A helix-breaking mutation in the epithelial Ca2+ channel TRPV5 leads to reduced Ca2+-dependent inactivation

Cell Calcium

Volumn 48, Issue 5, 2010, Pages 275-287

  Lee, Kyu Pil ^a   Nair, Anil V ^a   Grimm, Christian ^b   van Zeeland, Femke ^a   Heller, Stefan ^b   Bindels, René J M ^a   Hoenderop, Joost G J ^a  

^a RADBOUD UNIVERSITY MEDICAL CENTER   (Netherlands)

^b STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATASE (CALCIUM); CALCIUM CHANNEL; PROLINE; TRANSIENT RECEPTOR POTENTIAL CHANNEL M3; VANILLOID RECEPTOR 5; VANILLOID RECEPTOR 6;

AMINO ACID SUBSTITUTION; APOPTOSIS; ARTICLE; CALCIUM ABSORPTION; CALCIUM CELL LEVEL; CALCIUM CONDUCTANCE; CELL DEATH; CELL MEMBRANE; CONTROLLED STUDY; CYTOTOXICITY; ELECTROPHYSIOLOGY; GENE INACTIVATION; GENE MUTATION; HUMAN; HUMAN CELL; PATCH CLAMP; POINT MUTATION; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN EXPRESSION; PROTEIN FUNCTION; SODIUM CURRENT;

EID: 78649447840     PISSN: 01434160     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.ceca.2010.09.007     Document Type: Article

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