-
1
-
-
0037180771
-
Inflammation in atherosclerosis
-
Libby P, (2001) Inflammation in atherosclerosis. Nature 420: 868-874.
-
(2001)
Nature
, vol.420
, pp. 868-874
-
-
Libby, P.1
-
2
-
-
4644350285
-
Estrogen modulates inflammatory mediator expression and neutrophil chemotaxis in injured arteries
-
Miller AP, Feng W, Xing D, Weathington NM, Blalock JE, et al. (2004) Estrogen modulates inflammatory mediator expression and neutrophil chemotaxis in injured arteries. Circulation 110: 1664-1669.
-
(2004)
Circulation
, vol.110
, pp. 1664-1669
-
-
Miller, A.P.1
Feng, W.2
Xing, D.3
Weathington, N.M.4
Blalock, J.E.5
-
3
-
-
1642535529
-
Estradiol and progestins differentially modulate leukocyte infiltration after vascular injury
-
Xing D, Miller A, Novak L, Rocha R, Chen YF, et al. (2004) Estradiol and progestins differentially modulate leukocyte infiltration after vascular injury. Circulation 109: 234-241.
-
(2004)
Circulation
, vol.109
, pp. 234-241
-
-
Xing, D.1
Miller, A.2
Novak, L.3
Rocha, R.4
Chen, Y.F.5
-
4
-
-
0025782244
-
N-acetylglucosaminono-1,5-lactone oxime and the corresponding (phenylcarbamoyl)oxime. Novel and potent inhibitors of beta-N-acetyl-glucosaminidase
-
Horsch M, Hoesch L, Vasella A, Rast DM, (1991) N-acetylglucosaminono-1,5-lactone oxime and the corresponding (phenylcarbamoyl)oxime. Novel and potent inhibitors of beta-N-acetyl-glucosaminidase. Eur J Biochem 197: 815-818.
-
(1991)
Eur J Biochem
, vol.197
, pp. 815-818
-
-
Horsch, M.1
Hoesch, L.2
Vasella, A.3
Rast, D.M.4
-
5
-
-
49849106530
-
Increased protein O-GlcNAc modification inhibits inflammatory and neointimal responses to acute endoluminal arterial injury
-
Xing D, Feng W, Nöt LG, Miller AP, Zhang Y, et al. (2008) Increased protein O-GlcNAc modification inhibits inflammatory and neointimal responses to acute endoluminal arterial injury. Am J Physiol Heart Circ Physiol 295: H335-H342.
-
(2008)
Am J Physiol Heart Circ Physiol
, vol.295
, pp. 335-342
-
-
Xing, D.1
Feng, W.2
Nöt, L.G.3
Miller, A.P.4
Zhang, Y.5
-
6
-
-
34447578798
-
Estrogen modulates TNF-α-induced inflammation in rat aortic smooth muscle cells through estrogen receptor-β activation
-
Xing D, Feng W, Miller AP, Weathington NM, Chen YF, et al. (2007) Estrogen modulates TNF-α-induced inflammation in rat aortic smooth muscle cells through estrogen receptor-β activation. Am J Physiol Heart Circ Physiol 292: H2607-H2612.
-
(2007)
Am J Physiol Heart Circ Physiol
, vol.292
, pp. 2607-2612
-
-
Xing, D.1
Feng, W.2
Miller, A.P.3
Weathington, N.M.4
Chen, Y.F.5
-
7
-
-
0037022235
-
Activation of nuclear factor-kappa B significantly contributes to lumen loss in a rabbit iliac artery balloon angioplasty model
-
Breuss JM, Cejna M, Bergmeister H, Kadl A, Baumgartl G, et al. (2002) Activation of nuclear factor-kappa B significantly contributes to lumen loss in a rabbit iliac artery balloon angioplasty model. Circulation 105: 633-638.
