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Volumn 795, Issue , 2012, Pages 233-253

Investigation of acquired resistance to EGFR-targeted therapies in lung cancer using cDNA microarrays

Author keywords

Erlotinib; Gefitinib; Non small cell lung cancer; Proteomics; Resistance; Therapeutic response

Indexed keywords

ANTINEOPLASTIC AGENT; EPIDERMAL GROWTH FACTOR RECEPTOR; GEFITINIB; PROTEIN KINASE INHIBITOR; QUINAZOLINE DERIVATIVE;

EID: 80555135989     PISSN: 10643745     EISSN: None     Source Type: Book Series    
DOI: 10.1007/978-1-61779-337-0_16     Document Type: Article
Times cited : (1)

References (13)
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    • Structure and clinical relevance of the epidermal growth factor receptor in human cancer
    • Kumar, A., Petri, E. T., Halmos, B., and Boggon, T. J. (2008) Structure and clinical relevance of the epidermal growth factor receptor in human cancer. J. Clin. Oncol. 26, 1742-1751.
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    • Kumar, A.1    Petri, E.T.2    Halmos, B.3    Boggon, T.J.4
  • 5
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    • Somatic mutations of epidermal growth factor receptor signaling pathway in lung cancers
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    • Acquired resistance of lung adenocarcinomas to gefitinib or erlotinib is associated with a second mutation in the EGFR kinase domain
    • Pao, W., Miller, V. A., Politi, K. A., Riely, G. J., Somwar, R., Zakowski, M. F., Kris, M. G., and Varmus, H. (2005) Acquired resistance of lung adenocarcinomas to gefitinib or erlotinib is associated with a second mutation in the EGFR kinase domain. PLoS Med. 2, e73.
    • (2005) PLoS Med. , vol.2
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.