases the production of TNF-κ in human umbilical vein endothelial cells
-
Luan, Z.G., H. Zhang, P.T. Yang, X.C. Ma, C. Zhang, and R.X. Guo. 2010. HMGB1 activates nuclear factor-κB signaling by RAGE and increases the production of TNF-κ in human umbilical vein endothelial cells. Immunobiology 215(12): 956-962.
-
(2010)
Immunobiology
, vol.215
, Issue.12
, pp. 956-962
-
-
Luan, Z.G.1
Zhang, H.2
Yang, P.T.3
Ma, X.C.4
Zhang, C.5
Guo, R.X.6
-
52
-
-
0033213590
-
Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall components
-
DOI 10.1016/S1074-7613(00)80119-3
-
Takeuchi, O., K. Hoshino, T. Kawai, H. Sanjo, H. Takada, T. Ogawa, K. Takeda, and S. Akira. 1999. Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall components. Immunity 11(4): 443-451. (Pubitemid 29504199)
-
(1999)
Immunity
, vol.11
, Issue.4
, pp. 443-451
-
-
Takeuchi, O.1
Hoshino, K.2
Kawai, T.3
Sanjo, H.4
Takada, H.5
Ogawa, T.6
Takeda, K.7
Akira, S.8
-
53
-
-
0034680145
-
Toll-like receptors in the induction of the innate immune response
-
Aderem, A., and R.J. Ulevitch. 2000. Toll-like receptors in the induction of the innate immune response. Nature 406(6797): 782-787.
-
(2000)
Nature
, vol.406
, Issue.6797
, pp. 782-787
-
-
Aderem, A.1
Ulevitch, R.J.2
-
54
-
-
0032509295
-
Defective LPS signaling in C3H/HeJ and C57BL/10ScCr mice: Mutations in Tlr4 gene
-
DOI 10.1126/science.282.5396.2085
-
Poltorak, A., X. He, I. Smirnova, M.Y. Liu, C. Van Huffel, X. Du, D. Birdwell, E. Alejos, M. Silva, C. Galanos, M. Freudenberg, P. Ricciardi-Castagnoli, B. Layton, and B. Beutler. 1998. Defective LPS signaling in C3H/HeJ and C57BL/10ScCr mice: Mutations in Tlr4 gene. Science 282(5396): 2085-2088. (Pubitemid 29001265)
-
(1998)
Science
, vol.282
, Issue.5396
, pp. 2085-2088
-
-
Poltorak, A.1
He, X.2
Smirnova, I.3
Liu, M.-Y.4
Van Huffel, C.5
Du, X.6
Birdwell, D.7
Alejos, E.8
Silva, M.9
Galanos, C.10
Freudenberg, M.11
Ricciardi-Castagnoli, P.12
Layton, B.13
Beutler, B.14
-
55
-
-
77956042544
-
SRC: A century of science brought to the clinic
-
Aleshin, A., and R.S. Finn. 2010. SRC: a century of science brought to the clinic. Neoplasia 12(8): 599-607.
-
(2010)
Neoplasia
, vol.12
, Issue.8
, pp. 599-607
-
-
Aleshin, A.1
Finn, R.S.2
|