-
(2002)
Circulation
, vol.105
, pp. 633-638
-
-
Breuss, J.M.1
Cejna, M.2
Bergmeister, H.3
Kadl, A.4
Baumgartl, G.5
-
8
-
-
23444433359
-
IKKbeta-dependent NF-kappaB pathway controls vascular inflammation and intimal hyperplasia
-
Bu DX, Erl W, de Martin R, Hansson GK, Yan ZQ, (2005) IKKbeta-dependent NF-kappaB pathway controls vascular inflammation and intimal hyperplasia. FASEB J 19: 1293-1295.
-
(2005)
FASEB J
, vol.19
, pp. 1293-1295
-
-
Bu, D.X.1
Erl, W.2
de Martin, R.3
Hansson, G.K.4
Yan, Z.Q.5
-
9
-
-
0030052429
-
Glucocorticoid-mediated gene suppression of rat cytokine-induced neutrophil chemoattractant CINC-/gro, a member of the interleukin-8 family, through impairment of NF-kB activation
-
Ohtsuka T, Kubota A, Hirano T, Watanabe K, Yoshida H, et al. (1996) Glucocorticoid-mediated gene suppression of rat cytokine-induced neutrophil chemoattractant CINC-/gro, a member of the interleukin-8 family, through impairment of NF-kB activation. J Bio Chem 271: 1651-1659.
-
(1996)
J Bio Chem
, vol.271
, pp. 1651-1659
-
-
Ohtsuka, T.1
Kubota, A.2
Hirano, T.3
Watanabe, K.4
Yoshida, H.5
-
10
-
-
49249121580
-
Angiotensin II and tumor necrosis factor-alpha synergistically promote monocyte chemoattractant protein-1 expression: roles of NF-kappaB, p38, and reactive oxygen species
-
Takahashi M, Suzuki E, Takeda R, Oba S, Nishimatsu H, et al. (2008) Angiotensin II and tumor necrosis factor-alpha synergistically promote monocyte chemoattractant protein-1 expression: roles of NF-kappaB, p38, and reactive oxygen species. Am J Physiol Heart Circ Physiol 294: H2879-2888.
-
(2008)
Am J Physiol Heart Circ Physiol
, vol.294
, pp. 2879-2888
-
-
Takahashi, M.1
Suzuki, E.2
Takeda, R.3
Oba, S.4
Nishimatsu, H.5
-
11
-
-
36249022077
-
Nuclear translocation of p65 NF-kappaB is sufficient for VCAM-1, but not ICAM-1, expression in TNF-stimulated smooth muscle cells: Differential requirement for PARP-1 expression and interaction
-
Zerfaoui M, Suzuki Y, Naura AS, Hans CP, Nichols C, et al. (2008) Nuclear translocation of p65 NF-kappaB is sufficient for VCAM-1, but not ICAM-1, expression in TNF-stimulated smooth muscle cells: Differential requirement for PARP-1 expression and interaction. Cell Signal 20: 186-194.
-
(2008)
Cell Signal
, vol.20
, pp. 186-194
-
-
Zerfaoui, M.1
Suzuki, Y.2
Naura, A.S.3
Hans, C.P.4
Nichols, C.5
-
12
-
-
0347955360
-
Regulation of NF-kappaB signaling by Pin1-dependent prolyl isomerization and ubiquitin-mediated proteolysis of p65/RelA
-
Ryo A, Suizu F, Yoshida Y, Perrem K, Liou YC, et al. (2003) Regulation of NF-kappaB signaling by Pin1-dependent prolyl isomerization and ubiquitin-mediated proteolysis of p65/RelA. Mol Cell 12: 1413-26.
-
(2003)
Mol Cell
, vol.12
, pp. 1413-1426
-
-
Ryo, A.1
Suizu, F.2
Yoshida, Y.3
Perrem, K.4
Liou, Y.C.5
-
13
-
-
0030991201
-
The transcriptional activity of NF-kappaB is regulated by the IkappaB-associated PKAc subunit through a cyclic AMP-independent mechanism
-
Zhong H, SuYang H, Erdjument-Bromage H, Tempst P, Ghosh S, (1997) The transcriptional activity of NF-kappaB is regulated by the IkappaB-associated PKAc subunit through a cyclic AMP-independent mechanism. Cell 89: 413-24.
