-
1
-
-
33749018788
-
Genetic susceptibility to myocardial infarction and coronary artery disease
-
Topol E.J., Smith J., Plow E.F., Wang Q.K. Genetic susceptibility to myocardial infarction and coronary artery disease. Hum Mol Genet 2006, 15:R117-R123.
-
(2006)
Hum Mol Genet
, vol.15
-
-
Topol, E.J.1
Smith, J.2
Plow, E.F.3
Wang, Q.K.4
-
2
-
-
34249996115
-
A common allele on chromosome 9 associated with coronary heart disease
-
McPherson R., Pertsemlidis A., Kavaslar N., et al. A common allele on chromosome 9 associated with coronary heart disease. Science 2007, 316:1488-1491.
-
(2007)
Science
, vol.316
, pp. 1488-1491
-
-
McPherson, R.1
Pertsemlidis, A.2
Kavaslar, N.3
-
3
-
-
34250010480
-
A common variant on chromosome 9p21 affects the risk of myocardial infarction
-
Helgadottir A., Thorleifsson G., Manolescu A., et al. A common variant on chromosome 9p21 affects the risk of myocardial infarction. Science 2007, 316:1491-1493.
-
(2007)
Science
, vol.316
, pp. 1491-1493
-
-
Helgadottir, A.1
Thorleifsson, G.2
Manolescu, A.3
-
4
-
-
84969213492
-
Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared, controls
-
Wellcome Trust Case Control, Consortium
-
Wellcome Trust Case Control, Consortium Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared, controls. Nature 2007, 447:661-678.
-
(2007)
Nature
, vol.447
, pp. 661-678
-
-
-
5
-
-
34547623750
-
Genomewide association analysis of coronary artery disease
-
Samani N.J., Erdmann J., Hall A.S., et al. Genomewide association analysis of coronary artery disease. N Engl J Med 2007, 357:443-453.
-
(2007)
N Engl J Med
, vol.357
, pp. 443-453
-
-
Samani, N.J.1
Erdmann, J.2
Hall, A.S.3
-
6
-
-
70749096913
-
Genome-wide association of early-onset myocardial infarction with single nucleotide polymorphisms and copy number variants
-
Myocardial Infarction Genetics Consortium
-
Myocardial Infarction Genetics Consortium Genome-wide association of early-onset myocardial infarction with single nucleotide polymorphisms and copy number variants. Nat Genet 2009, 41:334-341.
-
(2009)
Nat Genet
, vol.41
, pp. 334-341
-
-
-
7
-
-
61349177857
-
New susceptibility locus for coronary artery disease on chromosome 3q22.3
-
Erdmann J., Grosshennig A., Braund P.S., et al. New susceptibility locus for coronary artery disease on chromosome 3q22.3. Nat Genet 2009, 41:280-282.
-
(2009)
Nat Genet
, vol.41
, pp. 280-282
-
-
Erdmann, J.1
Grosshennig, A.2
Braund, P.S.3
-
8
-
-
61349137526
-
Genome-wide haplotype association study identifies the SLC22A3-LPAL2-LPA gene cluster as a risk locus for coronary artery disease
-
Trégouët D.A., König I.R., Erdmann J., et al. Genome-wide haplotype association study identifies the SLC22A3-LPAL2-LPA gene cluster as a risk locus for coronary artery disease. Nat Genet 2009, 41:283-285.
-
(2009)
Nat Genet
, vol.41
, pp. 283-285
-
-
Trégouët, D.A.1
König, I.R.2
Erdmann, J.3
-
9
-
-
18744407845
-
Functional SNPs in the lymphotoxin-α gene that are associated with susceptibility to myocardial infarction
-
Ozaki K., Ohnishi Y., Iida A., et al. Functional SNPs in the lymphotoxin-α gene that are associated with susceptibility to myocardial infarction. Nat Genet 2002, 32:650-654.
-
(2002)
Nat Genet
, vol.32
, pp. 650-654
-
-
Ozaki, K.1
Ohnishi, Y.2
Iida, A.3
-
10
-
-
2342480580
-
Functional variation in LGALS2 confers risk of myocardial infarction and regulates lymphotoxin-α secretion in vitro
-
Ozaki K., Inoue K., Sato H., et al. Functional variation in LGALS2 confers risk of myocardial infarction and regulates lymphotoxin-α secretion in vitro. Nature 2004, 429:72-75.
-
(2004)
Nature
, vol.429
, pp. 72-75
-
-
Ozaki, K.1
Inoue, K.2
Sato, H.3
-
11
-
-
61349108698
-
SNPs in BRAP associated with risk of myocardial infarction in Asian populations
-
Ozaki K., Sato H., Inoue K., et al. SNPs in BRAP associated with risk of myocardial infarction in Asian populations. Nat Genet 2009, 41:329-333.
