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Volumn 49, Issue 2, 2012, Pages 120-127

Eosinophilic myeloid disorders

Author keywords

[No Author keywords available]

Indexed keywords

ALEMTUZUMAB; ALPHA INTERFERON; CORTICOSTEROID; DASATINIB; FIP1L1 PDGFRA ONCOPROTEIN; HYDROXYUREA; IMATINIB; MEPOLIZUMAB; MIDOSTAURIN; NILOTINIB; ONCOPROTEIN; PROTEIN TYROSINE KINASE INHIBITOR; RESLIZUMAB; SORAFENIB; UNCLASSIFIED DRUG;

EID: 84858779837     PISSN: 00371963     EISSN: 15328686     Source Type: Journal    
DOI: 10.1053/j.seminhematol.2012.01.008     Document Type: Article
Times cited : (13)

References (61)
  • 1
    • 0344987881 scopus 로고    scopus 로고
    • A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome
    • Cools J., DeAngelo D.J., Gotlib J., et al. A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome. N Engl J Med 2003, 348:1201-1214.
    • (2003) N Engl J Med , vol.348 , pp. 1201-1214
    • Cools, J.1    DeAngelo, D.J.2    Gotlib, J.3
  • 2
    • 33847176197 scopus 로고    scopus 로고
    • Tyrosine kinases as therapeutic targets in BCR-ABL negative chronic myeloproliferative disorders
    • Reiter A., Walz C., Cross N.C. Tyrosine kinases as therapeutic targets in BCR-ABL negative chronic myeloproliferative disorders. Curr Drug Targets 2007, 8:205-216.
    • (2007) Curr Drug Targets , vol.8 , pp. 205-216
    • Reiter, A.1    Walz, C.2    Cross, N.C.3
  • 3
    • 20144389913 scopus 로고    scopus 로고
    • The t(8;9)(p22;p24) is a recurrent abnormality in chronic and acute leukemia that fuses PCM1 to JAK2
    • Reiter A., Walz C., Watmore A., et al. The t(8;9)(p22;p24) is a recurrent abnormality in chronic and acute leukemia that fuses PCM1 to JAK2. Cancer Res 2005, 65:2662-2667.
    • (2005) Cancer Res , vol.65 , pp. 2662-2667
    • Reiter, A.1    Walz, C.2    Watmore, A.3
  • 4
    • 33746169020 scopus 로고    scopus 로고
    • FLT3 is fused to ETV6 in a myeloproliferative disorder with hypereosinophilia and a t(12;13)(p13;q12) translocation
    • Vu HA X.P., Masuda M., et al. FLT3 is fused to ETV6 in a myeloproliferative disorder with hypereosinophilia and a t(12;13)(p13;q12) translocation. Leukemia 2006, 20:1414-1421.
    • (2006) Leukemia , vol.20 , pp. 1414-1421
    • Vu Ha, X.P.1    Masuda, M.2
  • 5
    • 80052162708 scopus 로고    scopus 로고
    • Response of ETV6-FLT3-positive myeloid/lymphoid neoplasm with eosinophilia to inhibitors of FMS-like tyrosine kinase 3
    • Walz C., Erben P., Ritter M., et al. Response of ETV6-FLT3-positive myeloid/lymphoid neoplasm with eosinophilia to inhibitors of FMS-like tyrosine kinase 3. Blood 2011, 118:2239-2242.
    • (2011) Blood , vol.118 , pp. 2239-2242
    • Walz, C.1    Erben, P.2    Ritter, M.3
  • 6
    • 10744228486 scopus 로고    scopus 로고
    • CHIC2 deletion, a surrogate for FIP1L1-PDGFRA fusion, occurs in systemic mastocytosis associated with eosinophilia and predicts response to imatinib mesylate therapy
    • Pardanani A., Ketterling R.P., Brockman S.R., et al. CHIC2 deletion, a surrogate for FIP1L1-PDGFRA fusion, occurs in systemic mastocytosis associated with eosinophilia and predicts response to imatinib mesylate therapy. Blood 2003, 102:3093-3096.