-
(1997)
Cell
, vol.89
, pp. 413-424
-
-
Zhong, H.1
SuYang, H.2
Erdjument-Bromage, H.3
Tempst, P.4
Ghosh, S.5
-
14
-
-
2942716907
-
p53 induces NF-kappaB activation by an IkappaB kinase-independent mechanism involving phosphorylation of p65 by ribosomal S6 kinase 1
-
Bohuslav J, Chen LF, Kwon H, Mu Y, Greene WC, (2004) p53 induces NF-kappaB activation by an IkappaB kinase-independent mechanism involving phosphorylation of p65 by ribosomal S6 kinase 1. J Biol Chem 279: 26115-25.
-
(2004)
J Biol Chem
, vol.279
, pp. 26115-26125
-
-
Bohuslav, J.1
Chen, L.F.2
Kwon, H.3
Mu, Y.4
Greene, W.C.5
-
15
-
-
0037451357
-
Transcriptional activation of the NF-kappaB p65 subunit by mitogen- and stress-activated protein kinase-1 (MSK1)
-
Vermeulen L, De Wilde G, Van Damme P, Vanden Berghe W, Haegeman G, (2003) Transcriptional activation of the NF-kappaB p65 subunit by mitogen- and stress-activated protein kinase-1 (MSK1). EMBO J 22: 1313-24.
-
(2003)
EMBO J
, vol.22
, pp. 1313-1324
-
-
Vermeulen, L.1
De Wilde, G.2
Van Damme, P.3
Vanden Berghe, W.4
Haegeman, G.5
-
16
-
-
0042525909
-
Essential role of RelA Ser311 phosphorylation by zetaPKC in NF-kappaB transcriptional activation
-
Duran A, Diaz-Meco MT, Moscat J, (2003) Essential role of RelA Ser311 phosphorylation by zetaPKC in NF-kappaB transcriptional activation. EMBO J 22: 3910-8.
-
(2003)
EMBO J
, vol.22
, pp. 3910-3918
-
-
Duran, A.1
Diaz-Meco, M.T.2
Moscat, J.3
-
17
-
-
0034693133
-
Tumor necrosis factor alpha-induced phosphorylation of RelA/p65 on Ser529 is controlled by casein kinase II
-
Wang D, Westerheide SD, Hanson JL, Baldwin AS Jr, (2000) Tumor necrosis factor alpha-induced phosphorylation of RelA/p65 on Ser529 is controlled by casein kinase II. J Biol Chem 275: 32592-7.
-
(2000)
J Biol Chem
, vol.275
, pp. 32592-32597
-
-
Wang, D.1
Westerheide, S.D.2
Hanson, J.L.3
Baldwin Jr., A.S.4
-
18
-
-
0032589462
-
IkappaB kinases phosphorylate NF-kappaB p65 subunit on serine 536 in the transactivation domain
-
Sakurai H, Chiba H, Miyoshi H, Sugita T, Toriumi W, (1999) IkappaB kinases phosphorylate NF-kappaB p65 subunit on serine 536 in the transactivation domain. J Biol Chem 274: 30353-6.
-
(1999)
J Biol Chem
, vol.274
, pp. 30353-30356
-
-
Sakurai, H.1
Chiba, H.2
Miyoshi, H.3
Sugita, T.4
Toriumi, W.5
-
19
-
-
0034718570
-
Alternative O-glycosylation/O-phosphorylation of the murine estrogen receptor beta
-
Cheng X, Cole RN, Zaia J, Hart GW, (2000) Alternative O-glycosylation/O-phosphorylation of the murine estrogen receptor beta. Biochemistry 39: 11609-11620.
