Intracisternal administration of p-n-octylphenol into neonatal rats causes hyperactivity concomitantly with the terminal deoxynucleotidyl transferase-mediated dUTP nick end-labelling (TUNEL)-positive cells in the mesencephalon where immunoreactivity for tyrosine hydroxylase is reduced by the chemical

Journal of Health Science

Volumn 50, Issue 4, 2004, Pages 407-412

  Ishido, Masami ^a   Masuo, Yoshinori ^b   Oka, Syuichi ^b   Niki, Etsuo ^b   Morita, Masatoshi ^a  

^a NATIONAL INSTITUTE FOR ENVIRONMENTAL STUDIES   (Japan)

^b NATIONAL INSTITUTE OF ADVANCED INDUSTRIAL SCIENCE AND TECHNOLOGY AIST   (Japan)

Author keywords

Endocrine disruptor; Hyperactivity; p n octylphenol; Tyrosine hydroxylase

Indexed keywords

4 OCTYLPHENOL; ENDOCRINE DISRUPTOR; TYROSINE 3 MONOOXYGENASE;

ANIMAL BEHAVIOR; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BRAIN DEVELOPMENT; CONTROLLED STUDY; HYPERACTIVITY; IMMUNOHISTOCHEMISTRY; IMMUNOREACTIVITY; MALE; MESENCEPHALON; NICK END LABELING; NONHUMAN; RAT; RAT STRAIN;

ANIMALIA; RATTUS NORVEGICUS;

EID: 8444243127     PISSN: 13449702     EISSN: None     Source Type: Journal    
DOI: 10.1248/jhs.50.407     Document Type: Article

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