    • (2003) Blood , vol.102 , pp. 3093-3096
    • Pardanani, A.1    Ketterling, R.P.2    Brockman, S.R.3
  • 7
    • 77952314695 scopus 로고    scopus 로고
    • Screening for diverse PDGFRA or PDGFRB fusion genes is facilitated by generic quantitative reverse transcriptase polymerase chain reaction analysis
    • Erben P., Gosenca D., Muller M.C., et al. Screening for diverse PDGFRA or PDGFRB fusion genes is facilitated by generic quantitative reverse transcriptase polymerase chain reaction analysis. Haematologica 2010, 95:738-744.
    • (2010) Haematologica , vol.95 , pp. 738-744
    • Erben, P.1    Gosenca, D.2    Muller, M.C.3
  • 8
    • 33646900800 scopus 로고    scopus 로고
    • Approaches to the treatment of hypereosinophilic syndromes: a workshop summary report
    • Klion A.D., Bochner B.S., Gleich G.J., et al. Approaches to the treatment of hypereosinophilic syndromes: a workshop summary report. J Allergy Clin Immunol 2006, 117:1292-1302.
    • (2006) J Allergy Clin Immunol , vol.117 , pp. 1292-1302
    • Klion, A.D.1    Bochner, B.S.2    Gleich, G.J.3
  • 10
    • 70349558437 scopus 로고    scopus 로고
    • Myeloid and lymphoid neoplasms with eosinophilia and abnormalities of PDGFRA, PDGFRB or FGFR1
    • IARC Press, Lyon, France, S. Swerdlow, N.L. Harris, H. Stein, E.S. Jaffe, J. Theile, J.W. Vardiman (Eds.)
    • Bain BJ G.D., Horny H.P., et al. Myeloid and lymphoid neoplasms with eosinophilia and abnormalities of PDGFRA, PDGFRB or FGFR1. World Health Organization classification of tumours. Pathology and genetics of tumours of haematopoietic and lymphoid tissues 2008, 68-73. IARC Press, Lyon, France. S. Swerdlow, N.L. Harris, H. Stein, E.S. Jaffe, J. Theile, J.W. Vardiman (Eds.).
    • (2008) World Health Organization classification of tumours. Pathology and genetics of tumours of haematopoietic and lymphoid tissues , pp. 68-73
    • Bain BJ, G.D.1    Horny, H.P.2
  • 12
    • 63349092131 scopus 로고    scopus 로고
    • Validation of a new three-color fluorescence in situ hybridization (FISH) method to detect CHIC2 deletion, FIP1L1/PDGFRA fusion and PDGFRA translocations
    • Fink S.R., Belongie K.J., Paternoster S.F., et al. Validation of a new three-color fluorescence in situ hybridization (FISH) method to detect CHIC2 deletion, FIP1L1/PDGFRA fusion and PDGFRA translocations. Leuk Res 2009, 33:843-846.
    • (2009) Leuk Res , vol.33 , pp. 843-846
    • Fink, S.R.1    Belongie, K.J.2    Paternoster, S.F.3
  • 13
    • 33646152550 scopus 로고    scopus 로고
    • Identification of a novel imatinib responsive KIF5B-PDGFRA fusion gene following screening for PDGFRA overexpression in patients with hypereosinophilia
    • Score J., Curtis C., Waghorn K., et al. Identification of a novel imatinib responsive KIF5B-PDGFRA fusion gene following screening for PDGFRA overexpression in patients with hypereosinophilia. Leukemia 2006, 20:827-832.
    • (2006) Leukemia , vol.20 , pp. 827-832
    • Score, J.1    Curtis, C.2    Waghorn, K.3
  • 14
    • 34249742721 scopus 로고    scopus 로고
    • Recurrent finding of the FIP1L1-PDGFRA fusion gene in eosinophilia-associated acute myeloid leukemia and lymphoblastic T-cell lymphoma
    • Metzgeroth G., Walz C., Score J., et al. Recurrent finding of the FIP1L1-PDGFRA fusion gene in eosinophilia-associated acute myeloid leukemia and lymphoblastic T-cell lymphoma. Leukemia 2007, 21:1183-1188.
    • (2007) Leukemia , vol.21 , pp. 1183-1188
    • Metzgeroth, G.1    Walz, C.2    Score, J.3
  • 15
    • 79953091929 scopus 로고    scopus 로고
    • Novel imatinib-sensitive PDGFRA-activating point mutations in hypereosinophilic syndrome induce growth factor independence and leukemia-like disease
    • Elling C., Erben P., Walz C., et al. Novel imatinib-sensitive PDGFRA-activating point mutations in hypereosinophilic syndrome induce growth factor independence and leukemia-like disease. Blood 2011, 117:2935-2943.