-
(2000)
Biochemistry
, vol.39
, pp. 11609-11620
-
-
Cheng, X.1
Cole, R.N.2
Zaia, J.3
Hart, G.W.4
-
20
-
-
0029049198
-
c-Myc is glycosylated at threonine 58, a known phosphorylation site and a mutational hot spot in lymphomas
-
Chou TY, Hart GW, Dang CV, (1995) c-Myc is glycosylated at threonine 58, a known phosphorylation site and a mutational hot spot in lymphomas. J Biol Chem 270: 18961-5.
-
(1995)
J Biol Chem
, vol.270
, pp. 18961-18965
-
-
Chou, T.Y.1
Hart, G.W.2
Dang, C.V.3
-
21
-
-
38849199203
-
Shared principles in NF-kappaB signaling
-
Hayden MS, Ghosh S, (2008) Shared principles in NF-kappaB signaling. Cell 132: 344-362.
-
(2008)
Cell
, vol.132
, pp. 344-362
-
-
Hayden, M.S.1
Ghosh, S.2
-
22
-
-
0036203419
-
The phosphorylation status of nuclear NF-kappa B determines its association with CBP/p300 or HDAC-1
-
Zhong H, May MJ, Jimi E, Ghosh S, (2002) The phosphorylation status of nuclear NF-kappa B determines its association with CBP/p300 or HDAC-1. Mol Cell 9: 625-636.
-
(2002)
Mol Cell
, vol.9
, pp. 625-636
-
-
Zhong, H.1
May, M.J.2
Jimi, E.3
Ghosh, S.4
-
23
-
-
0037428385
-
The NF-kappa B activation in lymphotoxin beta receptor signaling depends on the phosphorylation of p65 at serine 536
-
Jiang X, Takahashi N, Matsui N, Tetsuka T, Okamoto T, (2003) The NF-kappa B activation in lymphotoxin beta receptor signaling depends on the phosphorylation of p65 at serine 536. J Biol Chem 278: 919-926.
-
(2003)
J Biol Chem
, vol.278
, pp. 919-926
-
-
Jiang, X.1
Takahashi, N.2
Matsui, N.3
Tetsuka, T.4
Okamoto, T.5
-
24
-
-
33846317766
-
Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAc
-
Champattanachai V, Chatham JC, Marchase RB, (2007) Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAc. Am J Physiol Cell Physiol 292: C178-C187.
-
(2007)
Am J Physiol Cell Physiol
, vol.292
, pp. 178-187
-
-
Champattanachai, V.1
Chatham, J.C.2
Marchase, R.B.3
-
25
-
-
78349253110
-
Inhibition of O-GlcNAcase in perfused rat hearts by NAG-thiazolines at the time of reperfusion is cardioprotective in an O-GlcNAc dependent manner
-
Laczy B, Marsh SA, Brocks CA, Wittmann I, Chatham JC, (2010) Inhibition of O-GlcNAcase in perfused rat hearts by NAG-thiazolines at the time of reperfusion is cardioprotective in an O-GlcNAc dependent manner. Am J Physiol Heart Circ Physiol 297: H1715-H1727.
-
(2010)
Am J Physiol Heart Circ Physiol
, vol.297
, pp. 1715-1727
-
-
Laczy, B.1
Marsh, S.A.2
Brocks, C.A.3
Wittmann, I.4
Chatham, J.C.5
-
26
-
-
33845632951
-
Glutamine-induced protection of isolated rat heart from ischemia/reperfusion injury is mediated via the hexosamine biosynthesis pathway and increased protein O-GlcNAc levels
-
Liu J, Chatham JC, Marchase RB, (2007) Glutamine-induced protection of isolated rat heart from ischemia/reperfusion injury is mediated via the hexosamine biosynthesis pathway and increased protein O-GlcNAc levels. J Mol Cell Cardiol 42: 177-185.
