Abnormal immune complex processing and spontaneous glomerulonephritis in complement factor h-deficient mice with human complement receptor 1 on erythrocytes

Journal of Immunology

Volumn 185, Issue 6, 2010, Pages 3759-3767

  Alexander, Jessy J ^a   Hack, Bradley K ^a   Jacob, Alexander ^a   Chang, Anthony ^a   Haas, Mark ^b   Finberg, Robert W ^c   Quigg, Richard J ^a  

^a UNIVERSITY OF CHICAGO   (United States)

^b CEDARS SINAI MEDICAL CENTER   (United States)

^c UNIVERSITY OF MASSACHUSETTS MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIGEN; APOFERRITIN; COMPLEMENT COMPONENT C3; COMPLEMENT COMPONENT C3B RECEPTOR; COMPLEMENT FACTOR H; IMMUNOGLOBULIN G;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIGEN ANTIBODY COMPLEX; ARTICLE; BLOOD CELL; CONTROLLED STUDY; ERYTHROCYTE; GLOMERULONEPHRITIS; GLOMERULUS; MOUSE; NONHUMAN; PODOCYTE; PRIORITY JOURNAL; THROMBOCYTE; TRANSGENIC MOUSE;

ANIMALS; BLOOD PLATELETS; COMPLEMENT ACTIVATION; COMPLEMENT FACTOR H; DISEASE MODELS, ANIMAL; ERYTHROCYTES; GLOMERULONEPHRITIS, MEMBRANOPROLIFERATIVE; HUMANS; IMMUNE COMPLEX DISEASES; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MICE, TRANSGENIC; PODOCYTES; PROTEIN PROCESSING, POST-TRANSLATIONAL; RECEPTORS, COMPLEMENT 3B; SERUM SICKNESS; SEVERITY OF ILLNESS INDEX;

EID: 78649852624     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1000683     Document Type: Article

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