Activated expression of cardiac adenylyl cyclase 6 reduces dilation and dysfunction of the pressure-overloaded heart

Biochemical and Biophysical Research Communications

Volumn 405, Issue 3, 2011, Pages 349-355

  Sugano, Yasuo ^a,b   Lai, N Chin ^a,b   Gao, Mei Hua ^a,b   Firth, Amy L ^a   Yuan, Jason X J ^a   Lew, Wilbur Y W ^a,b   Hammond, H Kirk ^a,b  

^a UNIVERSITY OF CALIFORNIA SAN DIEGO   (United States)

^b VA SAN DIEGO HEALTHCARE SYSTEM   (United States)

Author keywords

Animal models of human disease; Heart failure; Hypertrophy; Myocardial contraction; Remodeling

Indexed keywords

ADENYLATE CYCLASE; ADENYLYL CYCLASE 6; PHOSPHOLAMBAN; PHOSPHOPROTEIN PHOSPHATASE 1; SODIUM CALCIUM EXCHANGE PROTEIN; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM SIGNALING; CONTROLLED STUDY; ENZYME ACTIVATION; HEART LEFT VENTRICLE CONTRACTILITY; HEART LEFT VENTRICLE ENDSYSTOLIC VOLUME; HEART LEFT VENTRICLE FUNCTION; HEART LEFT VENTRICLE OVERLOAD; MONOCYTE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN PHOSPHORYLATION; SARCOPLASMIC RETICULUM; SODIUM CALCIUM EXCHANGE;

ADENYLATE CYCLASE; ANIMALS; CAFFEINE; CALCIUM; CALCIUM-BINDING PROTEINS; DILATATION, PATHOLOGIC; ENZYME ACTIVATION; HYPERTROPHY, LEFT VENTRICULAR; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; PHOSPHORYLATION; PRESSURE; RECEPTORS, NEUROPEPTIDE Y; SODIUM-CALCIUM EXCHANGER; VENTRICULAR DYSFUNCTION, LEFT;

ANIMALIA; MUS;

EID: 79951579724     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2010.12.113     Document Type: Article

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