-
(2007)
J Mol Cell Cardiol
, vol.42
, pp. 177-185
-
-
Liu, J.1
Chatham, J.C.2
Marchase, R.B.3
-
27
-
-
34548424133
-
Increased O-GlcNAc levels during reperfusion lead to improved functional recovery and reduced calpain proteolysis
-
Liu J, Marchase RB, Chatham JC, (2007) Increased O-GlcNAc levels during reperfusion lead to improved functional recovery and reduced calpain proteolysis. Am J Physiol Heart Circ Physiol 293: H1391-H399.
-
(2007)
Am J Physiol Heart Circ Physiol
, vol.293
, pp. 1391-1399
-
-
Liu, J.1
Marchase, R.B.2
Chatham, J.C.3
-
28
-
-
31444452075
-
Increased hexosamine biosynthesis and protein O-GlcNAc levels associated with myocardial protection against calcium paradox and ischemia
-
Liu J, Pang Y, Chang T, Bounelis P, Chatham JC, et al. (2006) Increased hexosamine biosynthesis and protein O-GlcNAc levels associated with myocardial protection against calcium paradox and ischemia. J Mol Cell Cardiol 40: 303-312.
-
(2006)
J Mol Cell Cardiol
, vol.40
, pp. 303-312
-
-
Liu, J.1
Pang, Y.2
Chang, T.3
Bounelis, P.4
Chatham, J.C.5
-
29
-
-
34250841706
-
Glucosamine cardioprotection in perfused rat hearts associated with increased O-linked N-acetylglucosamine protein modification and altered p38 activation
-
Fulop N, Zhang Z, Marchase RB, Chatham JC, et al. (2007) Glucosamine cardioprotection in perfused rat hearts associated with increased O-linked N-acetylglucosamine protein modification and altered p38 activation. Am J Physiol Heart Circ Physiol 292: H2227-H2236.
-
(2007)
Am J Physiol Heart Circ Physiol
, vol.292
, pp. 2227-2236
-
-
Fulop, N.1
Zhang, Z.2
Marchase, R.B.3
Chatham, J.C.4
-
30
-
-
41149165746
-
Cardioprotection by N-acetylglucosamine linkage to cellular proteins
-
Jones SP, Zachara NE, Ngoh GA, Hill BG, Teshima Y, et al. (2008) Cardioprotection by N-acetylglucosamine linkage to cellular proteins. Circulation 117: 1172-1182.
-
(2008)
Circulation
, vol.117
, pp. 1172-1182
-
-
Jones, S.P.1
Zachara, N.E.2
Ngoh, G.A.3
Hill, B.G.4
Teshima, Y.5
-
31
-
-
34548027093
-
Glucosamine administration improves survival rate after severe hemorrhagic shock combined with trauma in rats
-
Nöt LG, Marchase RB, Fülöp N, Brocks CA, Chatham JC, (2007) Glucosamine administration improves survival rate after severe hemorrhagic shock combined with trauma in rats. Shock 28: 345-352.
-
(2007)
Shock
, vol.28
, pp. 345-352
-
-
Nöt, L.G.1
Marchase, R.B.2
Fülöp, N.3
Brocks, C.A.4
Chatham, J.C.5
-
32
-
-
34247110931
-
The protective effects of PUGNAc on cardiac function after trauma-hemorrhage are mediated via increased protein O-GlcNAc levels
-
Zou L, Yang S, Hu S, Chaudry IH, Marchase RB, et al. (2007) The protective effects of PUGNAc on cardiac function after trauma-hemorrhage are mediated via increased protein O-GlcNAc levels. Shock 27: 402-408.
-
(2007)
Shock
, vol.27
, pp. 402-408
-
-
Zou, L.1
Yang, S.2
Hu, S.3
Chaudry, I.H.4
Marchase, R.B.5
-
33
-
-
61949134041
-
Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling
-
Zou L, Yang S, Champattanachai V, Hu S, Chaudry IH, et al. (2009) Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling. Am J Physiol Heart Circ Physiol 296: H515-H523.
