Mitochondrial DNA damage is sensitive to exogenous H 2O 2 but independent of cellular ROS production in prostate cancer cells

Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis

Volumn 716, Issue 1-2, 2011, Pages 40-50

  Chan, Sam W ^a   Nguyen, Phuong Nam ^a   Ayele, David ^a   Chevalier, Simone ^a   Aprikian, Armen ^a   Chen, Junjian Z ^a  

^a MCGILL UNIVERSITY HEALTH CENTRE   (Canada)

Author keywords

MtDNA damage; Oxidative stress; Prostate cancer; ROS; Supercoiling sensitive PCR

Indexed keywords

APOCYNIN; DIPHENYLIODONIUM SALT; HYDROGEN PEROXIDE; MITOCHONDRIAL DNA; REACTIVE OXYGEN METABOLITE; SUPEROXIDE;

ARTICLE; CANCER CELL; CONTROLLED STUDY; CYTOTOXICITY; DNA DAMAGE; HUMAN; HUMAN CELL; OXIDATION; OXIDATIVE STRESS; PHENOTYPE; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; PROSTATE CANCER;

CELL DEATH; CELL LINE; CELL LINE, TUMOR; DNA DAMAGE; DNA, MITOCHONDRIAL; HUMANS; HYDROGEN PEROXIDE; MALE; OXIDATIVE STRESS; PROSTATE; PROSTATIC NEOPLASMS; REACTIVE OXYGEN SPECIES; SUPEROXIDES;

EID: 80053562653     PISSN: 00275107     EISSN: 09218262     Source Type: Journal    
DOI: 10.1016/j.mrfmmm.2011.07.019     Document Type: Article

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