-
(2009)
Nat Genet
, vol.41
, pp. 329-333
-
-
Ozaki, K.1
Sato, H.2
Inoue, K.3
-
12
-
-
67349188883
-
A large-scale genome-wide association study of Asian populations uncovers genetic factors influencing eight quantitative traits
-
Cho Y.S., Go M.J., Kim Y.J., et al. A large-scale genome-wide association study of Asian populations uncovers genetic factors influencing eight quantitative traits. Nat Genet 2009, 41:527-534.
-
(2009)
Nat Genet
, vol.41
, pp. 527-534
-
-
Cho, Y.S.1
Go, M.J.2
Kim, Y.J.3
-
13
-
-
33746512512
-
Principal components analysis corrects for stratification in genome-wide association studies
-
Price A.L., Patterson N.J., Plenge R.M., et al. Principal components analysis corrects for stratification in genome-wide association studies. Nat Genet 2006, 38:904-909.
-
(2006)
Nat Genet
, vol.38
, pp. 904-909
-
-
Price, A.L.1
Patterson, N.J.2
Plenge, R.M.3
-
14
-
-
35748984421
-
The B7 homolog butyrophilin BTN2A1 is a novel ligand for DC-SIGN
-
Malcherek G., Mayr L., Roda-Navarro P., et al. The B7 homolog butyrophilin BTN2A1 is a novel ligand for DC-SIGN. J Immunol 2007, 179:3804-3811.
-
(2007)
J Immunol
, vol.179
, pp. 3804-3811
-
-
Malcherek, G.1
Mayr, L.2
Roda-Navarro, P.3
-
15
-
-
67349259050
-
Regulation of costimulation in the era of butyrophilins
-
Arnett H.A., Escobar S.S., Viney J.L. Regulation of costimulation in the era of butyrophilins. Cytokine 2009, 46:370-375.
-
(2009)
Cytokine
, vol.46
, pp. 370-375
-
-
Arnett, H.A.1
Escobar, S.S.2
Viney, J.L.3
-
16
-
-
20144388906
-
Sarcoidosis is associated with a truncating splice site mutation in BTNL2
-
Valentonyte R., Hampe J., Huse K., et al. Sarcoidosis is associated with a truncating splice site mutation in BTNL2. Nat Genet 2005, 37:357-364.
-
(2005)
Nat Genet
, vol.37
, pp. 357-364
-
-
Valentonyte, R.1
Hampe, J.2
Huse, K.3
-
17
-
-
33646272188
-
Analysis of a functional BTNL2 polymorphism in type 1 diabetes, rheumatoid arthritis, and systemic lupus erythematosus
-
Orozco G., Eerligh P., Sánchez E., et al. Analysis of a functional BTNL2 polymorphism in type 1 diabetes, rheumatoid arthritis, and systemic lupus erythematosus. Hum Immunol 2005, 66:1235-1241.
-
(2005)
Hum Immunol
, vol.66
, pp. 1235-1241
-
-
Orozco, G.1
Eerligh, P.2
Sánchez, E.3
-
18
-
-
29644442440
-
Association of the truncating splice site mutation in BTNL2 with multiple sclerosis is secondary to HLA-DRB1 15
-
Traherne J.A., Barcellos L.F., Sawcer S.J., et al. Association of the truncating splice site mutation in BTNL2 with multiple sclerosis is secondary to HLA-DRB1 15. Hum Mol Genet 2006, 15:155-161.
-
(2006)
Hum Mol Genet
, vol.15
, pp. 155-161
-
-
Traherne, J.A.1
Barcellos, L.F.2
Sawcer, S.J.3
-
19
-
-
77955091234
-
Genome-wide association studies identify IL23R-IL12RB2 and IL10 as Behçet's disease susceptibility loci
-
Mizuki N., Meguro A., Ota M., et al. Genome-wide association studies identify IL23R-IL12RB2 and IL10 as Behçet's disease susceptibility loci. Nat Genet 2010, 42:703-706.
-
(2010)
Nat Genet
, vol.42
, pp. 703-706
-
-
Mizuki, N.1
Meguro, A.2
Ota, M.3
-
20
-
-
0036924040
-
Nuclear export of NF90 is required for interleukin-2 mRNA stabilization
-
Shim J., Lim H., Yates J.R., Karin M. Nuclear export of NF90 is required for interleukin-2 mRNA stabilization. Mol Cell 2002, 10:1331-1344.
-
(2002)
Mol Cell
, vol.10
, pp. 1331-1344
-
-
Shim, J.1
Lim, H.2
Yates, J.R.3
Karin, M.4
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