Inositol 1,4,5-trisphosphate receptors are essential for the development of the second heart field

Journal of Molecular and Cellular Cardiology

Volumn 51, Issue 1, 2011, Pages 58-66

  Nakazawa, Maki ^a   Uchida, Keiko ^a   Aramaki, Megumi ^a   Kodo, Kazuki ^a   Yamagishi, Chihiro ^a   Takahashi, Takao ^a   Mikoshiba, Katsuhiko ^b,c   Yamagishi, Hiroyuki ^a  

^a KEIO UNIVERSITY SCHOOL OF MEDICINE   (Japan)

^b JAPAN SCIENCE AND TECHNOLOGY AGENCY   (Japan)

^c RIKEN BRAIN SCIENCE INSTITUTE   (Japan)

Author keywords

Ca 2+ channel; Ca 2+ signaling; Congenital heart defect; Heart development; Outflow tract

Indexed keywords

CALCIUM ION; INOSITOL 1,4,5 TRISPHOSPHATE RECEPTOR; INOSITOL 1,4,5 TRISPHOSPHATE RECEPTOR 1; INOSITOL 1,4,5 TRISPHOSPHATE RECEPTOR 3; MYOCYTE ENHANCER FACTOR 2; MYOCYTE ENHANCER FACTOR 2C; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR SMYD1; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CALCIUM CELL LEVEL; CALCIUM MOBILIZATION; CALCIUM TRANSPORT; CONTROLLED STUDY; EMBRYO; ENDOPLASMIC RETICULUM; GENE; GENE EXPRESSION; HEART DEVELOPMENT; HEART DISEASE; HEART HYPOPLASIA; HEART MUSCLE CELL; HEART RIGHT VENTRICLE OUTFLOW TRACT; HYPOPLASIA; INOSITOL 1,4,5 TRISPHOSPHATE RECEPTOR 1 GENE; INOSITOL 1,4,5 TRISPHOSPHATE RECEPTOR 3 GENE; MESODERM; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN EXPRESSION; SARCOPLASMIC RETICULUM; SECOND HEART FIELD;

ANIMALS; APOPTOSIS; CALCIUM SIGNALING; DNA-BINDING PROTEINS; ENDOPLASMIC RETICULUM; GENE EXPRESSION PROFILING; HEART; HEART DEFECTS, CONGENITAL; HEART VENTRICLES; IN SITU HYBRIDIZATION; IN SITU NICK-END LABELING; INOSITOL 1,4,5-TRISPHOSPHATE; INOSITOL 1,4,5-TRISPHOSPHATE RECEPTORS; MICE; MICE, KNOCKOUT; MICROARRAY ANALYSIS; MUSCLE PROTEINS; MYOGENIC REGULATORY FACTORS; SARCOPLASMIC RETICULUM; TRANSCRIPTION FACTORS;

MUS;

EID: 79956302999     PISSN: 00222828     EISSN: 10958584     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2011.02.014     Document Type: Article

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