    • (2011) Blood , vol.117 , pp. 2935-2943
    • Elling, C.1    Erben, P.2    Walz, C.3
  • 16
    • 56249123594 scopus 로고    scopus 로고
    • Five years since the discovery of FIP1L1-PDGFRA: what we have learned about the fusion and other molecularly defined eosinophilias
    • Gotlib J., Cools J. Five years since the discovery of FIP1L1-PDGFRA: what we have learned about the fusion and other molecularly defined eosinophilias. Leukemia 2008, 22:1999-2010.
    • (2008) Leukemia , vol.22 , pp. 1999-2010
    • Gotlib, J.1    Cools, J.2
  • 17
    • 77955231742 scopus 로고    scopus 로고
    • Cardiac manifestation of the hypereosinophilic syndrome: new insights
    • Kleinfeldt T., Nienaber C.A., Kische S., et al. Cardiac manifestation of the hypereosinophilic syndrome: new insights. Clin Res Cardiol 2010, 99:419-427.
    • (2010) Clin Res Cardiol , vol.99 , pp. 419-427
    • Kleinfeldt, T.1    Nienaber, C.A.2    Kische, S.3
  • 19
    • 36348971680 scopus 로고    scopus 로고
    • The severity of FIP1L1-PDGFRA-positive chronic eosinophilic leukaemia is associated with polymorphic variation at the IL5RA locus
    • Burgstaller S., Kreil S., Waghorn K., et al. The severity of FIP1L1-PDGFRA-positive chronic eosinophilic leukaemia is associated with polymorphic variation at the IL5RA locus. Leukemia 2007, 21:2428-2432.
    • (2007) Leukemia , vol.21 , pp. 2428-2432
    • Burgstaller, S.1    Kreil, S.2    Waghorn, K.3
  • 20
    • 3042678647 scopus 로고    scopus 로고
    • Clinical and molecular features of FIP1L1-PDFGRA (+) chronic eosinophilic leukemias
    • Vandenberghe P., Wlodarska I., Michaux L., et al. Clinical and molecular features of FIP1L1-PDFGRA (+) chronic eosinophilic leukemias. Leukemia 2004, 18:734-742.
    • (2004) Leukemia , vol.18 , pp. 734-742
    • Vandenberghe, P.1    Wlodarska, I.2    Michaux, L.3
  • 21
    • 0038792233 scopus 로고    scopus 로고
    • Elevated serum tryptase levels identify a subset of patients with a myeloproliferative variant of idiopathic hypereosinophilic syndrome associated with tissue fibrosis, poor prognosis, and imatinib responsiveness
    • Klion A.D., Noel P., Akin C., et al. Elevated serum tryptase levels identify a subset of patients with a myeloproliferative variant of idiopathic hypereosinophilic syndrome associated with tissue fibrosis, poor prognosis, and imatinib responsiveness. Blood 2003, 101:4660-4666.
    • (2003) Blood , vol.101 , pp. 4660-4666
    • Klion, A.D.1    Noel, P.2    Akin, C.3
  • 22
    • 71249101922 scopus 로고    scopus 로고
    • Hypereosinophilic syndrome: a multicenter, retrospective analysis of clinical characteristics and response to therapy
    • Ogbogu P.U., Bochner B.S., Butterfield J.H., et al. Hypereosinophilic syndrome: a multicenter, retrospective analysis of clinical characteristics and response to therapy. J Allergy Clin Immunol 2009, 124:1319-1325.
    • (2009) J Allergy Clin Immunol , vol.124 , pp. 1319-1325
    • Ogbogu, P.U.1    Bochner, B.S.2    Butterfield, J.H.3
  • 23
    • 0142183434 scopus 로고    scopus 로고
    • Serum concentration of cardiac Troponin T in patients with hypereosinophilic syndrome treated with imatinib is predictive of adverse outcomes
    • Pitini V., Arrigo C., Azzarello D., et al. Serum concentration of cardiac Troponin T in patients with hypereosinophilic syndrome treated with imatinib is predictive of adverse outcomes. Blood 2003, 102:3456-3457.
