Mechanisms of reduced contractility in an animal model of hypertensive heart failure

Clinical and Experimental Pharmacology and Physiology

Volumn 38, Issue 10, 2011, Pages 711-716

  Ward, Marie Louise ^a   Crossman, David J ^a   Cannell, Mark B ^a  

^a UNIVERSITY OF AUCKLAND   (New Zealand)

Author keywords

Ca 2+ transient; Heart failure; Na + Ca 2+ exchanger; Sarcoplasmic reticulum; Spontaneously hypertensive rat

Indexed keywords

ADENOSINE TRIPHOSPHATASE (CALCIUM); CALCIUM ION;

ANIMAL EXPERIMENT; ANIMAL MODEL; CALCIUM CELL LEVEL; CALCIUM TRANSPORT; CONFERENCE PAPER; CONFOCAL MICROSCOPY; CONTROLLED STUDY; EOSINOPHIL COUNT; HEART FAILURE; HEART LEFT VENTRICLE WALL; HEART MUSCLE CONTRACTILITY; IMMUNOHISTOCHEMISTRY; MUSCLE CELL; NONHUMAN; RAT; SARCOLEMMA; SODIUM CALCIUM EXCHANGE; SPONTANEOUSLY HYPERTENSIVE HEART FAILURE RAT; STEADY STATE;

ANIMALS; CAFFEINE; CALCIUM; DISEASE MODELS, ANIMAL; HEART FAILURE; HUMANS; HYPERTENSION; MYOCARDIAL CONTRACTION; MYOCARDIUM; MYOCYTES, CARDIAC; RATS; RATS, INBRED SHR; RATS, INBRED WKY; SARCOLEMMA; SARCOPLASMIC RETICULUM; SARCOPLASMIC RETICULUM CALCIUM-TRANSPORTING ATPASES; SODIUM-CALCIUM EXCHANGER;

EID: 80053161939     PISSN: 03051870     EISSN: 14401681     Source Type: Journal    
DOI: 10.1111/j.1440-1681.2011.05563.x     Document Type: Conference Paper

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