Androgen receptor transactivity is potentiated by TGF-β1 through Smad3 but checked by its coactivator Hic-5/ARA55 in balding dermal papilla cells

Journal of Dermatological Science

Volumn 64, Issue 2, 2011, Pages 149-151

  Inui, Shigeki ^a   Itami, Satoshi ^a  

^a OSAKA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

Author keywords

Androgen; Androgenetic alopecia; Hic 5 ARA55; Smad3; TGF

Indexed keywords

ANDROGEN; ANDROGEN RECEPTOR; LUCIFERASE; PROTEIN HIC 5; REGULATOR PROTEIN; SMAD3 PROTEIN; TRANSFORMING GROWTH FACTOR BETA1; UNCLASSIFIED DRUG;

ANIMAL CELL; AUTOCRINE EFFECT; BALD DERMAL PAPILLA CELL; CELL DIFFERENTIATION; CELL MATURATION; CONTROLLED STUDY; ENZYME ACTIVITY; GENE SILENCING; HUMAN; LETTER; MALE TYPE ALOPECIA; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; SKIN CELL; TRANSACTIVATION; TRANSIENT TRANSFECTION;

ALOPECIA; HUMANS; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; LIM DOMAIN PROTEINS; RECEPTORS, ANDROGEN; SMAD3 PROTEIN; TRANSCRIPTIONAL ACTIVATION; TRANSFORMING GROWTH FACTOR BETA1;

EID: 80053383694     PISSN: 09231811     EISSN: 1873569X     Source Type: Journal    
DOI: 10.1016/j.jdermsci.2011.08.010     Document Type: Letter

References (10)

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