-
(2009)
Am J Physiol Heart Circ Physiol
, vol.296
, pp. 515-523
-
-
Zou, L.1
Yang, S.2
Champattanachai, V.3
Hu, S.4
Chaudry, I.H.5
-
34
-
-
79954437198
-
Augmented O-GlcNAc signaling attenuates oxidative stress and calcium overload in cardiomyocytes
-
Ngoh GA, Watson LJ, Facundo HT, Jones SP, (2011) Augmented O-GlcNAc signaling attenuates oxidative stress and calcium overload in cardiomyocytes. Amino Acids 40: 895-911.
-
(2011)
Amino Acids
, vol.40
, pp. 895-911
-
-
Ngoh, G.A.1
Watson, L.J.2
Facundo, H.T.3
Jones, S.P.4
-
35
-
-
59649107122
-
Unique hexosaminidase reduces metabolic survival signal and sensitizes cardiac myocytes to hypoxia/reoxygenation injury
-
Ngoh GA, Facundo HT, Hamid T, Dillmann W, Zachara NE, et al. (2009) Unique hexosaminidase reduces metabolic survival signal and sensitizes cardiac myocytes to hypoxia/reoxygenation injury. Circ Res 104: 41-49.
-
(2009)
Circ Res
, vol.104
, pp. 41-49
-
-
Ngoh, G.A.1
Facundo, H.T.2
Hamid, T.3
Dillmann, W.4
Zachara, N.E.5
-
36
-
-
78049279413
-
O-linked β-N-acetylglucosamine transferase is indispensable in the failing heart
-
Watson LJ, Facundo HT, Ngoh GA, Ameen M, Brainard RE, et al. (2010) O-linked β-N-acetylglucosamine transferase is indispensable in the failing heart. Proc Natl Acad Sci U S A 107: 17797-17802.
-
(2010)
Proc Natl Acad Sci U S A
, vol.107
, pp. 17797-17802
-
-
Watson, L.J.1
Facundo, H.T.2
Ngoh, G.A.3
Ameen, M.4
Brainard, R.E.5
-
37
-
-
0029121917
-
Antisense oligonucleotides to the p65 subunit of NF-kB inhibit human vascular smooth muscle cell adherence and proliferation and prevent neointima formation in rat carotid arteries
-
Autieri MV, Yue TL, Ferstein GZ, Ohlstein E, (1995) Antisense oligonucleotides to the p65 subunit of NF-kB inhibit human vascular smooth muscle cell adherence and proliferation and prevent neointima formation in rat carotid arteries. Biochem Biophys Res Commun 213: 827-836.
-
(1995)
Biochem Biophys Res Commun
, vol.213
, pp. 827-836
-
-
Autieri, M.V.1
Yue, T.L.2
Ferstein, G.Z.3
Ohlstein, E.4
-
38
-
-
0036797999
-
Intraoperative transfection of vein grafts with the NFkappaB decoy in a canine aortocoronary bypass model: a strategy to attenuate intimal hyperplasia
-
Shintani T, Sawa Y, Takahashi T, Matsumiya G, Matsuura N, et al. (2002) Intraoperative transfection of vein grafts with the NFkappaB decoy in a canine aortocoronary bypass model: a strategy to attenuate intimal hyperplasia. Ann Thorac Surg 74: 1132-1137.
-
(2002)
Ann Thorac Surg
, vol.74
, pp. 1132-1137
-
-
Shintani, T.1
Sawa, Y.2
Takahashi, T.3
Matsumiya, G.4
Matsuura, N.5
-
39
-
-
0141988931
-
Overexpression of mutated IkappaBalpha inhibits vascular smooth muscle cell proliferation and intimal hyperplasia formation
-
Zuckerbraun BS, McCloskey CA, Mahidhara RS, Kim PK, Taylor BS, et al. (2003) Overexpression of mutated IkappaBalpha inhibits vascular smooth muscle cell proliferation and intimal hyperplasia formation. J Vasc Surg 38: 812-819.