    • (2003) Blood , vol.102 , pp. 3456-3457
    • Pitini, V.1    Arrigo, C.2    Azzarello, D.3
  • 24
    • 0034169552 scopus 로고    scopus 로고
    • Measurement of serum concentrations of cardiac troponin T in patients with hypereosinophilic syndrome: a sensitive non-invasive marker of cardiac disorder
    • Sato Y., Taniguchi R., Yamada T., et al. Measurement of serum concentrations of cardiac troponin T in patients with hypereosinophilic syndrome: a sensitive non-invasive marker of cardiac disorder. Intern Med 2000, 39:350.
    • (2000) Intern Med , vol.39 , pp. 350
    • Sato, Y.1    Taniguchi, R.2    Yamada, T.3
  • 25
    • 0035823530 scopus 로고    scopus 로고
    • Case report: rapid and complete control of idiopathic hypereosinophilia with imatinib mesylate
    • Schaller J.L., Burkland G.A. Case report: rapid and complete control of idiopathic hypereosinophilia with imatinib mesylate. Med Gen Med 2001, 3:9.
    • (2001) Med Gen Med , vol.3 , pp. 9
    • Schaller, J.L.1    Burkland, G.A.2
  • 27
    • 0036063077 scopus 로고    scopus 로고
    • Response of idiopathic hypereosinophilic syndrome to treatment with imatinib mesylate
    • Ault P., Cortes J., Koller C., Kaled E.S., Kantarjian H. Response of idiopathic hypereosinophilic syndrome to treatment with imatinib mesylate. Leuk Res 2002, 26:881-884.
    • (2002) Leuk Res , vol.26 , pp. 881-884
    • Ault, P.1    Cortes, J.2    Koller, C.3    Kaled, E.S.4    Kantarjian, H.5
  • 28
    • 0942276859 scopus 로고    scopus 로고
    • Molecular remission and reversal of myelofibrosis in response to imatinib mesylate treatment in patients with the myeloproliferative variant of hypereosinophilic syndrome
    • Klion A.D., Robyn J., Akin C., et al. Molecular remission and reversal of myelofibrosis in response to imatinib mesylate treatment in patients with the myeloproliferative variant of hypereosinophilic syndrome. Blood 2004, 103:473-478.
    • (2004) Blood , vol.103 , pp. 473-478
    • Klion, A.D.1    Robyn, J.2    Akin, C.3
  • 29
    • 0036678472 scopus 로고    scopus 로고
    • Clinical resistance to the kinase inhibitor STI-571 in chronic myeloid leukemia by mutation of Tyr-253 in the Abl kinase domain P-loop
    • Roumiantsev S., Shah N.P., Gorre M.E., et al. Clinical resistance to the kinase inhibitor STI-571 in chronic myeloid leukemia by mutation of Tyr-253 in the Abl kinase domain P-loop. Proc Natl Acad Sci U S A 2002, 99:10700-10705.
    • (2002) Proc Natl Acad Sci U S A , vol.99 , pp. 10700-10705
    • Roumiantsev, S.1    Shah, N.P.2    Gorre, M.E.3
  • 30
    • 40949147830 scopus 로고    scopus 로고
    • A single weekly dose of imatinib is sufficient to induce and maintain remission of chronic eosinophilic leukaemia in FIP1L1-PDGFRA-expressing patients
    • Helbig G., Stella-Holowiecka B., Majewski M., et al. A single weekly dose of imatinib is sufficient to induce and maintain remission of chronic eosinophilic leukaemia in FIP1L1-PDGFRA-expressing patients. Br J Haematol 2008, 141:200-204.
    • (2008) Br J Haematol , vol.141 , pp. 200-204
    • Helbig, G.1    Stella-Holowiecka, B.2    Majewski, M.3
  • 31
    • 34249732053 scopus 로고    scopus 로고
    • Low-dose imatinib mesylate leads to rapid induction of major molecular responses and achievement of complete molecular remission in FIP1L1-PDGFRA-positive chronic eosinophilic leukemia
    • Jovanovic J.V., Score J., Waghorn K., et al. Low-dose imatinib mesylate leads to rapid induction of major molecular responses and achievement of complete molecular remission in FIP1L1-PDGFRA-positive chronic eosinophilic leukemia. Blood 2007, 109:4635-4640.
