Oxidative stress

Vitiligo

Volumn , Issue , 2010, Pages 231-237

  Picardo, Mauro ^a   Dell'Anna, Maria Lucia ^a  

^a SAN GALLICANO DERMATOLOGICAL INSTITUTE   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

EID: 79955776982     PISSN: None     EISSN: None     Source Type: Book    
DOI: 10.1007/978-3-540-69361-1_27     Document Type: Chapter

References (46)

1. Agrawal D, Shajil EM, Marfatia YS et al (2004) Study of the antioxidant status of vitiligo patients of different age groups in Baroda. Pigment Cell Res 17:289-294
2. Boissy RE, Liu YY, Medrano EE et al (1991) Structural aberration of the rough endoplasmic reticulum and melanosome compartmentalization in long-term cultures of melanocytes from vitiligo patients. J Invest Dermatol 97:395-404
3. Boissy RE, Manga P (2004) On the etiology of contact/occupational vitiligo. Pigment Cell Res 17:208-214
4. Casp CB, She JX, McCormack WT (2001) Genetic association of the catalase gene (CAT) with vitiligo susceptibility. Pigment Cell Res 15:62-66
5. Chavan B, Gillbro JM, Rokos H et al (2006) GTP cyclohydrolase feedback regulatory protein controls cofactor 6-tetrahydrobiopterin synthesis in the cytosol and in the nucleus of epidermal keratinocytes and melanocytes. J Invest Dermatol 126:2481-2489
6. Cucchi ML, Frattini P, Santagostino G et al (2000) Higher plasma catecholamine and metabolite levels in the early phase of nonsegmental vitiligo. Pigment Cell Res 13:28-32
7. Dell'Anna ML, Maresca V, Briganti S et al (2001) Mitochondrial impairment in peripheral blood mononuclear cells during the active phase of vitiligo. J Invest Dermatol 117:908-913
8. Dell'Anna ML, Urbanelli S, Mastrofrancesco A et al (2003) Alterations of mitochondria in peripheral blood mononulcear cells of vitiligo patients. Pigment Cell Res 16:553-559
9. Dell'Anna ML, Camera E, Picardo M (2005) Free radicals. In: Bos JD (ed) Skin immune system, 3rd edn. CRC press, Boca Raton, pp 287-313
10. Dell'Anna ML, Ottaviani M, Albanesi V et al (2007) Membrane lipid alternations as a possible basis for melanocyte degeneration in vitiligo. J Invest Dermatol 127:1226-1233
11. Dell Anna HL, Picardo M (2006). A review and a new hypothesis for non-immunological pathogenetic mechanisms in vitiligo. Pigment Cell Res 19:406-411
12. Gillbro JM, Marles LK, Hibberts NA et al (2004) Autocrine catecholamine biosynthesis and the b2-adrenoceptor signal promote pigmentation in human epidermal melanocytes. J Invest Dermatol 123:346-353
13. Giovannelli L, Bellandi S, Pitozzi V et al (2004) Increased oxidative DNA damage in mononuclear leukocytes in vitiligo. Mut Res 556:101-106
14. Hann SK (1999) A role of the nervous system in the pathogenesis of segmental vitiligo. Pigment Cell Res 7:26
15. 2 stress. J Invest Dermatol 122:307-313
16. Jimbow K, Chen H, Park JS et al (2001) Increased sensitivity of melanocytes to oxidative stress and abnormal expression of tyrosinase-related protein in vitiligo. Br J Dermatol 144:55-65
17. Jimenez-Cervantes C, Martinez-Esparza M, Perez C et al (2001) Inhibition of melanogenesis in response to oxidative stress: transient downregulation of melanocyte differentiation markers and possible involvement of microphtalmia transcription factor. J Cell Sci 114:2335-2344
18. Koca R, Armutcu F, Altinyazar HC et al (2004) Oxidantantioxidant enzymes and lipid peroxidation in generalized vitiligo. Exp Dermatol 29:406-409
19. Kroll TM, Bommiasamy H, Boissy RE et al (2005) 4-tertiary butyl phenol exposure sensitizes human melanocytes to dendritic cell-mediated killing: relevance to vitiligo. J Invest Dermatol 124:798-806
20. Lee AY, Youm YH, Kim NH et al (2004) Keratinocytes in the depigmented epidermis of vitiligo are more vulnerable to trauma (suction) than keratinocytes in the normally pigmented epidermis, resulting in their apoptosis. Br J Dermatol 151:995-1003
