Congenital hyperinsulinism in Brazilian neonates: A study of histology, KATP channel genes, and proliferation of β cells

Pediatric and Developmental Pathology

Volumn 13, Issue 5, 2010, Pages 375-384

  Lovisolo, Silvana M ^a   Mendonça, Berenice B ^a   Pinto, Emilia M ^a   Manna, Thais Della ^a   Saldiva, Paulo Hilário N ^a   Zerbini, Maria Cláudia N ^a  

^a UNIVERSITY OF SÃO PAULO   (Brazil)

Author keywords

Congenital hyperinsulinism; KATP channel mutations; Ki 67 insulin double immunostaining; Kir 6.2; Pancreatic cells proliferation index; SUR1

Indexed keywords

GENOMIC DNA; INSULIN; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR6.2; KI 67 ANTIGEN; SULFONYLUREA RECEPTOR 1; ABC TRANSPORTER; DRUG RECEPTOR; INWARDLY RECTIFYING POTASSIUM CHANNEL; SULFONYLUREA RECEPTOR;

ADENOMA; ARTICLE; BRAZIL; CELL ENLARGEMENT; CELL PROLIFERATION; CHILD; CLINICAL ARTICLE; CONTROLLED STUDY; CYTOPLASM; DIFFUSE CONGENITAL HYPERINSULINISM; EXON; FEMALE; FOCAL CONGENITAL HYPERINSULINISM; HISTOPATHOLOGY; HUMAN; HUMAN CELL; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; INFANT; MALE; MUTATIONAL ANALYSIS; NEWBORN; NEWBORN DISEASE; PANCREAS; PANCREAS ISLET BETA CELL; PANCREAS RESECTION; PERSISTENT HYPERINSULINEMIC HYPOGLYCEMIA OF INFANCY; PRESCHOOL CHILD; PRIORITY JOURNAL; GENETICS; PATHOLOGY;

ATP-BINDING CASSETTE TRANSPORTERS; BRAZIL; CELL PROLIFERATION; HUMANS; IMMUNOHISTOCHEMISTRY; INFANT, NEWBORN; INSULIN-SECRETING CELLS; PANCREATECTOMY; PERSISTENT HYPERINSULINEMIA HYPOGLYCEMIA OF INFANCY; POTASSIUM CHANNELS, INWARDLY RECTIFYING; RECEPTORS, DRUG;

EID: 79952201881     PISSN: 10935266     EISSN: 16155742     Source Type: Journal    
DOI: 10.2350/08-12-0578.1     Document Type: Article

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