    • (2007) Blood , vol.109 , pp. 4635-4640
    • Jovanovic, J.V.1    Score, J.2    Waghorn, K.3
  • 32
    • 36348993783 scopus 로고    scopus 로고
    • Relapse following discontinuation of imatinib mesylate therapy for FIP1L1/PDGFRA-positive chronic eosinophilic leukemia: implications for optimal dosing
    • Klion A.D., Robyn J., Maric I., et al. Relapse following discontinuation of imatinib mesylate therapy for FIP1L1/PDGFRA-positive chronic eosinophilic leukemia: implications for optimal dosing. Blood 2007, 110:3552-3556.
    • (2007) Blood , vol.110 , pp. 3552-3556
    • Klion, A.D.1    Robyn, J.2    Maric, I.3
  • 33
    • 35348993411 scopus 로고    scopus 로고
    • The efficacy of imatinib mesylate in patients with FIP1L1-PDGFRalpha-positive hypereosinophilic syndrome. Results of a multicenter prospective study
    • Baccarani M., Cilloni D., Rondoni M., et al. The efficacy of imatinib mesylate in patients with FIP1L1-PDGFRalpha-positive hypereosinophilic syndrome. Results of a multicenter prospective study. Haematologica 2007, 92:1173-1179.
    • (2007) Haematologica , vol.92 , pp. 1173-1179
    • Baccarani, M.1    Cilloni, D.2    Rondoni, M.3
  • 34
    • 80054843232 scopus 로고    scopus 로고
    • [Successful treatment with low-dose dasatinib in a patient with chronic eosinophilic leukemia intolerant to imatinib]
    • Imagawa J., Harada Y., Yoshida T., et al. [Successful treatment with low-dose dasatinib in a patient with chronic eosinophilic leukemia intolerant to imatinib]. Rinsho Ketsueki 2011, 52:546-550.
    • (2011) Rinsho Ketsueki , vol.52 , pp. 546-550
    • Imagawa, J.1    Harada, Y.2    Yoshida, T.3
  • 35
    • 78650176427 scopus 로고    scopus 로고
    • Low-dose nilotinib can maintain complete molecular remissions in FIP1L1/PDGFRA-positive hypereosinophilic syndrome
    • Tabouret E., Charbonnier A., Mozziconacci M.J., Ivanov V. Low-dose nilotinib can maintain complete molecular remissions in FIP1L1/PDGFRA-positive hypereosinophilic syndrome. Leuk Res 2011, 35:136.
    • (2011) Leuk Res , vol.35 , pp. 136
    • Tabouret, E.1    Charbonnier, A.2    Mozziconacci, M.J.3    Ivanov, V.4
  • 36
    • 41449117618 scopus 로고    scopus 로고
    • Primary resistance to imatinib in Fip1-like 1-platelet-derived growth factor receptor alpha-positive eosinophilic leukemia
    • Simon D., Salemi S., Yousefi S., Simon H.U. Primary resistance to imatinib in Fip1-like 1-platelet-derived growth factor receptor alpha-positive eosinophilic leukemia. J Allergy Clin Immunol 2008, 121:1054-1056.
    • (2008) J Allergy Clin Immunol , vol.121 , pp. 1054-1056
    • Simon, D.1    Salemi, S.2    Yousefi, S.3    Simon, H.U.4
  • 37
    • 79952004905 scopus 로고    scopus 로고
    • The low frequency of clinical resistance to PDGFR inhibitors in myeloid neoplasms with abnormalities of PDGFRA might be related to the limited repertoire of possible PDGFRA kinase domain mutations in vitro
    • von Bubnoff N., Gorantla S.P., Engh R.A., et al. The low frequency of clinical resistance to PDGFR inhibitors in myeloid neoplasms with abnormalities of PDGFRA might be related to the limited repertoire of possible PDGFRA kinase domain mutations in vitro. Oncogene 2011, 30:933-943.
    • (2011) Oncogene , vol.30 , pp. 933-943
    • von Bubnoff, N.1    Gorantla, S.P.2    Engh, R.A.3
  • 38
    • 84856759425 scopus 로고    scopus 로고
    • Limited clinical activity of nilotinib and sorafenib in FIP1L1-PDGFRA positive chronic eosinophilic leukemia with imatinib-resistant T674I mutation
    • Metzgeroth G., Erben P., Martin H., et al. Limited clinical activity of nilotinib and sorafenib in FIP1L1-PDGFRA positive chronic eosinophilic leukemia with imatinib-resistant T674I mutation. Leukemia 2012, 26:162-164.