21. Lee YA, Kim NH, Choi WI et al (2005) Less keratinocytederived factors related to more keratinocyte apoptosis in depigmented than normally pigmented suction-blistered epidermis may cause passive melanocyte death in vitiligo. J Invest Dermatol 124:976-983
22. Le Poole IC, Das PK, van den Wijngaard RM et al (1993) Review of the etiopathomechanism of vitiligo: a convergence theory. Exp Dermatol 2:145-153
23. Le Poole IC, Das PK (1997) Microscopic changes in vitiligo. Clin Dermatol 15:863-873
24. Le Poole IC, van den Wijingaard R M J G J, Westerhof W et al (1997) Tenascin is overexpressed in vitiligo lesional skin and inhibits melanocyte adhesion. Br J Dermatol 137:171-178
25. Le Poole IC, Sarangarajan R, Zhao Y et al (2001) "VIT1", a novel gene associated with vitiligo. Pigment Cell Res 14:475-484
26. Le Poole IC, Wankowicz-Kalinska A, van den Wijngaard RM et al (2004) Autoimmune aspects of depigmentation in vitiligo. J Invest Dermatol Symp Proc 9:68-72
27. Maresca V, Roccella M, Roccella F et al (1997) Increased sensitivity to peroxidative agents as possible pathogenic factor of melanocyte damage in vitiligo. J Invest Dermatol 109:310-313
28. Medrano EE, Nordlund JJ (1990) Successful culture of adult human melanocytes obtained from normal and vitiligo donors. J Invest Dermatol 95:441-445
29. Montes LF, Abulafia J, Wilborn WH et al (2003) Value of histopathology in vitiligo. Int J Dermatol 42:57-61
30. Moretti S, Spallanzani A, Amato L et al (2002) New insights into the pathogenesis of vitiligo: imbalance of epidermal cytokines at sites of lesions. Pigment Cell Res 15:87-92
31. Moretti S, Spallanzani A, Amato L et al (2002) Vitiligo and epidermal microenvironment: possible involvement of keratinocytesderived cytokines. Arch Dermatol 138:273-274
32. Morrone A, Picardo M, De Luca C et al (1992) Catecholamines and vitiligo. Pigment Cell Res 5:65-69
33. Panucio AL, Vignale R (2003) Ultrastructure studies in stable vitiligo. Am J Dermopathol 25:16-20
34. Picardo M (2009). Lipid-mediated signalling and melanocyte function. Pigment Cell Mel Res 22:152-153
35. Puri N, Mojamdar M, Ramaiah A (1987) In vitro growth characteristics of melanocytes obtained from adult normal and vitiligo subjects. J Invest Dermatol 88:434-438
36. 2 and pterin-6-carboxylic acid. Biochem Biophys Res Commun 292:805-811
37. Schallreuter KU, Wood JM, Pittelkow MR et al (1996) Increased monoamine oxidase A activity in the epidermis of patients with vitiligo. Arch Dermatol Res 288:14-18
38. 2) accumulation in the epidermis of patients with vitiligo and its successful removal by a UVB-activated pseudocatalase. J Invest Dermatol Symp Proc 4:91-96
39. 2 accumulation alters tetrahydrobiopterin (6BH4) recycling in vitiligo: identification of a general mechanism in regulation of all 6BH4-dependent processes? J Invest Dermatol 116:167-174
40. Schallreuter KU, Wood JM, Berger J (2001) Low catalase levels in the epidermis of patients with vitiligo. J Invest Dermatol 97:1081-1085
41. Schallreuter KU, Baharodan P, Picardo M et al (2008) Vitiligo pathgenesis autoimmune disease, generic defect, excessive reactive oxygen species, calcium imbalnac, or what else? Exp Dermatol 17:139-160
42. Schallreuter KU, Chinchiarelli G, Cemeli E et al (2006). Estrogens can contribute to hydrogen peroxide generation and quinone-mediated DNA damage in peripheral blood lymphocytes from patients with vitiligo. J Invest Dermatol 126:1036-1042
43. Stromberg S, Bjorklund MG, Asplund A et al (2008) Transcriptional profiling of melanocytes from patients with vitiligo vulgaris. Pigment Cell Mel Res 21:162-171
44. Taieb A (2000) Intrinsic and extrinsic pathomechanisms in vitiligo. Pigment Cell Res 13:41-47
45. Tursen U, Kaya TI, Derici M E EE et al (2002) Association between catechol-O-methyltransferase polymorphism and vitiligo. Arch Dermatol Res 294:143-146
46. Yang F, Boissy RE (1999) Effects of 4-tertiary butylphenol on the tyrosinase activity in human melanocytes. Pigment Cell Res 12:237-245