Presynaptic silencing is an endogenous neuroprotectant during excitotoxic insults

Neurobiology of Disease

Volumn 43, Issue 2, 2011, Pages 516-525

  Hogins, Joshua ^a   Crawford, Devon C ^a,b   Jiang, Xiaoping ^a   Mennerick, Steven ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b WASHINGTON UNIVERSITY   (United States)

Author keywords

Excitotoxicity; Glutamate; Hippocampus; Homeostasis; Ischemia; Synaptic plasticity

Indexed keywords

CALCIUM ION; GLUCOSE; GLUTAMIC ACID; OXYGEN; PROTEASOME; PROTEASOME INHIBITOR;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CELL HYPOXIA; CELL LOSS; CONTROLLED STUDY; DEPOLARIZATION; ENZYME INHIBITION; EXCITOTOXICITY; FEMALE; MALE; NERVE CELL LESION; NERVE CELL PLASTICITY; NEUROPROTECTION; NEWBORN; NONHUMAN; PRESYNAPTIC NERVE; PRESYNAPTIC SILENCING; PRIORITY JOURNAL; RAT; SYNAPTOSOME;

ANIMALS; ANIMALS, NEWBORN; CALCIUM SIGNALING; CELLS, CULTURED; DISEASE MODELS, ANIMAL; EXCITATORY POSTSYNAPTIC POTENTIALS; FEMALE; GLUTAMIC ACID; HIPPOCAMPUS; HYPOXIA-ISCHEMIA, BRAIN; ISCHEMIC PRECONDITIONING; MALE; NEURAL INHIBITION; NEUROTOXINS; PRESYNAPTIC TERMINALS; PROTEASOME ENDOPEPTIDASE COMPLEX; PROTEASOME INHIBITORS; RATS; SYNAPTIC TRANSMISSION;

EID: 79958217982     PISSN: 09699961     EISSN: 1095953X     Source Type: Journal    
DOI: 10.1016/j.nbd.2011.05.005     Document Type: Article

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