    • (2012) Leukemia , vol.26 , pp. 162-164
    • Metzgeroth, G.1    Erben, P.2    Martin, H.3
  • 39
    • 66849087150 scopus 로고    scopus 로고
    • FIP1L1-PDGFRalpha D842V, a novel panresistant mutant, emerging after treatment of FIP1L1-PDGFRalpha T674I eosinophilic leukemia with single agent sorafenib
    • Lierman E., Michaux L., Beullens E., et al. FIP1L1-PDGFRalpha D842V, a novel panresistant mutant, emerging after treatment of FIP1L1-PDGFRalpha T674I eosinophilic leukemia with single agent sorafenib. Leukemia 2009, 23:845-851.
    • (2009) Leukemia , vol.23 , pp. 845-851
    • Lierman, E.1    Michaux, L.2    Beullens, E.3
  • 40
    • 77956426667 scopus 로고    scopus 로고
    • Long-term exposure of leukemia cells to multi-targeted tyrosine kinase inhibitor induces activations of AKT, ERK and STAT5 signaling via epigenetic silencing of the PTEN gene
    • Nishioka C., Ikezoe T., Yang J., Yokoyama A. Long-term exposure of leukemia cells to multi-targeted tyrosine kinase inhibitor induces activations of AKT, ERK and STAT5 signaling via epigenetic silencing of the PTEN gene. Leukemia 2010, 24:1631-1640.
    • (2010) Leukemia , vol.24 , pp. 1631-1640
    • Nishioka, C.1    Ikezoe, T.2    Yang, J.3    Yokoyama, A.4
  • 41
    • 33746967701 scopus 로고    scopus 로고
    • Allogeneic bone marrow transplantation for hypereosinophilic syndrome: long-term follow-up with eradication of FIP1L1-PDGFRA fusion transcript
    • Halaburda K., Prejzner W., Szatkowski D., Limon J., Hellmann A. Allogeneic bone marrow transplantation for hypereosinophilic syndrome: long-term follow-up with eradication of FIP1L1-PDGFRA fusion transcript. Bone Marrow Transplant 2006, 38:319-320.
    • (2006) Bone Marrow Transplant , vol.38 , pp. 319-320
    • Halaburda, K.1    Prejzner, W.2    Szatkowski, D.3    Limon, J.4    Hellmann, A.5
  • 42
    • 0036205204 scopus 로고    scopus 로고
    • Allogeneic blood stem cell transplantation following non-myeloablative conditioning for hypereosinophilic syndrome
    • Juvonen E., Volin L., Koponen A., Ruutu T. Allogeneic blood stem cell transplantation following non-myeloablative conditioning for hypereosinophilic syndrome. Bone Marrow Transplant 2002, 29:457-458.
    • (2002) Bone Marrow Transplant , vol.29 , pp. 457-458
    • Juvonen, E.1    Volin, L.2    Koponen, A.3    Ruutu, T.4
  • 43
    • 18644362639 scopus 로고    scopus 로고
    • Successful non-myeloablative allogeneic transplantation for treatment of idiopathic hypereosinophilic syndrome
    • Ueno N.T., Anagnostopoulos A., Rondon G., et al. Successful non-myeloablative allogeneic transplantation for treatment of idiopathic hypereosinophilic syndrome. Br J Haematol 2002, 119:131-134.
    • (2002) Br J Haematol , vol.119 , pp. 131-134
    • Ueno, N.T.1    Anagnostopoulos, A.2    Rondon, G.3
  • 44
    • 8644263305 scopus 로고    scopus 로고
    • FIP1L1-PDGFRA fusion: prevalence and clinicopathologic correlates in 89 consecutive patients with moderate to severe eosinophilia
    • Pardanani A., Brockman S.R., Paternoster S.F., et al. FIP1L1-PDGFRA fusion: prevalence and clinicopathologic correlates in 89 consecutive patients with moderate to severe eosinophilia. Blood 2004, 104:3038-3045.
    • (2004) Blood , vol.104 , pp. 3038-3045
    • Pardanani, A.1    Brockman, S.R.2    Paternoster, S.F.3
  • 45
    • 0343775800 scopus 로고    scopus 로고
    • Complete haematological and cytogenetic response to interferon alpha-2a of a myeloproliferative disorder with eosinophilia associated with a unique t(4;7) aberration
    • Schoffski P., Ganser A., Pascheberg U., Busche G., Gaede B., Hertenstein B. Complete haematological and cytogenetic response to interferon alpha-2a of a myeloproliferative disorder with eosinophilia associated with a unique t(4;7) aberration. Ann Hematol 2000, 79:95-98.
