Increased C-reactive protein expression exacerbates left ventricular dysfunction and remodeling after myocardial infarction

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 299, Issue 6, 2010, Pages

  Takahashi, Toshiyuki ^a   Anzai, Toshihisa ^a   Kaneko, Hidehiro ^a   Mano, Yoshinori ^a   Anzai, Atsushi ^a   Nagai, Toshiyuki ^a   Kohno, Takashi ^a   Maekawa, Yuichiro ^a   Yoshikawa, Tsutomu ^a   Fukuda, Keiichi ^a   Ogawa, Satoshi ^a  

^a KEIO UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

Apoptosis; Macrophage

Indexed keywords

C REACTIVE PROTEIN; GELATINASE B; MONOCYTE CHEMOTACTIC PROTEIN 1;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; CORONARY ARTERY LIGATION; DISEASE EXACERBATION; ENZYME ACTIVITY; HEART DILATATION; HEART INFARCTION; HEART INFARCTION SIZE; HEART LEFT VENTRICLE FAILURE; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; HISTOLOGY; MACROPHAGE MIGRATION; MALE; MORTALITY; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION;

ANIMALS; ANTERIOR WALL MYOCARDIAL INFARCTION; APOPTOSIS; C-REACTIVE PROTEIN; CARDIOMEGALY; CHEMOKINE CCL2; DISEASE MODELS, ANIMAL; FIBROSIS; GENOTYPE; HUMANS; MACROPHAGES; MALE; MATRIX METALLOPROTEINASE 9; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MYOCARDIUM; PHENOTYPE; TIME FACTORS; UP-REGULATION; VENTRICULAR DYSFUNCTION, LEFT; VENTRICULAR FUNCTION, LEFT; VENTRICULAR REMODELING;

EID: 78649735144     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00001.2010     Document Type: Article

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