ATM is a redox sensor linking genome stability and carbon metabolism

Science Signaling

Volumn 4, Issue 167, 2011, Pages

  Krüger, Antje ^a   Ralser, Markus ^a,b  

^a MAX PLANCK INSTITUTE FOR MOLECULAR GENETICS   (Germany)

^b UNIVERSITY OF CAMBRIDGE   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ATM PROTEIN; GLUCOSE 6 PHOSPHATE DEHYDROGENASE; HEAT SHOCK PROTEIN 27; NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE; PROTEIN P53; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE; CARBON; CELL CYCLE PROTEIN; DNA BINDING PROTEIN; PROTEIN SERINE THREONINE KINASE; TUMOR SUPPRESSOR PROTEIN;

CARBON METABOLISM; CELL CYCLE; CELL METABOLISM; CELL STRESS; DNA REPAIR; GENOMIC INSTABILITY; GLYCOLYSIS; HUMAN; MAMMAL CELL; NONHUMAN; NUCLEOTIDE METABOLISM; PENTOSE PHOSPHATE CYCLE; POSTTRANSCRIPTIONAL GENE SILENCING; PRIORITY JOURNAL; REVIEW; YEAST; ANIMAL; ARTICLE; BIOLOGICAL MODEL; DRUG ANTAGONISM; METABOLISM; MOUSE; NEOPLASM; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PHYSIOLOGY;

ATAXIA TELANGIECTASIA; EUKARYOTA; MAMMALIA;

ANIMALS; CARBON; CELL CYCLE; CELL CYCLE PROTEINS; DNA-BINDING PROTEINS; GENOMIC INSTABILITY; GLUCOSEPHOSPHATE DEHYDROGENASE; HSP27 HEAT-SHOCK PROTEINS; HUMANS; MICE; MODELS, BIOLOGICAL; NEOPLASMS; OXIDATION-REDUCTION; OXIDATIVE STRESS; PROTEIN-SERINE-THREONINE KINASES; REACTIVE OXYGEN SPECIES; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

EID: 79953855431     PISSN: 19450877     EISSN: 19379145     Source Type: Journal    
DOI: 10.1126/scisignal.2001959     Document Type: Review

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28. Acknowledgments. We thank our lab members for useful suggestions and for supporting this article. Funding: Supported by the Max Planck Society.