    • (2000) Ann Hematol , vol.79 , pp. 95-98
    • Schoffski, P.1    Ganser, A.2    Pascheberg, U.3    Busche, G.4    Gaede, B.5    Hertenstein, B.6
  • 46
    • 34250894934 scopus 로고    scopus 로고
    • Eosinophilia in systemic mastocytosis: clinical and molecular correlates and prognostic significance
    • Bohm A., Fodinger M., Wimazal F., et al. Eosinophilia in systemic mastocytosis: clinical and molecular correlates and prognostic significance. J Allergy Clin Immunol 2007, 120:192-199.
    • (2007) J Allergy Clin Immunol , vol.120 , pp. 192-199
    • Bohm, A.1    Fodinger, M.2    Wimazal, F.3
  • 48
    • 34548272148 scopus 로고    scopus 로고
    • KIT D816V-associated systemic mastocytosis with eosinophilia and FIP1L1/PDGFRA-associated chronic eosinophilic leukemia are distinct entities
    • Maric I., Robyn J., Metcalfe D.D., et al. KIT D816V-associated systemic mastocytosis with eosinophilia and FIP1L1/PDGFRA-associated chronic eosinophilic leukemia are distinct entities. J Allergy Clin Immunol 2007, 120:680-687.
    • (2007) J Allergy Clin Immunol , vol.120 , pp. 680-687
    • Maric, I.1    Robyn, J.2    Metcalfe, D.D.3
  • 49
    • 0028224348 scopus 로고
    • Fusion of PDGF receptor beta to a novel ets-like gene, tel, in chronic myelomonocytic leukemia with t(5;12) chromosomal translocation
    • Golub T.R., Barker G.F., Lovett M., Gilliland D.G. Fusion of PDGF receptor beta to a novel ets-like gene, tel, in chronic myelomonocytic leukemia with t(5;12) chromosomal translocation. Cell 1994, 77:307-316.
    • (1994) Cell , vol.77 , pp. 307-316
    • Golub, T.R.1    Barker, G.F.2    Lovett, M.3    Gilliland, D.G.4
  • 50
    • 0037103624 scopus 로고    scopus 로고
    • Response to imatinib mesylate in patients with chronic myeloproliferative diseases with rearrangements of the platelet-derived growth factor receptor beta
    • Apperley J.F., Gardembas M., Melo J.V., et al. Response to imatinib mesylate in patients with chronic myeloproliferative diseases with rearrangements of the platelet-derived growth factor receptor beta. N Engl J Med 2002, 347:481-487.
    • (2002) N Engl J Med , vol.347 , pp. 481-487
    • Apperley, J.F.1    Gardembas, M.2    Melo, J.V.3
  • 51
    • 0036202529 scopus 로고    scopus 로고
    • Myeloproliferative disorders with translocations of chromosome 5q31-35: role of the platelet-derived growth factor receptor beta
    • Steer E.J., Cross N.C. Myeloproliferative disorders with translocations of chromosome 5q31-35: role of the platelet-derived growth factor receptor beta. Acta Haematol 2002, 107:113-122.
    • (2002) Acta Haematol , vol.107 , pp. 113-122
    • Steer, E.J.1    Cross, N.C.2
  • 52
    • 0031454003 scopus 로고    scopus 로고
    • CGP 57148, a tyrosine kinase inhibitor, inhibits the growth of cells expressing BCR-ABL, TEL-ABL, and TEL-PDGFR fusion proteins
    • Carroll M., Ohno-Jones S., Tamura S., et al. CGP 57148, a tyrosine kinase inhibitor, inhibits the growth of cells expressing BCR-ABL, TEL-ABL, and TEL-PDGFR fusion proteins. Blood 1997, 90:4947-4952.
    • (1997) Blood , vol.90 , pp. 4947-4952
    • Carroll, M.1    Ohno-Jones, S.2    Tamura, S.3
  • 53
    • 67349165083 scopus 로고    scopus 로고
    • Myeloproliferative disorder with eosinophilia and ETV6-ABL gene rearrangement: efficacy of second-generation tyrosine kinase inhibitors
    • Nand R., Bryke C., Kroft S.H., Divgi A., Bredeson C., Atallah E. Myeloproliferative disorder with eosinophilia and ETV6-ABL gene rearrangement: efficacy of second-generation tyrosine kinase inhibitors. Leuk Res 2009, 33:1144-1146.
