Prenatal lead acetate exposure induces apoptosis and changes GFAP expression during spinal cord development

Ecotoxicology and Environmental Safety

Volumn 75, Issue 1, 2012, Pages 223-229

  Müller, Yara Maria Rauh ^a   Kobus, Karoline ^a   Schatz, Janaína Chaves ^a   Ammar, Dib ^a   Nazari, Evelise Maria ^a  

^a FEDERAL UNIVERSITY OF SANTA CATARINA   (Brazil)

Author keywords

Apoptosis; Chick embryos; GFAP expression; Lead acetate; Spinal cord

Indexed keywords

GLIAL FIBRILLARY ACIDIC PROTEIN; LEAD ACETATE;

ACETATE; APOPTOSIS; EMBRYONIC DEVELOPMENT; GENE EXPRESSION; HISTOLOGY; LEAD; PHYSIOLOGICAL RESPONSE; POLLUTION EFFECT; POLLUTION EXPOSURE; TOXICITY; ULTRASTRUCTURE;

ANIMAL CELL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BIOACCUMULATION; CELL DENSITY; CELLULAR DISTRIBUTION; CHICK; CONGENITAL MALFORMATION; CONTROLLED STUDY; DEVELOPMENTAL STAGE; EMBRYO; EMBRYONAL TISSUE; EPENDYMA CELL; GRAY MATTER; NERVOUS SYSTEM DEVELOPMENT; NONHUMAN; PRENATAL EXPOSURE; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; SPINAL CORD DEVELOPMENT; TISSUE DISTRIBUTION; WHITE MATTER; YOLK SAC;

ANIMALS; APOPTOSIS; CHICK EMBRYO; GLIAL FIBRILLARY ACIDIC PROTEIN; HAZARDOUS SUBSTANCES; IN SITU NICK-END LABELING; NERVOUS SYSTEM; NEUROGLIA; ORGANOMETALLIC COMPOUNDS; SPINAL CORD;

SPONDIAS PURPUREA;

EID: 80655148996     PISSN: 01476513     EISSN: 10902414     Source Type: Journal    
DOI: 10.1016/j.ecoenv.2011.08.004     Document Type: Article

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