-
(2003)
J Vasc Surg
, vol.38
, pp. 812-819
-
-
Zuckerbraun, B.S.1
McCloskey, C.A.2
Mahidhara, R.S.3
Kim, P.K.4
Taylor, B.S.5
-
40
-
-
0141703513
-
Tumor necrosis factor-alpha-induced IKK phosphorylation of NF-kappaB p65 on serine 536 is mediated through the TRAF2, TRAF5, and TAK1 signaling pathway
-
Sakurai H, Suzuki S, Kawasaki N, Nakano H, Okazaki T, et al. (2003) Tumor necrosis factor-alpha-induced IKK phosphorylation of NF-kappaB p65 on serine 536 is mediated through the TRAF2, TRAF5, and TAK1 signaling pathway. J Biol Chem 278: 36916-36923.
-
(2003)
J Biol Chem
, vol.278
, pp. 36916-36923
-
-
Sakurai, H.1
Suzuki, S.2
Kawasaki, N.3
Nakano, H.4
Okazaki, T.5
-
41
-
-
0037385065
-
Glucosamine inhibits IL-1beta-induced NFkappaB activation in human osteoarthritic chondrocytes
-
Largo R, Alvarez-Soria MA, Diez-Ortego I, Calvo E, Sanchez-Pernaute O, et al. (2003) Glucosamine inhibits IL-1beta-induced NFkappaB activation in human osteoarthritic chondrocytes. Osteoarthritis Cartilage 11: 290-298.
-
(2003)
Osteoarthritis Cartilage
, vol.11
, pp. 290-298
-
-
Largo, R.1
Alvarez-Soria, M.A.2
Diez-Ortego, I.3
Calvo, E.4
Sanchez-Pernaute, O.5
-
42
-
-
0035871802
-
N-acetylglucosamine prevents IL-1β-mediated activation of human chondrocytes
-
Shikhman AR, Kuhn K, Alaaeddine N, Lotz M, (2001) N-acetylglucosamine prevents IL-1β-mediated activation of human chondrocytes. J Immunol 166: 5155-5160.
-
(2001)
J Immunol
, vol.166
, pp. 5155-5160
-
-
Shikhman, A.R.1
Kuhn, K.2
Alaaeddine, N.3
Lotz, M.4
-
43
-
-
0037116589
-
Glucosamine modulates IL-1-induced activation of rat chondrocytes at a receptor level, and by inhibiting the NF-κB pathway
-
Gouze JN, Bianchi A, Becuwe P, Dauca M, Netter P, et al. (2002) Glucosamine modulates IL-1-induced activation of rat chondrocytes at a receptor level, and by inhibiting the NF-κB pathway. FEBS Lett 510: 166-170.
-
(2002)
FEBS Lett
, vol.510
, pp. 166-170
-
-
Gouze, J.N.1
Bianchi, A.2
Becuwe, P.3
Dauca, M.4
Netter, P.5
-
44
-
-
33644869195
-
Glucosamine sulfate inhibits TNF-alpha and IFN-gamma-induced production of ICAM-1 in human retinal pigment epithelial cells in vitro
-
Chen JT, Liang JB, Chou CL, Chien MW, Shyu RC, et al. (2006) Glucosamine sulfate inhibits TNF-alpha and IFN-gamma-induced production of ICAM-1 in human retinal pigment epithelial cells in vitro. Invest Ophthalmol Vis Sci 47: 664-672.
-
(2006)
Invest Ophthalmol Vis Sci
, vol.47
, pp. 664-672
-
-
Chen, J.T.1
Liang, J.B.2
Chou, C.L.3
Chien, M.W.4
Shyu, R.C.5
-
45
-
-
0036554336
-
Inhibitory actions of glucosamine, a therapeutic agent for osteoarthritis, on the functions of neutrophils
-
Hua J, Sakamoto K, Nagaoka I, (2002) Inhibitory actions of glucosamine, a therapeutic agent for osteoarthritis, on the functions of neutrophils. J Leukoc Biol 71: 632-640.