    • (2009) Leuk Res , vol.33 , pp. 1144-1146
    • Nand, R.1    Bryke, C.2    Kroft, S.H.3    Divgi, A.4    Bredeson, C.5    Atallah, E.6
  • 54
    • 0029010203 scopus 로고
    • Myeloproliferative disorder associated with 8p11 translocations
    • Aguiar R.C., Macdonald D., Mason P.J., Cross N.C., Goldman J.M. Myeloproliferative disorder associated with 8p11 translocations. Blood 1995, 86:834-835.
    • (1995) Blood , vol.86 , pp. 834-835
    • Aguiar, R.C.1    Macdonald, D.2    Mason, P.J.3    Cross, N.C.4    Goldman, J.M.5
  • 55
  • 56
    • 0033558253 scopus 로고    scopus 로고
    • The t(6;8)(q27;p11) translocation in a stem cell myeloproliferative disorder fuses a novel gene, FOP, to fibroblast growth factor receptor 1
    • Popovici C., Zhang B., Gregoire M.J., et al. The t(6;8)(q27;p11) translocation in a stem cell myeloproliferative disorder fuses a novel gene, FOP, to fibroblast growth factor receptor 1. Blood 1999, 93:1381-1389.
    • (1999) Blood , vol.93 , pp. 1381-1389
    • Popovici, C.1    Zhang, B.2    Gregoire, M.J.3
  • 57
    • 11044238012 scopus 로고    scopus 로고
    • Clinical variability of patients with the t(6;8)(q27;p12) and FGFR1OP-FGFR1 fusion: two further cases
    • Vizmanos J.L., Hernandez R., Vidal M.J., et al. Clinical variability of patients with the t(6;8)(q27;p12) and FGFR1OP-FGFR1 fusion: two further cases. Hematol J 2004, 5:534-537.
    • (2004) Hematol J , vol.5 , pp. 534-537
    • Vizmanos, J.L.1    Hernandez, R.2    Vidal, M.J.3
  • 58
    • 5144234541 scopus 로고    scopus 로고
    • PKC412 inhibits the zinc finger 198-fibroblast growth factor receptor 1 fusion tyrosine kinase and is active in treatment of stem cell myeloproliferative disorder
    • Chen J., Deangelo D.J., Kutok J.L., et al. PKC412 inhibits the zinc finger 198-fibroblast growth factor receptor 1 fusion tyrosine kinase and is active in treatment of stem cell myeloproliferative disorder. Proc Natl Acad Sci U S A 2004, 101:14479-14484.
    • (2004) Proc Natl Acad Sci U S A , vol.101 , pp. 14479-14484
    • Chen, J.1    Deangelo, D.J.2    Kutok, J.L.3
  • 59
    • 0037866468 scopus 로고    scopus 로고
    • Prevalence and clinical characteristics of myelodysplastic syndrome with bone marrow eosinophilia or basophilia
    • Matsushima T., Handa H., Yokohama A., et al. Prevalence and clinical characteristics of myelodysplastic syndrome with bone marrow eosinophilia or basophilia. Blood 2003, 101:3386-3390.
    • (2003) Blood , vol.101 , pp. 3386-3390
    • Matsushima, T.1    Handa, H.2    Yokohama, A.3
  • 60
    • 77952556491 scopus 로고    scopus 로고
    • Evaluation of the prognostic significance of eosinophilia and basophilia in a larger cohort of patients with myelodysplastic syndromes
    • Wimazal F., Germing U., Kundi M., et al. Evaluation of the prognostic significance of eosinophilia and basophilia in a larger cohort of patients with myelodysplastic syndromes. Cancer 2010, 116:2372-2381.
    • (2010) Cancer , vol.116 , pp. 2372-2381
    • Wimazal, F.1    Germing, U.2    Kundi, M.3
  • 61
    • 79955947379 scopus 로고    scopus 로고
    • The renaissance of interferon therapy for teh treatment of myeloid malignancies
    • Kiladjian JJ M.R., Hoffman R. The renaissance of interferon therapy for teh treatment of myeloid malignancies. Blood 2011, 117:4701-4715.
    • (2011) Blood , vol.117 , pp. 4701-4715
    • Kiladjian JJ, M.R.1    Hoffman, R.2


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