-
(2002)
J Leukoc Biol
, vol.71
, pp. 632-640
-
-
Hua, J.1
Sakamoto, K.2
Nagaoka, I.3
-
46
-
-
0141482053
-
Immunobiological effects of glucosamine in vitro
-
Forchhammer L, Thorn M, Met O, Gad M, Weidner MS, et al. (2003) Immunobiological effects of glucosamine in vitro. Scand J Immunol 58: 404-411.
-
(2003)
Scand J Immunol
, vol.58
, pp. 404-411
-
-
Forchhammer, L.1
Thorn, M.2
Met, O.3
Gad, M.4
Weidner, M.S.5
-
47
-
-
72449187655
-
The intersections between O-GlcNAcylation and phosphorylation: implications for multiple signaling pathways
-
Zeidan Q, Hart GW, (2010) The intersections between O-GlcNAcylation and phosphorylation: implications for multiple signaling pathways. J Cell Sci 123: 13-22.
-
(2010)
J Cell Sci
, vol.123
, pp. 13-22
-
-
Zeidan, Q.1
Hart, G.W.2
-
48
-
-
33749160125
-
Modification of p53 with O-linked N-acetylglucosamine regulates p53 activity and stability
-
Yang WH, Kim JE, Nam HW, Ju JW, Kim HS, et al. (2006) Modification of p53 with O-linked N-acetylglucosamine regulates p53 activity and stability. Nat Cell Biol 8: 1074-1083.
-
(2006)
Nat Cell Biol
, vol.8
, pp. 1074-1083
-
-
Yang, W.H.1
Kim, J.E.2
Nam, H.W.3
Ju, J.W.4
Kim, H.S.5
-
49
-
-
56249095514
-
NFkappaB activation is associated with its O-GlcNAcylation state under hyperglycemic conditions
-
Yang WH, Park SY, Nam HW, Kim do H, Kang JG, et al. (2008) NFkappaB activation is associated with its O-GlcNAcylation state under hyperglycemic conditions. Proc Natl Acad Sci U S A 105: 17345-50.
-
(2008)
Proc Natl Acad Sci U S A
, vol.105
, pp. 17345-17350
-
-
Yang, W.H.1
Park, S.Y.2
Nam, H.W.3
Kim, D.H.4
Kang, J.G.5
-
50
-
-
0036231781
-
Flux through the hexosamine pathway is a determinant of nuclear factor kappaB- dependent promoter activation
-
James LR, Tang D, Ingram A, Ly H, Thai K, et al. (2002) Flux through the hexosamine pathway is a determinant of nuclear factor kappaB- dependent promoter activation. Diabetes 51: 1146-1156.
-
(2002)
Diabetes
, vol.51
, pp. 1146-1156
-
-
James, L.R.1
Tang, D.2
Ingram, A.3
Ly, H.4
Thai, K.5
-
51
-
-
0035856980
-
Biochemistry and molecular cell biology of diabetic complications
-
Brownlee M, (2001) Biochemistry and molecular cell biology of diabetic complications. Nature 414: 813-20.
-
(2001)
Nature
, vol.414
, pp. 813-820
-
-
Brownlee, M.1
-
52
-
-
58049197848
-
Elevation of global O-GlcNAc levels in 3T3-L1 adipocytes by selective inhibition of O-GlcNAcase does not induce insulin resistance
-
Macauley MS, Bubb AK, Martinez-Fleites C, Davies GJ, Vocadlo DJ, (2008) Elevation of global O-GlcNAc levels in 3T3-L1 adipocytes by selective inhibition of O-GlcNAcase does not induce insulin resistance. J Biol Chem 283: 34687-34695.
-
(2008)
J Biol Chem
, vol.283
, pp. 34687-34695
-
-
Macauley, M.S.1
Bubb, A.K.2
Martinez-Fleites, C.3
Davies, G.J.4
Vocadlo, D.J.5
|